关于行列式D≤50的本原五元恒正二次型

是行嬲式为D的五元恒正二次犁。(辞fj—aji) 在D.~25时①已柽求出所有的本原代表型和类数,本文将糟稽求出25相似文献   

关键词索引《广西科学》2001年第8卷

B白花泡桐 1 3 5　根分化 1 3 5　过氧化物酶 1 3 5　IAA氧化酶 1 3 5　过氧化氢酶 1 3 5斑 1 5 6　含肉率 1 5 6　营养成分 1 5 6　营养评价 1 5 6半欧氏空间 2 6 6　半黎曼流形 2 6 6　调和同态 2 6 6　调和映射 2 6 6铋 1 92　亚硝基 R盐 1 92　乙基紫 1 92　分光光度法 1 92　　　D胆囊结石 1 2 4　超微结构 1 2 4　形态特征 1 2 4等式与不等式约束 2 74　最优化 2 74　广义投影 2 74　强可行方向法 2 74　收敛性 2 74点源二维电场 1　有限元方法 1　电导率分块连续变化 1断裂带 1 1 1　分形分析 1 1 1　分维值 1 1 1多项式理想计…     

一系列新的组合数的恒等式

在熟知的组合恒等式Cn^m=Cn-1^m-1＋Cn^m,1/Cn^m=m/m-1（1/Cn-1^m-1＋Cn^m-1/Cn^m-1）,1/Cn^m＋1/Cn^m＋1=n＋1/n^Cn-1^m的基础上，利用复变函数与初等的方法，得出组合数倒数和的一组非常有趣的组合恒等式，即1/Cn^m＋1/Cn＋1^n＋1/Cn＋2^n＋…＋1/Cn＋m-1^n=n/n-1（1-1/Cn＋m-1^n-1）,1/Cn^m-1/Cn^m＋1＋1/Cn^m＋3＋…＋（-1）^k 1/Cn^m＋k=n＋1/n＋2（1/Cn＋1^m＋（-1）^k 1/Cn＋1^m＋k＋1）等。     

Numerical Value Results OF Guassian Beam Focussing

1　Lensisplacedinbeamwaist　　WeconsiderthecaseofaGaussianbeamthatisincidentatitswaistonathinlensoffocallengthf.Tofindthelocationofthewaistoftheoutputbeamandthebeamradiusatthatpoint,westartwiththeABCDlaw .Attheinputplane( 1 )ω =ω0 1 ,R1 =∝sothat1q1 =1R1 -i λπω20 1 n =-i λπω20 1 nUsingtherelationω2 =ω1and 1 /R2 =( 1 /R1 ) - ( 1 /f)leadsto 1q2 =1q1 - 1f =- 1f -i λπω20 1 nq2 = 1- 1f -i λπω20 1 n=-a+iba2 +b2 ,a=1f,b=λπω20 1 n,q3=q2 + 1 =-aa2 +b2 + 1 + iba2 +b2Atplane( 3)weo…     

关于丢番图方程11~a-17~b=11~c 17~d和17~a-11~b=17~c 11~d

利用初等方法证明了丢番图方程 1 1 a- 1 7b=1 1 c 1 7d 和 1 7a- 1 1 b=1 7c 1 1 d 无非负整数解     

学术影响范围最广的前30名期刊

位次刊　　名他引期刊数引文年代跨度1科学通报 3 73 1 952 - 1 9992中国科学Ｂ 2 54 1 982 - 1 9993生物化学与生物物理进展 1 761 975- 1 9994中国科学Ａ 1 651 982 - 1 9995ＣｈｉｎＳｃｉＢｕｌｌ 1 64 1 961 - 1 9996植物学报 1 551 952 - 1 9997高等学校化学学报 1 3 4 1     

华东师范大学学报(自然科学版)2003年(总111～114期)总目录

题　　　　目作　　者总期号页码数　学加边矩阵自反广义逆的性质 (英)郭文彬 ,魏木生 1 1 1 1切线极坐标的一个应用潘生亮 1 1 1 1 3具有变系数Lyness方程的有界持久性 (英 )李先义 1 1 1 1 7g1 ∞(C)的导子代数 (英)夏利猛 ,杨恒云 ,林　磊 1 1 1 2 1时变系统Laguerre模型辨识及设计变量 (2 )丁肇红 ,沙　泉 ,袁震东 1 1 1 2 5相互ε-临界点球定理的一个注记 (英 )廖蔡生 1 1 2 1特征 2李代数G2 变形的中心扩张李可峰 ,林　磊 1 1 2 6一类非线性三波耦合方程组的精确解贺秀霞 ,聂小兵 1 1 2 1 1单圈图的Laplacian谱 (英 )肖恩利 ,束金龙…     

非连通图(C_(n_1)⊙r_1K_1)∪(C_(n_2)⊙r_2K_1)∪P_2的优美性

讨论非连通图(Cn1⊙r1K1)∪(Cn2⊙r2K1)∪P2的优美性,证明如下结论:设n1,n2,r1,r2是任意自然数,n1≥1,n2≥1,当n1(r1+1)=n2(r2+1)或3n1(r1+1)=n2(r2+1)时,(C4n1⊙r1K1)∪(C4n2⊙r2K1)∪P2是交错图;当n1(r1+1)=n2(r2+1)或(3n1-1)(r1+1)=n2(r2+1)时,非连通图(C4n1-1⊙r1K1)∪(C4n2⊙r2K1)∪P2是优美的,其中P2是2个顶点的路,Cn是n个顶点的圈,Cm⊙rK1是圈Cm的r-冠.     

美国《化学文摘》CA摘转本刊2000年论文统计

美国《化学文摘》CA　　　　　　　　　　　　信阳师范学院学报 (自然科学版 )　　　　　　　　年 ,卷 (期 ) :页码　　　文摘号作者姓名年 ,卷 (期 ) :页码　　　　2 0 0 0 ,1 3 2 (2 6) :1 1 60 3 5 3 3 4 0 s董焕杰 2 0 0 0 ,1 3 (1 ) :2 42 0 0 0 ,1 3 3 (8) :2 4 7 1 0 0 85 0 k古红梅等 2 0 0 0 ,1 3 (1 ) :1 0 22 0 0 0 ,1 3 3 (8) :1 2 81 1 1 1 3 4 4z 张宏伟等 2 0 0 0 ,1 3 (1 ) :12 0 0 0 ,1 3 3 (1 0 ) :61 0 1 3 4 42 8u 金春雪等 2 0 0 0 ,1 3 (1 ) :882 0 0 0 ,1 3 3 (1 0 ) :61 7 1 3 4 5 1 1 r刘彦明等 2 0 0 0 ,1 3 (1 …     

作者索引

薄煜明 ( 2 0 4 )蔡　骅 ( 1 2 1 )陈　昊 ( 1 4 9)陈建华 ( 2 2 4 )陈　晶 ( 4 1 )陈庆伟 ( 1 35)陈森发 ( 2 1 9)陈　勇 ( 35)程　颖 ( 2 2 4 )戴　俊 ( 1 95)戴施华 ( 1 82 )戴先中 ( 35) ,( 1 0 1 ) ,( 1 0 8)单　梁 ( 2 0 )董学平 ( 1 99)杜国平 ( 2 0 4 )费树岷 ( 80 ) ,( 1 1 3)葛晓青 ( 1 86 )巩敦卫 ( 58)郭　毓 ( 53) ,( 1 35)郝国生 ( 58)何亚群 ( 6 8)胡寿松 ( 1 5) ,( 1 31 )胡维礼 ( 53) ,( 1 2 1 ) ,( 1 35)华　冰 ( 1 90 )黄　昆鸟 ( 2 1 9)姬　伟 ( 96 )嵇丽丽 ( 1 4 9)纪志成 ( 71 )姜海波 ( 4 1 )蒋国平 ( 1 1 )蒋　珉 (…     

1 1维N=1超Poincaré张量运算

超引力张量运算目的是构造代数离壳封闭的各种超引力模型.文中建立了1 1维 N=1超Poincare张量运算并详细地导出了多重态的定域超对称变换性质及不变作用量密度公式.     

关于Diophantine方程axm-1/ax-1=yn+1

设a是大于1的正整数,方程(ax~m-1)/(ax-1)=y~n 1是一类重要的指数Diophantine方程,利用同余的方法给出了该方程的适合min(x,y,n)>1的正整数解的取值范围,并将已有的结果m=3和m=4时方程解的情况以一个推论给出.     

Structure of the Cul1-Rbx1-Skp1-F boxSkp2 SCF ubiquitin ligase complex

SCF complexes are the largest family of E3 ubiquitin-protein ligases and mediate the ubiquitination of diverse regulatory and signalling proteins. Here we present the crystal structure of the Cul1-Rbx1-Skp1-F boxSkp2 SCF complex, which shows that Cul1 is an elongated protein that consists of a long stalk and a globular domain. The globular domain binds the RING finger protein Rbx1 through an intermolecular beta-sheet, forming a two-subunit catalytic core that recruits the ubiquitin-conjugating enzyme. The long stalk, which consists of three repeats of a novel five-helix motif, binds the Skp1-F boxSkp2 protein substrate-recognition complex at its tip. Cul1 serves as a rigid scaffold that organizes the Skp1-F boxSkp2 and Rbx1 subunits, holding them over 100 A apart. The structure suggests that Cul1 may contribute to catalysis through the positioning of the substrate and the ubiquitin-conjugating enzyme, and this model is supported by Cul1 mutations designed to eliminate the rigidity of the scaffold.     

DBC1 is a negative regulator of SIRT1

The NAD-dependent protein deacetylase Sir2 (silent information regulator 2) regulates lifespan in several organisms. SIRT1, the mammalian orthologue of yeast Sir2, participates in various cellular functions and possibly tumorigenesis. Whereas the cellular functions of SIRT1 have been extensively investigated, less is known about the regulation of SIRT1 activity. Here we show that Deleted in Breast Cancer-1 (DBC1), initially cloned from a region (8p21) homozygously deleted in breast cancers, forms a stable complex with SIRT1. DBC1 directly interacts with SIRT1 and inhibits SIRT1 activity in vitro and in vivo. Downregulation of DBC1 expression potentiates SIRT1-dependent inhibition of apoptosis induced by genotoxic stress. Our results shed new light on the regulation of SIRT1 and have important implications in understanding the molecular mechanism of ageing and cancer.     

Ginzburg-Landau型泛函极小元的W1,p收敛性

证明当ε→0时，一类Ginzburg-Landau型泛函Eε(u，G)于集合W     

Three-dimensional structure of the neuronal-Sec1-syntaxin 1a complex

Syntaxin 1a and neuronal Sec1 (nSec1) form an evolutionarily conserved heterodimer that is essential for vesicle trafficking and membrane fusion. The crystal structure of the nSec1-syntaxin 1a complex, determined at 2.6 A resolution, reveals that major conformational rearrangements occur in syntaxin relative to both the core SNARE complex and isolated syntaxin. We identify regions of the two proteins that seem to determine the binding specificity of particular Sec1 proteins for syntaxin isoforms, which is likely to be important for the fidelity of membrane trafficking. The structure also indicates mechanisms that might couple the action of upstream effector proteins to conformational changes in syntaxin 1a and nSec1 that lead to core complex formation and membrane fusion.