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1.
针对城市区域多无人机协同物流任务分配问题, 综合考虑不同无人机性能、物流时效性、飞行可靠性等影响因素, 以经济成本、时间损失和安全风险最小为目标函数, 构建多无人机协同物流任务分配模型。因问题规模大、求解复杂度高, 设计改进的量子粒子群算法进行求解。首先,为增强粒子遍历性和多样性, 采用均匀化级联Logistic映射进行粒子初始化; 其次,为避免算法陷入局部最优解, 引入基于高斯分布的粒子变异方式; 最后,为提高算法运行效率, 运用自适应惯性权重方法对粒子赋值。仿真实验结果表明,所构建的模型能够实现任务分配多目标优化, 贴近城市区域无人机物流配送实际; 所提算法与传统量子粒子群算法和遗传算法相比, 任务分配代价分别下降了5.9%和6.3%;并进一步对参数权重设置进行分析, 当3个子目标函数权重系数分别为0.225、0.275和0.500, 种群规模为150时, 算法规划的结果最优。  相似文献   
2.
结合数字化创新的特征与发展趋势,基于5W2H的分析思路,本文从数字化创新特征(WHAT)、数字化创新研究时间线(WHEN)、数字化创新场景(WHERE)、数字化创新动因(WHY)、数字化创新参与者(WHO)、数字化创新途径(HOW)以及数字化创新代价(HOW MUCH)七个方面对数字化创新相关文献进行了梳理.研究发现,现有研究缺乏对数字化创新动机、内在机理、影响结果等方面的深度挖掘与分析,未来数字化创新的研究方向应更加关注数字化创新的跨领域研究、数字化创新的跨层次分析、数字化创新的本土化研究、数字生态系统研究等.  相似文献   
3.
针对经验风险最小化(ERM)这一无约束优化问题,使用随机优化算法.为了提高损失函数值的下降速度,考虑三项共轭梯度法的优点,提出一类新的随机三项共轭梯度算法.并将新算法与经典的随机梯度下降(SGD)类算法和已有的随机共轭梯度算法CGVR进行比较,数值结果表明新算法比SGD类算法在求解ERM问题上更有优势.  相似文献   
4.
 从“十四五”保护与发展的生态关系角度,探讨了生态保护与绿色发展的十大生态关系,阐述了生态保护和绿色发展中的一系列有关生态关系的生态智慧及生态技术,并对长江、黄河等流域的生态保护和修复提出了相关建议。  相似文献   
5.
For this study, we synthesized Aurivillius Bi5Ti3FeO15 ceramic using the generic solid-state reaction route and then performed room-temperature X-ray diffraction to confirm that the compound had a single phase with no impurities. The surface morphology of the prepared sample was observed to contain microstructural grains approximately 0.2–2 μm in size. The dielectric properties of the sample were determ-ined as a function of frequency in a range of approximately 100 Hz to 1 MHz at various temperatures (303 K ≤ T ≤ 773 K). Nyquist plots of the impedance data were found to exhibit a semi-circular arc in the high-temperature region, which is explained by the equivalent electrical circuit (R1C1)(R2QC2), where R1 and R2 represent the resistances associated with the grains and grain boundaries, respectively, C1 and C2 are the re-spective capacitances, and Q is the constant phase element (CPE), which accounts for non-Debye type of behavior. Our results indicate that both the resistance and capacitance of the grain boundaries are more prominent than those of the grains. The alternating current (ac) conductiv-ity data were analyzed based on the Jonscher universal power law, which indicated that the conduction process is dominated by the hopping mechanism. The calculated activation energies of the relaxation and conduction processes were very similar (0.32 to 0.53 eV), from which we conclude that the same type of charge carriers are involved in both processes.  相似文献   
6.
Cobalt (Co)-modified brownmillerite KBiFe2O5 (KBFO; [KBiFe2(1?x)Co2xO5 (x = 0, 0.05)]) polycrystalline is synthesized following the solid-state reaction route. Rietveld refinement of X-ray diffraction data confirmed the phase purity of KBFO and KBiFe1.9Co0.1O5 (KBFCO). The optical bandgap energy (Eg) of KBFO decreased from 1.59 to 1.51 eV because of Co substitution. The decrease in bandgap can be attributed to the tilting of the Fe–O tetrahedral structure of KBFCO. The observed room-temperature Raman peaks of KBFCO shifted by 3 cm?1 toward a lower wavenumber than that of KBFO. The shift in Raman active modes can be attributed to the change in the bond angles and bond lengths of the Fe–O tetrahedral structure and modification in response to oxygen deficiency in KBFO because of Co doping. Compared with that of KBFO, the frequency-dependent dielectric constant and dielectric loss of KBFCO decrease at room temperature, which is a con-sequence of the reduction in oxygen migration and modification in response to vibrational modes present in the sample.  相似文献   
7.

Age-related macular degeneration (AMD) is a chronic and progressive degenerative disease of the retina, which culminates in blindness and affects mainly the elderly population. AMD pathogenesis and pathophysiology are incredibly complex due to the structural and cellular complexity of the retina, and the variety of risk factors and molecular mechanisms that contribute to disease onset and progression. AMD is driven by a combination of genetic predisposition, natural ageing changes and lifestyle factors, such as smoking or nutritional intake. The mechanism by which these risk factors interact and converge towards AMD are not fully understood and therefore drug discovery is challenging, where no therapeutic attempt has been fully effective thus far. Genetic and molecular studies have identified the complement system as an important player in AMD. Indeed, many of the genetic risk variants cluster in genes of the alternative pathway of the complement system and complement activation products are elevated in AMD patients. Nevertheless, attempts in treating AMD via complement regulators have not yet been successful, suggesting a level of complexity that could not be predicted only from a genetic point of view. In this review, we will explore the role of complement system in AMD development and in the main molecular and cellular features of AMD, including complement activation itself, inflammation, ECM stability, energy metabolism and oxidative stress.

  相似文献   
8.
This is a contribution towards a history and philosophy of modeling in its early stages in electromagnetism. In 1873, James Clerk Maxwell (1831–1879) hinted at the methodology of modeling at the end of his Treatise on Electricity and Magnetism. We focus on Maxwell's impact on physicists who immediately followed him, specifically Oliver Lodge (1851–1940) and George Francis FitzGerald (1851–1901). We begin with the role that the scientific concept of model played in the late nineteenth century, as assessed by Ludwig Boltzmann (1844–1906). We then discuss the role of hypothesis as a methodology, the appeal to (dynamical) illustration, and the way Maxwell applied model and working model in his studies of electromagnetism. We show that for Maxwell these key terms were kept distinct, but Lodge did not maintain these distinctions and, in this regard, FitzGerald followed Lodge. Notwithstanding Lodge's influence, Fitzgerald modified Maxwell's theory based on the mechanical model he designed, thereby implicitly taking the first step towards modeling. This methodology consists in drawing consequences from the (mechanical) model to the (electrodynamic) theory and modifying the latter in light of the functioning of the former. At the core of our argument is the thesis that it was a methodological novelty to move from the concept of model to the methodology of modeling. The introduction of modeling as a new methodology into physics in the late nineteenth century was a major event which deserves proper recognition.  相似文献   
9.
Taking a cue from remarks Thomas Kuhn makes in 1990 about the historical turn in philosophy of science, I examine the history of history and philosophy of science within parts of the British philosophical context in the 1950s and early 1960s. During this time, ordinary language philosophy's influence was at its peak. I argue that the ordinary language philosophers' methodological recommendation to analyze actual linguistic practice influences several prominent criticisms of the deductive-nomological model of scientific explanation and that these criticisms relate to the historical turn in philosophy of science. To show these connections, I primarily examine the work of Stephen Toulmin, who taught at Oxford from 1949 to 1954, and Michael Scriven, who completed a dissertation on explanation under Gilbert Ryle and R.B. Braithwaite in 1956. I also consider Mary Hesse's appeal to an ordinary language-influenced account of meaning in her account of the role of models and analogies in scientific reasoning, and W.H. Watson's Wittgensteinian philosophy of science, an early influence on Toulmin. I think there are two upshots to my historical sketch. First, it fills out details of the move away from logical positivism to more historical- and practice-focused philosophies of science. Second, questions about linguistic meaning and the proper targets and aims of philosophical analysis are part and parcel of the historical turn, as well as its reception. Looking at the philosophical background during which so-called linguistic philosophers also had a hand in bringing these questions to prominence helps us understand why.  相似文献   
10.
Diagnosing Darwin's health problems has a long history. Innumerable diagnoses have been proffered in letters, articles, and a handful of dedicated books. Diagnostic speculation has exhibited contrasting somatogenic or psychogenic preferences. Psychogenic accounts dominated mid-century but were soon challenged by somatic explanations citing specific infectious or toxic aetiologies. This tension remains, although psychogenic accounts have tended to be swamped by an array of somatogenic diagnoses championed by biomedical specialists. As a whole, this parade of diagnoses has a striking syndrome du jour quality. Successive conjectures speak of the preoccupations and diagnostic fads of their particular age. Moreover, for many participants in this diagnostic game, unconscious presentism combined with self-serving projection, turning the question of what ailed Darwin into something of a Rorschach test. Never far from the surface was a sometimes-fraught tussle over the man's reputation, animated by the moral valences of the conjectures in play. In a broader sense, this diagnostic game has shaped biographical accounts of Darwin and his career in some significant ways.  相似文献   
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