基于Qt的高性能网络音乐播放器的设计与实现

目前基于网络的音乐播放器功能普遍存在2个主要问题:广告太多;在后台运行不必要的进程而导致性能较低.针对这些情况,设计并实现了一款基于Qt的高性能网络音乐播放器.该播放器利用开放的音乐搜索应用程序编程接口,实现了在线搜索、在线播放、下载音乐及桌面歌词等功能.此外,用户界面设计中采用双缓冲绘图技术,并且程序经过大量代码层面的优化,使得该播放器纯净并拥有非常良好的性能表现.     

基于C++的高效内存池的设计与实现

为了高效、安全地利用计算机内存资源,在大型的软件设计中,往往要进行大量的内存分配与回收操作,为此,C++专门提供了malloc等相关函数进行操作,这些函数能够满足一般的使用,但由于它们调用了操作系统API,所以实际使用时会在操作系统中产生大量的内存碎片,让内存分配成为效率瓶颈,从而降低系统性能.基于此,通过对循环首次适应算法进行改进,设计并实现了基于C++的高效内存池,大幅提升了内存分配与回收的效率.同时,还为内存池编写了相关的分配子,使其能与C++标准库无缝对接,提供了若干具有垃圾回收功能的智能指针,提高了内存管理与程序运行的效率.     

西秦岭中生代花岗岩锆石U-Pb-Lu-Hf同位素特征及地质意义

对西秦岭地区中川岩体、柏家庄岩体和教场坝花岗岩体进行LA-ICP-MS锆石U-Pb同位素年代学分析, 获得其岩浆侵位年龄分别为220±1, 216±6和222±3 Ma, 表明3个岩体均形成于中?晚三叠世。样品的全岩地球化学分析结果表明, 3个岩体具有相似的稀土及微量元素特征, 均表现为显著亏损高场强元素Nb, Ti和P等, 具有明显的右倾式球粒陨石标准化稀土元素配分模式。锆石Lu-Hf同位素分析结果表明, 3个岩体锆石测年样品的ε_Hf(t)值介于?3.31~+1.68之间, 二阶段模式年龄介于1151~1456 Ma之间。岩石成因分析表明, 这些印支期花岗岩体的母岩浆主要来源于新元古代地壳物质的部分熔融。结合岩体形成时代、岩石成因和区域岩浆作用, 认为这些岩体形成于南秦岭与华南板块沿勉略缝合带相碰撞的造山动力学背景, 可能与华南板块的俯冲板片断离有关。     

保持中度生长势成为建筑外表面绿化的选择策略

针对建筑外表面绿化的特殊性提出了中度生长势概念,分析了生长势和绿化寿命的影响因素并提出了概念模型,进一步结合实验探讨了中度生长势的判定.研究结果将补充和丰富城市特殊生境绿化条件下的植物栽培养护理念,为实现节约、可持续的栽培养护模式,延长绿化生命周期提供指导.     

粒度组成对KR搅拌头用浇注料性能的影响

以Andreassen颗粒紧密堆积理论为基础,研究KR搅拌头外衬莫来石浇注料临界粒度和粒度组成对其密度、强度和热震稳定性的影响.结果表明,随着临界粒度的增大,浇注料干燥和烧结后冷态密度与强度均明显提高;临界粒度一定时,粒度组成对浇注料冷态密度、强度以及热震稳定性的影响基本一致,但临界粒度不同的浇注料试样达到最佳性能所对应的粒度组成并不完全相同;与冷态性能相比,浇注料热震稳定性受临界粒度的影响较小.     

New cooperative frequency domain differential modulation and demodulation

An innovative method of cooperative frequency domain differential modulation and demodulation is presented.This method applies the prior knowledge of channel propagation to selecting the variable differential length and carrying out frequency domain modulation.This strategy optimizes the design of system parameters to effectively improve the anti-interference ability of the differential system in time-varied multipath channel circumstance without making the execution more complicating.The simulations and comparisons demonstrate the proposed method is effective,and the results show that it is especially suitable for the fading channel with strong propagation and fast time-variation.     

Oracle 分区表和分区索引在 VLDB 中的研究

分区表和分区索引功能是数据库管理中的关键技术之一,是海量数据库管理(Very Large Databases,VLDB)中一个重要的性能提升机制.分区技术的原理是将逻辑上的一个大表拆分成多个独立的物理分区来存储,从而提高数据I/O性能.面对大数据的存储,几乎所有Oracle数据库都用分区技术来提高查询数据的性能.通过分析一个项目案例,说明如何综合使用分区表和分区索引来提高大数据的查询速度.     

热处理对以Al2O3为绝缘层的并五苯薄膜晶体管性能的影响

采用阳极氧化法制备了Al_2O_3绝缘材料,并制备了以Al_2O_3为绝缘层的并五苯薄膜晶体管器件(OTFT)。Al_2O_3绝缘层经过10 min 350℃氢热处理后,OTFT迁移率比未经氢热处理的增大近20倍,且阈值电压降到了-7 V。Al_2O_3膜MIS结构电容—电压(C–V)特性的平带电压平移量数据表明,Al_2O_3膜经过热处理后,并五苯半导体与Al_2O_3绝缘体界面处以及绝缘体内缺陷态密度显著地降低,Al_2O_3绝缘层和并五苯半导体层之间的接触得到改善,这使得经热处理Al_2O_3膜的OTFT器件性能得到显著的改善。     

HDAC6 rescues neurodegeneration and provides an essential link between autophagy and the UPS

A prominent feature of late-onset neurodegenerative diseases is accumulation of misfolded protein in vulnerable neurons. When levels of misfolded protein overwhelm degradative pathways, the result is cellular toxicity and neurodegeneration. Cellular mechanisms for degrading misfolded protein include the ubiquitin-proteasome system (UPS), the main non-lysosomal degradative pathway for ubiquitinated proteins, and autophagy, a lysosome-mediated degradative pathway. The UPS and autophagy have long been viewed as complementary degradation systems with no point of intersection. This view has been challenged by two observations suggesting an apparent interaction: impairment of the UPS induces autophagy in vitro, and conditional knockout of autophagy in the mouse brain leads to neurodegeneration with ubiquitin-positive pathology. It is not known whether autophagy is strictly a parallel degradation system, or whether it is a compensatory degradation system when the UPS is impaired; furthermore, if there is a compensatory interaction between these systems, the molecular link is not known. Here we show that autophagy acts as a compensatory degradation system when the UPS is impaired in Drosophila melanogaster, and that histone deacetylase 6 (HDAC6), a microtubule-associated deacetylase that interacts with polyubiquitinated proteins, is an essential mechanistic link in this compensatory interaction. We found that compensatory autophagy was induced in response to mutations affecting the proteasome and in response to UPS impairment in a fly model of the neurodegenerative disease spinobulbar muscular atrophy. Autophagy compensated for impaired UPS function in an HDAC6-dependent manner. Furthermore, expression of HDAC6 was sufficient to rescue degeneration associated with UPS dysfunction in vivo in an autophagy-dependent manner. This study suggests that impairment of autophagy (for example, associated with ageing or genetic variation) might predispose to neurodegeneration. Morover, these findings suggest that it may be possible to intervene in neurodegeneration by augmenting HDAC6 to enhance autophagy.