基于人工神经网络BP算法的可编程控制器的设计与应用

本文介绍了一种基于人工神经网络ＢＰ算法的可编程控制器的设计法，此种方法可用于对复杂的工业生产过程量进行控制。     

三阶张弛振子的临界行为

本文报道最简单三阶张弛振子的实验电路、映象描述,及对其从准周期向混沌过渡的临界行为的实验研究,并说明了这些临界行为的普遍意义。我们测得沿临界线准周期集的分数维为:D=0.88±0.01;以及当绕数逼近黄金分割比时,映象参数的收敛比平均值为:■-2.85±0.1。这些结果与圆映象理论很好地符合,从而对Cumming等人得出的D=0.795±0.005及δ_n 的值在-3.3±0.1与-2.7±0.2之间的结论作了较圆满的解释。     

最大需量问题处理中的算法研究

本文给出了处理最大需量问题的算法1和算法2，讨论了算法2的数据结构。     

一类线性不等式组的代数特征解法

按本文的方法，由ｎ×（ｎ＋１）阶欠线性方程组Ａ＾ＴＹ＝０的一个非零解，便能确定线性不等式组ＡＸ≤ｂ之解集合有哪些顶点和棱，以及这些顶点和棱是哪些ｎ×ｎ阶线性方程组的解，从而求得解集合。     

青藏铁路多年冻土区电力铁塔基础寒季施工技术

文章通过在青藏线近1年的施工实践,介绍了青藏铁路多年冻土区电力铁塔基础的寒季施工技术及施工中应注意的问题.     

巷道支护与围岩共同承载的规律与支护方式

巷道支护与围岩的相互作用、相互依赖，具有共同承载作用，利用支护与围岩相互作用实现共同承载是合理利用围岩自承力的途径。     

工程项目管理模式探析

通过分析几种国际上常用的项目管理模式和介绍我国工程项目管理模式的历史,对我国工程项目管理模式的发展提出几点建议.     

MicroRNA Mirn140 modulates Pdgf signaling during palatogenesis

Disruption of signaling pathways such as those mediated by sonic hedgehog (Shh) or platelet-derived growth factor (Pdgf) causes craniofacial abnormalities, including cleft palate. The role that microRNAs play in modulating palatogenesis, however, is completely unknown. We show that, in zebrafish, the microRNA Mirn140 negatively regulates Pdgf signaling during palatal development, and we provide a mechanism for how disruption of Pdgf signaling causes palatal clefting. The pdgf receptor alpha (pdgfra) 3' UTR contained a Mirn140 binding site functioning in the negative regulation of Pdgfra protein levels in vivo. pdgfra mutants and Mirn140-injected embryos shared a range of facial defects, including clefting of the crest-derived cartilages that develop in the roof of the larval mouth. Concomitantly, the oral ectoderm beneath where these cartilages develop lost pitx2 and shha expression. Mirn140 modulated Pdgf-mediated attraction of cranial neural crest cells to the oral ectoderm, where crest-derived signals were necessary for oral ectodermal gene expression. Mirn140 loss of function elevated Pdgfra protein levels, altered palatal shape and caused neural crest cells to accumulate around the optic stalk, a source of the ligand Pdgfaa. These results suggest that the conserved regulatory interactions of mirn140 and pdgfra define an ancient mechanism of palatogenesis, and they provide candidate genes for cleft palate.