油藏数值反演数学模型的研究

文章利用已知的压力和钻井数据,考虑已知实测数据和误差,根据数值反演理论、高斯牛顿分布和井筒生产数据计算敏感矩阵,并使用梯度法减少目标函数,得到最大可能分布;并通过统计分析方法得到最大可能分布的可信度及其密度函数,最终得到准确度更高的渗透率和孔隙度的分布。为油藏数值模拟提供合理的油藏特性参数,从而为制定油藏开发方案提供有效的科学依据。     

一种新型级联码

提出一种由Turbo与纠正多个随机错误的循环码构成的新型级联码,旨在用纠正多个随机错误的循环码纠正Turbo不能纠正的错误,进一步降低误码率.仿真结果表明:信噪比超过一定值后,此级联码的误码率明显低于传统的Turbo码.     

Turbo码译码算法的时延改进分析

通过对Turbo码最大似然译码MAP算法（maximum posteriori probability algorithm）和SOVA算法（soft output viterbi algorithm）的比较和对SOVA算法的比较深入的分析，对Turbo码在译码过程中存在的时延较大的问题做出了一些改进，并且针对引进时延的不同原因讨论了具体的解决方案，通过Matlab进行仿真，表明这些改进是有意义的。     

Function and interaction of maturation-promoting factor and mitogen-activated protein kinase during meiotic maturation and fertilization of oocyte

Mitogen-activated protein kinase (MAP kinase) cascade and maturation-promoting factor (MPF) play very important roles during meiotic maturation and fertilization of oocyte. Interaction between MAP kinase and MPF influences meiotic maturation and fertilization of oocyte throughout the animal kingdom, including stimulation of germinal vesicle breakdown (GVBD), suppression of DNA replication, control of meiotic chromosome segregation, maintenance of metaphase II arrest, and resumption and completion of second meiosis. This review focuses on the function and interaction of MAP kinase and MPF during meiotic maturation and fertilization of oocyte.     

NGF诱导PC12细胞转化为神经样细胞微管相关蛋白2(MAP2)的表达

目的探讨神经生长因子(NGF)诱导PC12细胞分化为神经元样细胞时MAP2的表达变化情况.方法细胞培养6 d后应用共聚焦显微镜观察MAP2表达情况;Western Blot法检测诱导不同时间点MAP2的表达情况.结果免疫组化结果显示:MAP2表达呈阳性;免疫印迹结果显示:MAP2蛋白表达在不同时间点呈动态变化,即随诱导时间延长呈递增趋势.结论 NGF成功诱导PC12细胞分化为神经样细胞,为缺血性脑卒中的体外细胞研究提供细胞参考.     

Turbo／MAP编译码器中的交织器优化设计

讨论采用软输出迭代译交织级联卷积码（Ｔｕｒｂｏ码）编译码器中的交织器优化设计问题．从基于最大后验概率（ＭＡＰ）算法与迭代译码的Ｔｕｒｂｏ码编译码原理出发,给出交织器设计的一些基本方法并分析其特点;提出“保奇偶”的随机交织器设计方案,它能有效地改善某些以提高编码效率为目的的“删余截短”Ｔｕｒｂｏ码的纠错性能．通过计算机仿真得出一些Ｔｕｒｂｏ码在加性高斯白噪声信道中采用不同交织器时的误码率数据,分析了交织类型和交织深度的变化对Ｔｕｒｂｏ码纠错性能的影响．针对Ｔｕｒｂｏ码的特点提出了在卫星通信等应用中交织器设计的实用性结论．     

基于PIC的摩托车发动机电控单元

电控单元(ECU)是电控汽油喷射系统的核心智能部件,其主要功能是根据节气门开度和发动机转速并结合其他参数对点火提前角和喷油脉宽信号进行控制,实现发动机燃烧过程的优化.本文以MAP工况为例,介绍了基于PIC单片机的电控单元开发及其工作原理和硬件系统.     

MAP kinases in plant signal transduction

Mitogen-activated protein kinase (MAPK) pathways are modules involved in the transduction of extracellular signals to intracellular targets in all eukaryotes. Distinct MAPK pathways are regulated by different extracellular stimuli and are implicated in a wide variety of biological processes. In plants there is evidence for MAPKs playing a role in the signaling of abiotic stresses, pathogens and plant hormones. The large number and divergence of plant MAPKs indicates that this ancient mechanism of bioinformatics is extensively used in plants and may provide a new molecular handle on old questions.     

FK506 sensitizes mammalian cells to high osmolarity by modulating p38 MAP kinase activation

The immunosuppressants tacrolimus (FK506) and cyclosporin A (CsA) have increased the survival rates in organ transplantation. Both drugs inhibit the protein phosphatase calcineurin (CaN) in activated T cells, exhibiting similar side-effects. Diabetes is observed more often in FK506 than CsA therapy, probably due to inhibition of new molecular targets other than CaN. We studied FK506 toxicity in mammalian cells. FK506, but not CsA, regulated p38 activation by osmotic stress, and decreased viability in osmostressed cells. In addition, FK506 treatment strongly increased the phosphorylation of the eukaryotic initiation factor-2a (eIF-2a) subunit. eIF-2a phosphorylation, p38 inhibition and cell lethality were relieved by addition of excess amino acids to the medium, suggesting that amino acid availability mediated FK506 toxicity. Therefore, these FK506-dependent responses could be relevant to the non-therapeutic effects of FK506 therapy.Received 16 October 2003; received after revision 8 January 2004; accepted 14 January 2004     

MIPv6切换延迟的优化方案

移动节点从一个接入路由器移到另一个接入路由器时会发生切换.由于链路交换延迟和IP层的相关操作使得移动节点在这个过程中不能够发送和接收数据.为减少移动IPv6切换延迟和满足时实需求,分析了快速移动IPv6和阶层移动IPv6切换协议特点,采用预先注册的方法给出了一种快速和阶层移动IPv6切换的融合方案,并详细的描述了该方案在宏观移动和微观移动情景下的原理架构结构和数据包的流程图.切换延迟分析结果,证明了方案的优越性.