基于复指数矩的图像旋转角检测

提出基于复指数矩多畸变不变性的图像及图像子块的旋转角检测算法.根据复指数矩的定义和算法,计算原图像及旋转图像的复指数矩,求出两图像复指数矩幅角差,即可检测图像的转角.分析了低阶图像复指数矩角向一、二、三阶的性能和特征,指出应用角向一阶复指数矩,可以检测任意旋转角度,其检测误差不超过2°.实验验证了算法的有效性.     

化学抗旱在农业生产中的应用

获鹿县南寨属冀中南太行山半干旱区,山多地少,丘陵起伏,全村大部为旱地,水资源严重不足,地下水埋藏深且不丰富,主要靠天然降水从事农业生产。干旱是南寨村农业生产发展的主要限制因素。为了推动南寨的农业生产,我们积极采用先进的现代旱作技术(如化学抗旱),增强作物、果树本身的抗旱能力,充分发挥它们的增产潜力,以实现高产和稳产。     

我省科研院所目前工作中的几个问题及其对策

本文针对目前我省科研院所工作中存在的诸多问题,提出了１２条对策建议。     

Pathways of proton transfer in the light-driven pump bacteriorhodopsin

The mechanism of proton transport in the light-driven pump bacteriorhodopsin is beginning to be understood. Light causes the all-trans to 13-cis isomerization of the retinal chromophore. This sets off a sequential and directed series of transient decreases in the pKa's of a) the retinal Schiff base, b) an extracellular proton release complex which includes asp-85, and c) a cytoplasmic proton uptake complex which includes asp-96. The timing of these pKa changes during the photoreaction cycle causes sequential proton transfers which result in the net movement of a proton across the protein, from the cytoplasmic to the extracellular surface.     

函数空间Gp（Y│X）的一些基数函数

本文讨论了函数空间Ｃｐ（Ｙ│Ｘ）的基数函数，主要结果有：１，ψ（Ｃｐ（Ｙ│Ｘ）＝Δ（Ｃｐ（Ｙ│Ｘ）＝ｄ（Ｙ） ２，ｔ（Ｃｐ（Ｙ│Ｘ））＝ωＬ（Ｙ） ３，ｈｄ（Ｃｐ（Ｙ│Ｘ））＝ｈＬ（Ｙ＾ω）     

线性搜索方法的改进

线性搜索是最优化计算中最普遍应用的方法，文中对Ｆｌｅｔｃｈｅｒ线性搜索方法作了改进，它具有减少梯度值计算次数的特点，并用数值结果表明了方法的有效性。     

“威霸”除草剂的试验研究

一、前言 我省种植花生的面积比较大,多以春植花生为主。花生地使用除草剂习惯于芽前施用除草剂的较多,如遇上早春阴雨天气,春花生出苗后与杂草混生,给人工除草带来了困难。目前,我省能在花生出苗后使用的除草剂品种较少,因此,1996年我们引     

基于GEO-SLOPE软件的煤矿密集区典型危岩形成机理分析

贵州省凯里市龙场镇渔洞村岔河组老山新村同一位置先后2次发生崩塌灾害,崩塌堆积体灾害导致5人死亡,掩埋6栋房屋,转移安置21户79人。本文针对其高陡斜坡为缓倾坡内上硬下软型,下卧软层薄,采空区距离陡崖面较远的具体特征,通过geo-slope分析软件,结合现场测绘实际情况,建立地质模型并对其进行数值模拟分析,分析其形成机理。     

Glucose sensing by POMC neurons regulates glucose homeostasis and is impaired in obesity

A subset of neurons in the brain, known as 'glucose-excited' neurons, depolarize and increase their firing rate in response to increases in extracellular glucose. Similar to insulin secretion by pancreatic beta-cells, glucose excitation of neurons is driven by ATP-mediated closure of ATP-sensitive potassium (K(ATP)) channels. Although beta-cell-like glucose sensing in neurons is well established, its physiological relevance and contribution to disease states such as type 2 diabetes remain unknown. To address these issues, we disrupted glucose sensing in glucose-excited pro-opiomelanocortin (POMC) neurons via transgenic expression of a mutant Kir6.2 subunit (encoded by the Kcnj11 gene) that prevents ATP-mediated closure of K(ATP) channels. Here we show that this genetic manipulation impaired the whole-body response to a systemic glucose load, demonstrating a role for glucose sensing by POMC neurons in the overall physiological control of blood glucose. We also found that glucose sensing by POMC neurons became defective in obese mice on a high-fat diet, suggesting that loss of glucose sensing by neurons has a role in the development of type 2 diabetes. The mechanism for obesity-induced loss of glucose sensing in POMC neurons involves uncoupling protein 2 (UCP2), a mitochondrial protein that impairs glucose-stimulated ATP production. UCP2 negatively regulates glucose sensing in POMC neurons. We found that genetic deletion of Ucp2 prevents obesity-induced loss of glucose sensing, and that acute pharmacological inhibition of UCP2 reverses loss of glucose sensing. We conclude that obesity-induced, UCP2-mediated loss of glucose sensing in glucose-excited neurons might have a pathogenic role in the development of type 2 diabetes.