用串行干扰消除算法提高同频组网信道估计精度

为降低TD-SCDMA系统同频组网时邻区干扰对信道估计精度的影响,提出了一种逐个邻区串行干扰消除的信道估计算法.算法先消除本小区用户的信号,然后依次对每个邻区进行信道估计,重构其中的强干扰用户信号,再逐个从接收训练序列中加以消除,最后用串行消除各个邻区干扰后的接收训练序列对本小区用户进行信道估计.仿真结果表明,采用串行干扰消除的信道估计方法信道估计误差较低,将其用于上行链路的联合检测接收机,解调性能约提升2 dB.因此,所提出的信道估计算法,可有效消除邻区强干扰用户对信道估计精度的影响,从而提高了同频组网时系统的性能.     

IPv6中的网络隐蔽通道技术研究

为了扩展和丰富信息隐藏的载体，探讨了使用IPv6数据包进行信息隐藏的可能性，针对IPv6的不完善之处，对IPv6协议的数据包格式进行了详尽的分析，在此基础上发现了IPv6数据包中存在保留字段、转发时被节点忽略的字段、定义不完整字段和非关键字段等可隐匿信息的字段，这表明IPv6的冗余字段太多，协议设计时语法规则限定过宽，从而得出IPv6数据包是信息隐藏的良好载体的结论，上述字段构成了对象排序隐蔽通道等19种新的网络隐蔽通道，可用于特定目的的网络隐蔽通信，归纳了这些隐蔽通道的类型，对不同类型的隐蔽通道进行了阐述，最后对IPv6网络隐蔽通道的研究方向进行了展望。     

一种多载波CDMA的子信道分配策略

为了获得高速、高质量的无线数据通信，增强多载波CDMA抗多址干扰和信道衰落能力，提出了一种子载波分配与功率分配相结合的信道分配策略。根据不同子载波上衰落幅度的不同，选择性地传输信息，对于衰落幅度较大的子载波不分配信号能量，将其信号能量分配到传输信号的子载波上，能有效地减小多址干扰，同时又能获得较大的分集增益。这种基于子载波衰落幅度的分配策略与其它文献报道方案比较，具有较低的计算量与较好的收敛性，是一种工程可实现性方案。     

正压送风系统设计探讨

对正压送风系统的设计进行探讨，结论是，当工程的实际情况与规范编制送风量表的假定情况有较大差异时，应通过计算确定正压送风量；矩形风道断面的长宽比宜小于6；在计算风压时当量绝对粗糙度修正系数取较大值，以应对施工过程中的一些不可预见因素；防烟楼梯间的地上部分和地下（半地下）发的防排烟应分别考虑；楼梯间设置正压送风系统时，楼梯间设置的外窗应明确为固定窗；技术交底时应与土建施工管理人员进行沟通，确保建筑风道内表面严密、光滑。     

矩形窄缝流道流动过冷沸腾起始点的实验研究

通过高速摄像可视化观察并结合基础传热数据采集 ,给出了模化工质在中压条件下 ,竖直矩形窄缝流道内过冷流动沸腾起始点的实验结果。讨论了影响矩形窄缝流道内过冷流动沸腾起始点的各因素 ,在Bowering关系式的基础上 ,考虑质量流速和压力对过冷流动沸腾起始点的影响 ,使用多元线性回归的方法 ,得到了关于矩形窄缝流道过冷流动沸腾起始点的修正关系式 ,与实验数据进行比较 ,误差范围为± 30 %。     

传统扩频与混沌扩频的CDMA系统在Rappaport Indoor信道下的性能比较

 对Logistic映射的混沌序列作为扩频序列构成的混沌扩频CDMA系统与传统的m序列扩频CDMA系统进行了对比研究,并且对其二者在Rappaport Indoor信道下的性能在SPW上进行了仿真、分析,具有重要实际意义.     

采用混沌扩频序列的异步码分多址通信系统仿真

为了获取适用于异步码分多址系统的混沌序列 ,提出了一种基于扩频序列性能分析准则的优选方法 ,并用系统仿真的方法进行了验证。通过对异步码分多址系统的性能分析 ,总结了扩频序列的性能准则 ,以此为依据对初值不同的混沌序列进行筛选 ,可以得到性能优异的混沌扩频序列。采用 Monte Carlo的方法 ,在无线衰落信道下对采用混沌序列和 Gold序列的异步码分多址系统进行了仿真。结果表明 :优选的混沌序列性能要优于 Gold序列 ,是扩频序列的一种新的选择     

Research at channel level on the effect of LaCl3 on Radish (Raphanus satirus L.) vacuolar membrane

We recorded slow vacuolar (SV-type) channel currents of Radish vacuoles successfully for the first time by using the whole-vacuolar patch-clamp recording mode. SVtrye currents would increase and threshold potentials of activation would shift towards more negative values with the increase of concentrations of cytosolic Ca2+. When 2.5 mmol/L LaCl3 and 4 mmol/L EGTA were added to bath solutions,SV-type currents were suppressed remarkably. Then adding LaCl3 with different concentrations to pipette solutions, we found that LaCl3 with higher concentrations (＞ 4 × 10-7 mol/L)had a strong inhibitory effect on SV-type currents, while LaCl3 with lower concentrations (≤4 × 10-7 mol/L) promoted channel currents. This promoting effect provides an important basis at channel level for researching further the effects of rare earth on physiological activities of plants and the production-increase effects of rare earth fertilizers on crops.``     

Study of transmembrane La3+ movement in rat ventricular myocytes by the patch-clamp technique

We have studied transmembrane La3+ movement in rat ventricular myocytes for the first time by using the whole-cell patch-clamp recording mode. La3+ (0.01-5.0 mmol/L) could not bring out inward currents through the L-type calcium channel in rat ventricular myocytes, while it could enter the cells by the same way carried by 1μmol/L ionomycin. When the outward Na+ concentration gradient is formed, La3+ can enter the cells via Na-Ca exchange, and the exchange currentsincrease with the increase of external La3+ concentrations. But compared with Na-Ca exchange currents in the same concentration, the former is only 14%-38% of the latter. The patch-clamp experiment indicates that La3+ normally can not enter ventricular myocytes through L-type calcium channel, but it can enter the cells via Na-Ca exchange.     

Calmodulin modulation of ion channels and receptors

Ion channels and receptors are the structural basis for neural signaling and transmission. Recently, the function of ion channels and receptors has been demonstrated to be modulated by many intracellular and extracellular chemicals and signaling molecules. Increasing evidence indicates that the complexity and plasticity of the function of central nervous system is determined by the modulation of ion channels and receptors. Among various mechanisms, Ca 2+ signaling pathways play important roles in neuronal activity and some pathological changes. Ca 2+ influx through ion channels and receptors can modulate its further influx in a feedback way or modulate other ion channels and receptors. The common feature of the modulation is that Ca 2+ /calmodulin (CaM) is the universal mediator. CaM maintains the coordination among ion channels/receptors and intracellular Ca 2+ homeostasis by feedback modulation of ion channels/receptors activity. This review focuses on the modulating processes of ion channels and receptors mediated by CaM, and further elucidates the mechanisms of Ca 2+ signaling.