利用NDIS中间驱动技术实现局域网的并行容错

在Windows2000系统平台上采用NDIS中间驱动技术进行并行容错局域网的研究，并就一些关键技术如失效检测、并行传输的NDIS驱动程序实现给出了较为详细的描述。     

试对比喻重新分类

1、现行的明喻、暗喻和借喻的分类方法未抓住人们的认识规律；2、我们此处提出的新分法将比喻分为初级比喻、中级比喻和高级比喻三种；3、新分法能揭示人们由简单到复杂，由低级到高级的认识规律；4、新分法能有效地帮助我们构造中、高级比喻。     

用蒙特—卡罗模型模拟铪等离子体光谱

阐述了中间耦合情况下的Monte-Carlo理论模型。在局域热动平衡条件下，用Monte-Carlo模型模拟Hf等离子体复杂光谱的部分光谱；类锆铪的光谱，其主要方法是：由未分辩跃迁阵（UTA）方法计算出类锆铪等离子体的4d^4-4d^35p束缚束缚跃迁阵的谱线数目，从高斯分布中随机确定每条谱线的能量，再根据完全中间耦合时能量与强度的关系琰确定每条谱线的强度，从而模拟未分辨跃迁阵Hf^32 4d^4-4d^35p的光谱线。     

关于中值定理"中间点"的渐近问题

在过去的二十年中，对于微分与积分的许多中值公式的“中间点”的渐近性问题已被广泛讨论，关于高阶微分中值公式与二元函数的泰勒公式的“中间点”的渐近性问题也是有趣和有意义的问题，这时将讨论上述两类渐近性问题。     

如何构造辅助函数证明含有“中值点”的导数值的等式

通过实例介绍了在利用微分中值定理证明含有“中值点”的导数值的等式时,如何利用构造法引进辅助函数的方法。     

20MnVK钢含碳量对矿用圆环链性能的影响

本文测试了不同含碳量的20MnVK钢圆环链的性能,通过调整中频淬火回火温度可提高圆环链性能.对20MnVK钢制造C级φ18×64mm矿用圆环链提出看法.     

Alexander disease: putative mechanisms of an astrocytic encephalopathy

Alexander disease (AXD) is the first primary astrocytic disorder. This encephalopathy is caused by dominant mutations in the glial fibrillary acidic protein (GFAP) gene, encoding the main intermediate filament of astrocyte. Pathologically, this neurodegenerative disease is characterised by dystrophic astrocytes containing intermediate filament aggregates associated with myelin abnormalities.More than 20 GFAP mutations have been reported. Many of them cluster in highly conserved regions between several intermediate filaments. Contrary to other intermediate filament-related diseases, AXD seems to be the consequence of a toxic gain of function induced by aggregates. This is supported by the phenotype of mice overexpressing human GFAP. Nevertheless, GFAP null mice display myelin abnormalities and blood-brain barrier dysfunction that are present in AXD.Given the pivotal role of astrocytes in brain physiology, there are many possibilities for astrocytes to dysfunction and to impair the functions of other cells. Physiopathological hypotheses are discussed in the frame of AXD.Received 11 April 2003; received after revision 22 July 2003; accepted 31 July 2003Both authors contributed equally to this work.     

积分中值定理“中间点”的分析性质

讨论了积分中值定理中间点的单调性、连续性、可导性,给出了一组充分条件,并证明了三个相关定理．进一步完善了积分中值定理“中间点”的分析性质．     

Protein folding revisited. A polypeptide chain at the folding – misfolding – nonfolding cross-roads: which way to go?

The structure-function paradigm claims that a specific function of a protein is determined by its unique and rigid three-dimensional (3D) structure. Thus, following its biosynthesis on the ribosome, a protein must fold to be functional. This idea represents one of the cornerstones of modern biology. Numerous cases when, due to the effect of environmental factors or because of genetic defects (mutations), a polypeptide chain has lost its capability to gain a proper functional 3D structure (i.e. became misfolded), seem to confirm this concept. Consequences of such misfolding are well known and represent lost of function, aggregation, development of conformational disorders and cell death. However, the recent revelation of countless examples of intrinsically disordered proteins has cast doubt on the general validity of the structure-function paradigm and revealed an intriguing route of functional disorder. Thus, in a living cell, a polypeptide chain chooses between three potential fates – functional folding, potentially deadly misfolding and mysterious nonfolding. This choice is dictated by the peculiarities of amino acid sequence and/or by the pressure of environmental factors. The aim of the present review is to outline some interesting features of these three routes.Received 5 March 2003; received after revision 28 March 2003; accepted 31 March 2003