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111.
基于多频通信终端设备对天线工作频段和尺寸的要求,提出了一种新型的三频段(GSM/DcS/ISM)小型平面倒F天线,并制作了实物模型.通过测量,得到的3个频段的相对带宽分别为3.4%0,10.3%和2.1%o.分析了在有塑料外壳和手持时对该天线特性的影响,并给出了在自由空间测量得到的方向图和增益.实验结果表明,该天线完全适用于多频无线通信系统. 相似文献
112.
113.
Zhang Yu-xia Yu Lun-yin Liu Ming-qiu Tang Zhi-jiao Xia Dong Wang Ming 《武汉大学学报:自然科学英文版》2002,7(4):493-498
The current study was to investigate mRNA expression of cyclin D2 and p16 during the transition from cardiac myocyte hyperplasia to hypertrophy. Cultured cardiac myocytes (CM) and fibroblasts
(FC) obtained from 1-day-old Sparague-Dawley rats were used in this study. We have determined (1) hyperplasia by cell growth
curve and fluorescence activated cell sorting (FACS); and (2) ultrastructure by electron microscope observation; and (3) expresions
of cyclin D2 mRNA and p16 mRNA by using in situ hybridization and image analysis. The results were shown (1) Results of cell growth curve
and FACS analysis showed CM could proliferate in the first 3 cultured days (4 days in postnatal development). But the ability
decreased quickly, concomitant with the differentiation. (2) The ultrastructure of CM showed the large amount of myofilaments
and mitochondrion and FC showed moderate amount of rough endoplasmic reticulum. (3) The expression of cyclin D2 mRNA in 3−, 4−, 5−day CM group was 0.89 times (p<0.05), 0.80 times (p<0.05) and 0.56 times (p<0.01) of that in 1-day group respectively. P16 mRNA in 2−, 3−, 4−, 5−day CM group were 1.63 times (p<0.01), 1.72 times (p<0.01), 1.99 times (p<0.01) and 2.84 times (p<0.01) of that in 1−day group respectively. It can be concluded that cultured neonatal rat cardiac myocytes could proliferate
during the first 3 cultured days, but the ability of proliferation decreased, from the fourth day, concomitant with differentiation.
Cyclin D2 and p16 have the key roles during the transition from myocyte hyperplasia to hypertrophy.
Biography: Zhang Yu-xia (1974-), female, Master, research direction: cardiovascular pathology. 相似文献
114.
叶华松 《浙江万里学院学报》2004,17(6):66-69
共同富裕是社会主义的本质特征和终极目标.以江泽民同志为核心的党的第三代领导集体继往开来,为全面建设小康社会,最终实现共同富裕的伟大目标,进行了卓有成效的探索. 相似文献
115.
归纳了C8051F020的122个特殊功能寄存器(SFR)的复位状态,对需做初始化的SFR进行了排序,给出了应用于船用减摇跨控制系统的初始化程序(中文注释)范例,并结合范例程序注释了对关键SFR配置内涵的理解,讨论了集成开发环境(IDE)中配置向导的使用方法。 相似文献
116.
117.
内置BS接收画中画的三高频调谐彩色电视机研制李元密,陈皓光,张毅(厦门大学电子工程系)(厦门达真磁记录有限公司)卫星电视广播是解决幅员辽阔和地形复杂所造成的电视复盖率低、节目源少的最好途径。随着“东方红号”和“亚洲一号”广播卫星的发射,必将导致卫星电... 相似文献
118.
本文在基于高速DSP处理芯片Motorola DSP56FS07的佩带式EEG回馈仪上实现采用Kaiser窗的500阶FIR数字陷波器,并进行了计算机仿真与硬件实现。 相似文献
119.
探讨了控轧控冷改善16Mn钢性能的可行性,结果表明,合适的控轧控冷工艺,可细化晶粒,消除带状组织,显著提高16Mn钢的强韧性。此外,依据回归处理得出的冷速与晶粒尺寸及强度之间的关系式,可预报一定冷速下的晶粒尺寸与强度。 相似文献
120.
Prochownik EV 《Cellular and molecular life sciences : CMLS》2005,62(21):2438-2459
The discovery of oncogenes (c-onc’s) and tumor suppressors (TS’s) has led to the concept that cancer arises from defects in
each of these classes of genes or their products. More recently, it has been appreciated that c-onc and TS proteins often
affect one another’s functions. Within this context, I review the two classical TS’s, p53 and the retinoblastoma protein,
and the consequences of their inactivation. The various forms of genomic instability (GI) that underly the high mutation rates
of transformed cells are then discussed. Particular emphasis is placed upon the concept that GI is not only an integral part
of the transformed state but is a prerequisite. Increased oxidative DNA damage, and/or an inabiliy to repair it, can lead
to GI. The review then discusses recent observations showing that loss of the TS protein peroxiredoxin 1 (prdx1) and increased
expression of the c-onc protein c-Myc, each leads to increased oxidative DNA damage. The critical nature of the c-onc-TS interaction
is underscored by that occurring between prdx1 and c-Myc, with the former protein regulating the production of DNA-damaging
reactive oxygen species by the latter. The intimate association between these proteins and others serves as a paradigm for
the exquisite balancing act that c-onc’s and TS’s must maintain in order to properly control normal DNA replication and cellular
proliferation while simultaneously minimizing the acquisition of potentially neoplastic mutations.
Received 10 May 2005; received after revision 3 July 2005; accepted 19 July 2005 相似文献