改性花生壳对重金属含铬废水的吸附研究

对改性花生壳处理含Cr6＋废水进行研究,考察吸附时间、改性花生壳投加量、pH值、Cr6＋溶液初始浓度对吸附效果的影响。实验结果表明,在吸附时间100min、改性花生壳投加量为5.0g/L、pH值2.0、Cr6＋溶液初始浓度25mg/L、常温的优化实验条件下,硝酸改性花生壳比盐酸改性花生壳吸附效果好,硝酸改性花生壳吸附率达到87%,盐酸改性花生壳为71%。改性花生壳是一种较高效的重金属离子吸附剂。     

基于“3＋2＋1”模式的大学英语课程体系改革——以温州大学瓯江学院为例

无论是《大学英语课程教学要求》的几经修改还是大学英语四、六级的改革,都将学生的英语综合应用能力特别是听说能力提到了极其重要的地位。很显然,单一的以精读课为主的课程体系设置已经不能适应新时期大学英语教学的需要,我们迫切需要一个多元化的,能够全方位培养学生英语综合能力的新课程体系。但在课程体系的设置过程中,如何去协调各方面的关系,让课程体系真正发挥其应有的作用,成为大学英语教学实践领域的一个重要课题。     

盐腺在红树耐盐中作用初探

红树林分布在热带及亚热带海岸潮间带,具有多种抗盐机制以适应高盐环境.本文以采自海南东寨港国家级自然保护区潮间带滩涂的19种红树植物为实验材料,探究盐腺在红树耐盐中的作用.利用微分干涉差显微镜观察其叶片的表皮结构,发现老鼠簕（Acanthus ilicifolius）、美国大红树（Rhizophora mangle）、无瓣海桑（Sonneratia apetala）、海桑（Sonneratia caseolaris）、白骨壤（Avicennia marina）、尖瓣海莲（Bruguiera sexangula var.rhynchopetala）、萌芽白骨壤（Avicennia germinans）、桐花树（Parmentiera cerifera）、阿吉木（Aegialitis annulata）等9种红树叶片表面具有盐腺,而玉蕊（Barringtonia racemosa）、水黄皮（Pongamia pinnata）、卤蕨（Acrostichum aureum）、木果楝（Xylocarpus granatum）、银叶树（Heritiera littoralis）、拉贡木（Laguncularia racemosa）、秋茄树（Kandelia obovata）、木榄（Bruguiera gymnorrhiza）、红茄苳（Rhizophora mucronata）和榄李（Lumnitzera racemosa）等10种红树没有盐腺.通过对相同生境下19种红树成熟叶片表面及内部Na＋含量的测定发现,老鼠簕、无瓣海桑、白骨壤等有盐腺红树叶片Na＋含量及Na＋分泌量均较无盐腺红树高.这些结果表明,具盐腺的红树具有积累盐分的特性,也通过盐腺把过多盐分分泌到体外而适应高盐环境.     

电磁场与电磁波实验仿真系统

电磁场与电磁波实验仿真系统是利用计算机仿真技术,对电磁场实验中DH926A型电磁波综合测试仪进行仿真,该系统的虚拟仪器可以代替实际仪器进行相关实验,实现仪器外观相像、实验操作相像和实验结果相像等教学实践要求．该仿真系统由三维显示模块、用户界面模块、数据处理模块和控制模块组成．详细介绍了仿真系统的关键技术,选用专业的零件设计软件SolidWorks进行三维实体建模,使用OSG（OpenSceneGraph）和C＋＋技术进行编程实现实验功能,还创新性提出了仿真系统在效果上保留人为误差的思想．该系统在技术上通过使各模块保持独立,提高了系统的可扩展性,不仅为开发新的实验内容提供方便,也为类似项目开发提供参考．     

“3＋1”人才培养模式下的机械类课程教学改革实践

论述了在“3＋1”人才培养模式下如何重新构建机械类本科教学课程体系;提出了进行相关课程教学和实践环节改革的举措。实施“3＋1”人才培养模式是对现有的机械类本科人才培养机制的创新,促进了学生工程实践与创新能力的提高。     

四年制建筑学专业人才培养模式的研究与实践

四年制建筑学专业人才培养目标应突出重技术和强适应性两个方面,学生毕业后能直接从事中小型工程的建筑设计．“3＋1”人才培养模式能强化工程实践,强化课程设计,强化实验教学,体现了从实践中学习的教育理念．     

职业学校“体育艺术2＋1项目”的实践与思考

伴随着我国社会经济的不断发展，也相应地促进了我国职业学校的发展，在实际的人才培养方面，职业学校不仅在培养学生专业技能方面有所提高，还不断地注重学生综合能力和综合素质的提高，进一步促进了学生的全面发展。因此，本文针对职业学校体育艺术“2＋1项目”的实践进行了具体的分析和研究，希望通过本文的探讨，能够为职业学校“2＋1项目”的实施和推广提供理论性的参考。     

Label-free selective sensing of Pb2＋ lead（II） sensors based on the aggregation of a pyrene fluorescent probe

In the present work, we report a label-free fluo- rescence turn-on approach for the sensitive and selective sensing of Pb2＋. Pyrene with one positive charge was used as the fluorescent probe, and thrombin aptamer （TBA）, which was a G-rich oligonucleotide, was employed to form G-quadruplex with lead（II）. When TBA and Pb2＋ were mixed with lead（II） in an aqueous solution, it was folded into a stable G-quadruplex. Subsequently, a single-stranded nucleic acid-specific nuclease S1 was added. The G-quad- ruplex stabilized by Pb2＋ lead（II） had markedly a significant resistant ability to nuclease S1 digestion. However, in the absence of Pb2＋ lead（II）, no quadruplex or less stable quadruplex was formed and TBA was digested by nuclease S1 in 3 min under the optimized experimental conditions. Finally, pyrene probe was Pb2＋ lead（II）. Electrostatic mixed with oligonucleotide in interactions between oligonu- cleotide （a polyanion） and the probe induced the aggregation of the probe, which in turn produced strong emission of the strong pyrene excimer emission. The intensity of the induced excimer emission was directly proportional to the amount of Pb2＋ added. Our approach shows good selectivity and sen- sitivity for the detection of Pb2＋ with a limit of detection limit as low as 800 nmol/L.     

HERG1 K+ channels on the leukemic cells mediated angiogenesis in vitro

Human ether-a-go-go-related gene （HERG1） K^＋ channels are overexpressed in leukemia, which contributes to neoangiogene- sis. The purpose of this study was to investigate the role of HERG1 K^＋ channels on leukemia angiogenesis. We cultured human umbili- cal vein endothelial cells （HUVECs） in conditioned media, which were derived from leukemic cells with or without E-4031, a HERG1 K^＋ channel special inhibitor. The HUVECs proliferation was mea- sured using CCK-8 assay and migration by a Trans-well. Endothelial tube formation was investigated using Matrigel. Vascular endothelial growth factor （VEGF） levels were tested by ELISA and VEGF mRNA expression using RT-PCR. Our results revealed that blocking HERG1 K^＋ channels could inhibit leukemia-induced HUVECs pro- liferation, migration, and tube formation in vitro. The results sug- gested that HERG1 K~ channels could increase leukemia angio- genesis. Furthermore, blockage of HERG1 K^＋ channels could also decrease leukemic cells secreting VEGF and expressing VEGF mRNA. HERG1 K^＋ channels have a promoting effect on leukemia angiogenesis, and the possible mechanism may be that HERG1 K^＋ channels enhance VEGF expression. Thus, HERG1 K4 channel is a potential target of antiangiogenesis in leukemia.