锂离子电池正极材料LiCu0.05Mn1.95O3.9F0.1的结构与电化学特性

以溶胶-凝胶法合成了阴阳离子复合掺杂尖晶石型锰酸锂正极材料LiCu0.05Mn1.95O3.9F0.1,XRD表征合成产物具有良好的尖晶石结构;SEM测试表明所合成产物的颗粒达到了亚微米级,且分布均匀,形貌较好.以该物质作为锂离子电池的正极材料组装成扣式电池,经充放电循环测试可知 LiCu0.05Mn1.95O3.9F0.1材料比LiMn2O4正极材料能够更好地抑制电池的可逆容量在充放电过程中的衰减,循环性能有了很大改善,表现出很好的电化学可逆特性.     

以MSP430FW427为核心的远程数字水表设计

介绍了以MSP430为核心的低功耗远程数字水表设计。以MSP430FW427为微处理器,采集处理水流量信号;内嵌简化的TCP/IP协议,采用CS8900A的I/O SPACE模式实现数据的传输。分析了LC振荡电路检测流量信号的原理,给出了流量信号的检测、处理、显示及网络通信等模块的软硬件设计及流程。实验表明,该水表具有低功耗、高精度等优点,测得的水流量既可本地显示,也可通过网络实现远程抄表。     

Genetic study on the behavior of honeybees influenced by polyandry system

Within a honeybee population, due to polyandry, there are supersister and halfsister relations, thus many subfamilies exist. For the Chinese honeybee (Apis cerana cerana F.) a phylogenetic dendrogram has been constructed in which 4 subfamilies are clustered based on DNA fingerprint patterns. And it has been observed that each kind of workers is distributed to several dierent sub-families.     

锅炉水位智能控制器

采用ＡＴ８９Ｃ５１微控制器自动检测锅炉的水位状态，自动控制水泵的运行和停止，保证锅炉在正常的水位状态下工作，并且具有上，下限水位报警及锅炉超低水位紧急停炉等功能。     

表面自由能参数表征氟-硅凝聚物系的酸碱性质

通过测定１２种已知酸碱性质的探针分子在不同凝聚物系表面吸附的净保留体积ＶＮ，根据ＶＮ～ｒｉ的关系式，计算了表面自由能参数ｒｉ＾ＬＷ、ｒｉ＾＋、ｒｉ＾－等一系列数据，以酸性作用参数ｒｉ＾＋和碱性作用参数ｒｉＡ＾－相对大小为指标，确定物质表面酸碱性质，该研究为探讨聚合物增强以及偶联、复合机理开辟新的途径，对于开发和选择粘合材料具有一定的实际指导意义。     

可测函数列关于谱测度几乎处处收敛的若干特征

本文把可测函数列几乎处处收敛的几个重要特征推广到谱测度的情形，并证明了它们之间的相互等价性．     

双选择性电极三电极系统中氟离子选择性电极响应特性研究

用双选择性电极三电极系统研究了在非最佳酸度条件下直接测试F^-的可能性，从理论上推导出F^-选择性电极其响应特性满足△E池F|PH2↑PH1dEiF=-2.303RT/Flg(Ka′ [H^ ])|↑[H^ ]tn 1 [H^ ]tn并通过实验可知△E池F的实验值与理论值基本吻合。     

混联法生产氧化铝的熟料烧结问题探讨

采用贵州低品位铝土矿和溶出拜耳赤泥、碱、石灰配制的混合料烧结,在A/S=2.84±、[N]/[A]+[F]=0.97±、[C]/[S]=2.11±、t℃=1275±时,可以获得较好的效果。氧化铝标准溶出率η(?)标=94.64%,氧化铝工业溶出率=92.68%。     

ε-HNIW/F2311 PBX界面结构力学行为模拟

研究了ε-六硝基六氮杂异伍兹烷(ε-HNIW)/F2311界面结构的力学行为. 采用分子动力学(MD)方法,在298～423 K 温度范围内以及NVT系综和COMPASS力场下,对F2311与ε-HNIW (110)晶面所构成PBX界面结构的各种力学性能进行模拟计算. 结果表明,温度达到348 K后,ε-HNIW/F2311界面结构比内层ε-HNIW形状变化(G)和体积变化(K)都变得相对容易,弹性增强的同时可以有效地分散应力,有利于PBX的加工;进一步分析K/G表明,ε-HNIW/F2311界面结构在298～373 K内成型性较好,尤其在348 K左右时最适合进行PBX的压制成型.      

Metformin targets liver tumor-initiating cells through the PI3K/Akt/mTOR survival pathway

Liver tumor-initiating cells （T-ICs） are thought to be inherently resistant to the cytotoxic effects of chemo- therapy, and can self-renewal and maintain tumor-initiating potential. Therefore, effective anticancer research strategies should target the unique properties of T-ICs. In this study, we found that metformin, a first-line drug of choice for the treatment of type 2 diabetes, inhibited liver T-ICs both in vivo and in vitro. Metformin inhibited the formation of hepato- spheres and epithelial-specific antigen-positive （ESA, CD133＋） cell colonies by hepatocellular carcinoma （HCC） cell lines. Metformin also downregulated the expression of several T-IC-related genes which are involved in the signal- ing pathways, governing the self-renewal, proliferation and differentiation of T-ICs. Furthermore, the targeting of liver T-ICs by metformin was PI-3-kinase-Akt-mTOR （PI3K/Akt/ mTOR）-pathway dependent. The PI3K/Akt/mTOR inhibitorLY294002 and rapamycin abolished the inhibitory effect of metformin on CD133＋ cells, and the PI3K/Akt/mTOR stimulator EGF promoted the inhibitory effect of mefformin on CD 133＋ cells. Metformin also dramatically decreased the tumor volume and number of CD133 expressing tumor cells in a xenograft mouse model. Mefformin exerted a synergistic effect with cisplatin to target both T-ICs and non-T-ICs, and resulted in the smallest tumor volume and lowest number of CD133 expressing tumor cells. This study indicates that the antidiabetic drug metformin could potentially be used in combination therapy with chemotherapeutic agents to improve the treatment of liver cancer.