线性系统和迭加原理

以系统的观点给出电路分析中的一个重要定理——迭加定理，并对迭加定理的证明。迭加定理与线性系统的关系．迭加定理的齐次性和可加性，作了较详细的论证。     

超微粒子－有机聚合物薄膜的研究

我们用离子团束－飞行时间质谱（ＩＣＢ－ＴｏＦＭＳ）系统制备了金（Ａｕ）超微粒子－聚乙烯（ＰＥ）薄膜和Ｃ＿（６０）－聚乙烯薄膜。用透射电子显微镜（ＴＥＭ）分析了所制备的样品。它们的结构是Ａｕ超徽粒子镶嵌在多晶的ＰＥ薄膜中，其中Ａｕ原子团呈球形，直径分布在２．０－５．０ｎｍ窄范围内，当沉积基底温度为９０℃时，Ａｕ原子团相互靠近，几乎连接起来，但仍保持原来大小。基底温度为１４０℃蒸积的Ｃ＿（６０）－ｐＥ薄膜具有晶态结构，其晶格常数为１．４５４ｎｍ。     

神经网络在成组技术方面的应用

阐述了成组技术及其在高级制造技术中的作用，在此基础上描述了神经网络在零件编码分类系统中的应用以及用神经网络设计制造单元的方法。     

逻辑学语境下的科学实用主义

作为实用主义的鼻祖,皮尔士首先和主要是一位逻辑学家.他的实用主义从根本上讲是一种逻辑学说,要求从逻辑学理论上对其进行解读.审视科学实用主义同逻辑学的内在的、历史的联系及其形而上的蕴含,可以更清晰地认识实用主义理论的本真意义.     

Liquid-Liquid Equilibrium for 1-Butanol-Water-KF and 1-Butanol-Water-K2CO3 Systems

KF or K2CO3 was added into the 1-butanol-water system and two phases were formed： water-rich phase （water phase） and 1-butanol-rich phase （1-butanol phase）. The liquid liquid equilibrium （LLE） data for 1-butanol-water-KF and 1-butanol-water-K2CO3 systems were measured at 25℃ and showed that 1-butanol phase contained negligible salt and water phase contained negligible 1-butanol when the concentrations of KF and K2CO3 in the water phase were equal to or higher than 27.11% and 31.68% , respectively. Thus water could be separated efficiently from 1-butanol-water by adding KF or K2CO3 into the system. A theoretical calculation of LLE data was calculated by using the Pitzer theory to get water activity in the water phase, and by the models, such as the Wilson, NRTL or the UNIQUAC for the 1-butanol phase. For 1-hutanol-water-KF system, the experimental data were found in good agreement with the calculated results by using Pitzer theory and Wilson equa tion, while for 1-butanol-water-K2CO3 system, the experimental data were found in good agreement with the calculated results by using Pitzer theory and UNIQUAC eauation.     

基于YT0-101总线的PC与单片机通信系统的研究

在对YT0—101通信接口电路及其通信协议研究的基础上，探讨了PC机与8051单片机之间进行etech—bus串口通信软件和硬件设计的原理及方法，给出了单片机与YTO—101总线的部分接口程序。     

Windows环境下光伏水泵报价系统的实现

该文介绍了在Ｗｉｎｄｏｗｅ３．１环境下．利用ＭｉｃｒｏｓｏｆｔＣ（＋＋）及其基本类库（ＭＦＣ）开发光伏水泵报价系统的方法。该方法采用了面向对象技术，使软件具备可重用性。同时，ＭＦＣ类库的使用使复杂的Ｗｉｎ－ｄｏｗｓ应用程序的编程接口变得更容易。该方法对其它工程报价问题也具借鉴意义。     

Induction of hemopoietic chimerism in the caprine fetus by intraperitoneal injection of fetal liver cells

Summary Intraperitoneal injection of allogeneic liver cells from 43-day-old male fetuses into normal 60-day female goat fetuses resulted in persistent hemopoietic chimerism in surviving recipients without clinical evidence of graft-versus-host disease. Transplantation of normal fetal liver cells into preimmunocompetent goat fetuses affected with -D-mannosidosis may provide an alternative strategy for evaluating hemopoietic stem cell transplantation in the treatment of human lysosomal storage diseases.     

Changes in glycosaminoglycan sulfation and protein kinase C subcellular distribution during differentiation of the human colon tumor cell line Caco-2

Summary During the spontaneous differentiation (day 5 to day 15 of the culture) of Caco-2 cells, the sulfation of cell layer glycosaminoglycans increased, whereas protein kinase C activity was concomitantly redistributed from the membrane to the cytosol. The protein kinase C activators, 4-phorbol 12-myristate, 13-acetate and 1,2-dioctanoyl-glycerol inhibited glycosaminoglycan sulfation. By contrast, 4-phorbol 12, 13 didecanoate was ineffective.These results suggest that membrane-bound PKC may exert a modulatory effect on glycosaminoglycan sulfation, and this effect is gradually attenuated as Caco-2 cell differentiation progresses.     

Effect of protein kinase C activation and depletion on insulin stimulation of glycogen synthesis in cultured hepatoma cells

Summary Insulin stimulation of glycogen synthesis was nearly abolished in hepatoma cells shortly treated with 4 ß-phorbol 12 \-myristate, 13 -acetate (protein kinase C activation) but remained unmodified in cells chronically treated with the phorbol ester (protein kinase C depletion). Thus, although exogenous activation of protein kinase C results in an inhibition of insulin action, protein kinase C depletion has no influence on this process. The results suggest that, in hepatoma cells, no endogenous activation of protein kinase C may occur in response to the signal triggered by insulin.