兽用超短波治疗机的装制及临床治疗试验

作者根据人医用机型改装的兽用超短波治疗机,经对患乳房炎的乳牛16例和惠跗关节肿胀的乳牛4例进行超短波治疗。结果急性乳房炎8例和亚急性乳房炎6例全部治愈;慢性乳房炎2例好转。急性和亚急性跗关节肿胀4例基本痊愈。与常规疗法比较,超短波治疗具有省时、省力和节约费用等优点。     

龈沟液炎性内容物及在牙周病中的临床意义

牙周病患者的龈沟液内容物及水平均可发生变化,不同的内容物的变化反映了牙周病变的类型和程度.因此检测龈沟液内容物水平的变化可对牙周炎的诊断、治疗和预后提供一定的客观依据.     

全口覆盖义齿不同固位方式的分析研究

利用全口覆盖式种植义齿对牙列缺失患者进行修复治疗有利于获得义齿良好的稳定和固位的修复效果.全口覆盖式种植义齿根据其固位形式的不同可分为杆卡附着式种植义齿、球帽附着式种植义齿、磁性附着式种植义齿,不同的设计方式对临床应用将带来不同的影响.本文综述了覆盖种植义齿相关的生物力学、种植体数目、即刻加载等方面的研究进展.     

ASICs阻断剂对大鼠脑I/R损伤炎性反应的影响

探讨了酸敏感离子通道阻断剂阿米洛利对大鼠脑I/R损伤性炎性反应的影响。采用Zea Longa线栓法建立Sprague-Dawley(SD)大鼠局灶性脑缺血再灌注(I/R)模型,于脑缺血2h再灌注24h时间点测定血清肿瘤坏死因子-α和血浆内皮素的含量。结果显示各组间比较差异有统计学意义:血清肿瘤坏死因子-α:F=9.937,P=0.000;血浆内皮素:F=49.487,P=0.000;阿米洛剂Ⅰ组和Ⅱ组的血清肿瘤坏死因子-α和血浆内皮素较相应I/R模型组低(P<0.05)。这表明,阿米洛利可能通过阻断酸敏感离子通道减轻Ca2超载,降低肿瘤坏死因子-α和血浆内皮素的含量而抑制炎性反应,发挥神经保护作用。     

颌下腺炎合并良性肿物2例

1一般资料 病例1:患者,男,18岁,该患右侧颌下肿物3月余,初起肿物如小指头大,随着时间的推移而日渐增大,曾在当地抗炎药物治疗,肿物无缩小.入院检查:右侧颌下可扪及3.5 cm×3.0 cm×2.0 cm肿物,质中等,无压痛,边界清,活动好,穿刺未抽出内容物.     

奶牛乳房炎盖医综合防治措施的研究

本文利用HMT试验对重庆市三个奶牛场，153头泌乳牛583个乳区进行了奶牛隐性乳房炎检测，结果表明，阳性乳区为165个，占28．30％，各乳区隐性乳房炎的易感情况存在差异，并提出了防治乳房炎的综合措施。     

牙源性前葡萄膜炎病例分析

急慢性虹膜睫状体炎是常见的一类致盲眼病 其病因及发病机制复杂,病程长,难以治愈 分析研究牙源性前葡萄膜炎病例,有助于更好地诊断治疗此病     

Suppressing progress of pancreatitis through selective inhibition of NF-κB activation by using NAC

OBJECTIVE: To explore the characteristics of NF-KappaB activation in the progress of pancreatitis, the relationship with expression of TNF-alpha in the inflammatory reaction, and prevent the exacerbation of pancreatitis by using NAC. METHOD: Forty-eight rats were divided into three groups: therapy (group C), pancreatitis (group B) and control (group A). NAC served as the inhibitor of NF-KappaB activation. In the time intervals of 1.5, 3.0, 6.0, 12.0 hour, NF-KappaB activation was detected with flow cytometry (FCM) and the expression of TNF-alpha mRNA and protein with in situ hybridization (ISH) and enzyme-linked immuno-sorbent assay (ELISA) respectively. Meanwhile, the level of lipase and amylase in the serum was assayed and the pathological change was evaluated. RESULT: NF-KappaB activation in the pancreatitis group was higher than that in the control group (P<0.01), peaked at 3 hours, and was depressed by the inhibitor of NF-KappaB, NAC. The expression of TNF-alpha as well as the level of lipase and amylase in the serum also rose synchronously with activation of NF-KappaB. In contrast to group A, it was significantly different (P<0.01) in group B. After using NAC in group C, all of these values were decreased and the inflammatory reaction in the pancreas abated evidently. The pathology changes of the pancreas were shown to be alleviated in group C. CONCLUSION: First, NF-KappaB activity is intensively initiated in the course of pancreatitis and shown to have closely relationship with the release of cytokines. Second, use of NAC markedly depressed NF-KappaB activation. TNF-alpha expression is down regulated by cytokines. It is suggested that NAC probably acts as a useful agent for treatment of pancreatitis by indirectly inhibiting activation of NF-KappaB.