具有简单G~2条件的类Bézier曲线曲面

文章给出了一组由3个含参数的4次多项式构成的基函数,在此基础上递推定义了由任意n+1(n≥3)个含参数的代数三角混合函数构成的函数组,称之为n阶λ-Bernstein基,它具有Bernstein基函数的非负性、规范性、对称性等性质。由之定义的λ-Bézier曲线除了具备Bézier曲线的基本性质以外,还具有2个突出的优点:其形状可以在不改变控制顶点的情况下自由调整;当相邻λ-Bézier曲线的控制顶点满足普通Bézier曲线的G1光滑拼接条件时,曲线在公共连接点处可达G2光滑拼接。运用张量积方法定义的λ-Bézier曲面同样具有很多良好的性质。     

水稻bHLH转录因子Os11g39000的功能研究

水稻Os11g39000为非典型的bHLH家族成员,其功能尚不清楚.酵母双杂交实验表明,转录因子Os11g39000可以形成同源蛋白聚合体.随机结合位点筛选实验表明,转录因子Os11g39000可以与DNA结合,并且其结合位点初步确定为TT/CG/CACC/GT/C.Os11g39000基因的组织表达模式分析发现,Os11g39000基因主要在叶片和根中表达,推测该基因可能在叶和根的发育调控中发挥作用.水培实验发现,该基因功能缺陷型转基因株系的根长显著短于野生型,表现出明显的根部发育缺陷,表明Os11g39000在水稻根的发育中起调控作用.Q-PCR分析进一步证明,功能缺陷型转基因植株体内生长素合成和信号转导相关基因表达量显著上升,表明转录因子Os11g39000参与了水稻生长素的合成和信号转导的调控.     

Analysis on Nipper Balance Shaft Movement with Transmission Drive of Comber

The alternating movement of comber nipper balance shaft causes comber vibration, which is considered as the primary limitation for its velocity. First, optimal angular acceleration coefficients of nipper balance shaft were set in this study to acquire theoretical limiting angular velocity. Second,the transmission input model of the input shaft was developed by adopting a numerical approximation method and by considering the Bézier curve as the motion curve of the input shaft. Hence, the motion of nipper balance shaft was controlled. The optimum transmission driver curve of the input shaft was obtained by contrasting the analyses of the actual angular acceleration of nipper balance shaft for each optimal coefficient. Experimental tests validated the results. The nipper balance shaft of the comber reduced comber vibration because the shaft adopted the controllable unit of transmission input.Our research sheds light on the application of controlled mechanisms in textile machines and provides a theoretical basis for increasing the velocity and decreasing the vibration or noise of the comber.     

双路信号电源监测及故障记录装置研究

介绍了双路信号电源监测装置的工作原理、实现方法、主要功能与抗干扰措施。     

On the physiology of metazoa

The problem of integration and control of the various processes of the metazoan organism is a major challenge to the physiologist. The traditional research strategy in dealing with the problem is neuron-oriented and its roots extend back into the last century when knowledge of hormones was lacking. In the present article, the traditional strategy is analyzed in the light of available data and its logical basis is questioned. Different levels of communication are supposed to occur in the animal or human body. Circulating hormones are responsible for the highest level of communication that occurs between organs or tissues. The central concept in the article is that regulation of circulating hormones constitutes a higher level of control relative to regulation of intercellular hormones. This is regardless of whether the latter occurs in the nervous system or elsewhere. The approach is utilized in defining the mechanism that integrates and controls the part processes of the body. The mechanism is defined as endothelial; the vascular endothelial system is the controlling part and the nervous system is one of the subordinate parts. Thanks to the new approach, meaningful biological explanations of major psychiatric disorders are now possible.     

The biotransformation of tritiated 3-dehydroecdysone by crayfish,Procambarus clarkii

The injection of 3-dehydroecdysone (3dhE, 5 /g), the major ecdysteroid secreted by the Y-organ of crayfishProcambarus clarkii, resulted in apolysis within about 5 days. The hormonal response at the molecular level was investigated by injection of the radio-labeled compound; within 3 h of injection of [³H]3dhE, most radio-isotope was found in the extracted epidermal tissues and identified as ecdysone, 20-hydroxyecdysone (20E), and their 3-hydroxy epimers. The biotransformation was undoubtedly performed in the peripheral area of the Y-organ. Cleavage of the polar conjugates, using an enzyme fromHelix pomatia, gave all of the above ecdysteroids including 3dhE. It was also found that the biosynthetic site of 3dhE was different from that of ecdysone at the subcellular level of the Y-organ.     

α-Particle track autoradiographic study of the distribution of a [211At]-astatinated drug in normal tissues of the mouse

Summary The microscopic distribution of the potential endoradiotherapeutic drug, 6-[²¹¹At]-astato-2-methyl-1,4-naphthoquinol bis (diphosphate salt) in normal tissues of the mouse has been studied by -particle track autoradiography. The uptake into critical radiosensitive tissues, especially bone marrow, colon and lung, was low.     

基于神经网络灰色Verhulst算法的装备研制费用预测模型

为了提高装备寿命周期费用LCC(life cycle cost)中研制费用的预测精度,对于呈S型的研制费用累加序列,利用灰色Verhulst模型对其预测.构造基于时间序列的人工神经网络输入输出模式对,利用BP神经网络对灰色Verhulst预测结果进行训练.仿真实例表明,该组合算法预测结果比单纯使用灰色Verhulst模型所得到的结果总体误差要小,将神经网络引入到灰色Verhulst模型中能较好地提高预测精度.     

Hermite-Fejér插值在一重积分Wiener空间下的平均误差

在L2-范数下讨论基于第二类Chebyshev多项式零点的Hermite—Fejér插值多项式列在一重积分Wiener空间下的平均误差,得到了相应量的弱渐近阶．     

Alzheimer’s disease: the impact of age-related changes in reproductive hormones

Two classes of ovarian steroids, estrogens and progestins, are potent in protecting neurons against acute toxic events as well as chronic neurodegeneration. Herein we review the evidence for neuroprotection by both classes of steroids, provide plausible mechanisms for these potent neuroprotective activities and indicate the need for further clinical trials of both estrogens and progestins in protection against acute and chronic conditions that cause neuronal death. Estrogens at concentrations ranging from physiological to pharmacological are neuroprotective in a variety of in vitro and in vivo models of cerebral ischemia and brain trauma as well as in reducing key neuropathologies of Alzheimers disease. While the mechanisms of this potent neuroprotection are currently unresolved, a mitochondrial mechanism is involved. Progestins have been recently shown to activate many of the signaling pathways used by estrogens to neuroprotect, and progestins have been shown to protect against neuronal loss in vitro and in vivo in a variety of models of acute insult. Collectively, results of these animal and tissue culture models suggest that the loss of both estrogens and progestins at the menopause makes the brain more vulnerable to acute insults and chronic neurodegenerative diseases. Further clinical assessment of appropriate regimens of estrogens, progestins and their combination are supported by these data.