系统结构模型分部的底层要素法

本文从系统的底层要素出发，提出并证明了底层要素存在性、底层要素后果集并集的完全性以及它们在同一部分的充分必要条件，由此导出了系统结构模型分部的算法，实际应用进一步说明了这一方法的有效性。     

基于信号重构的一种系统仿真方法

本文根据信号重构的思想，提出了一种系统仿真方法。该方法是对时域矩阵仿真方法的扩展，即保留了时域矩阵方法快速性的特点，又提高了仿真精度。文中还对该方法的截断误差做出了估计，并证明了该方法的收敛性。最后，通过仿真例子说明了该方法的有效性。     

非对称极化散射矩阵的目标识别方法研究

本文针对单基地测量时线性目标的极化散射矩阵为非对称这一实际情况．根据散射矩阵理论将其进行了对称性修正，在此基础之上，利用本征极化作为雷达目标极化特性的表征并进一步提取了目标的极化特征，同时对五种飞机目标进行了识别实验研究，取得了良好的目标分类或识别效果。     

多层弹性体空间一般问题的解析解

本文利用广义柔度矩阵递推法,成功地导出了多层半无限弹性体空间一般问题,在任意荷载作用和两种层间接触条件下的通解.对于这个通解,不管弹性体的层数为多少,最后均不需再解任何联立方程.作为应用,文末计算了地基位移和应力问题实例.     

离散大系统的多级控制

本文使用反馈控制和广义逆矩阵研究离散大系统的多级控制问题,即在局部控制器上加所谓“校正”控制器,达到中性关联作用,使整个系统的性能指标为极小,且得到较小的性能指标偏差的上界。     

华佗再造丸治疗脑血管病疗效评估

用华佗再造丸治疗脑血栓、脑出血及其后遗症52例,其有效率达90.5％,对照组20例,10例脑出血用20％Mannitol及止血剂治疗,10例脑血栓用低分子右旋糖酐及脑络通治疗,有效率为70％,有高度显著性差异,P<0.001,笔者认为该药是治疗脑血管病较理想的中成药,具有镇静神经,滋阴强心,舒经活络,活血化瘀等作用。对病程短的年纪青的病人的疗效更为显著,脑出血性与脑缺血性疾病均有疗效,正是中药一药多用、一药多治的微妙体现。推测对脑血管病后遗症有效可能与该药能促进神经再生因子(NGF)的合成和利用有关。     

粘结平面对称分叉裂纹的奇异积分方程数值解

本文对一个含分叉裂纹的弹性半平面与另一不同材料的半平面粘结的问题用复势方法化为一组三个复Caucby型奇异积分方程。采用修正的Gauss-Legendre和修正的Lobatto-Legendre数值求积法则化成一代数方程组,裂纹尖端的应力强度因子值可从代数方程组的解求得。本文计算得到了弹性半平面、刚体与弹性半平面相粘结、两种不同材料的弹性半平面相粘结的三种问题的几种几何形状的对称分叉裂纹的应力强度因子。本文的结果扩充了“应力强度因子手册”的内容。     

Adrenocortical metabolism and plasma corticosterone in soman intoxicated rabbits

Summary The organophosphate neurotoxin soman produced impairments in adrenocortical RNA and protein metabolism. Fasciculate and reticular cell RNA and protein contents were supporessed with sublethal to acutely lethal dosages (20, 30 and 40 g/kg, s.c.) during the acute excitatory phase of intoxication and at 6–8 h post injection. All three dosages produced ca 90% inactivation of plasma cholinesterase. A transient elevation of plasma corticosterone occurred with 20 g/kg soman whereas there was a protracted increase with 30 g/kg. Corticosterone was not significantly elevated with 40 g/kg, but death occurred at 13±4 min. Thus, the magnitude and/or nature of soman-induced metabolic impairments does not appear to prevent adrenal activation.Supported by US Army Medical Research and Development Command Contract DAMD 17-81-C-1202.     

Quantitative genetic models of sexual selection

Summary Quantitative genetic models of sexual selection have disporven some of the central tenets of both the handicap mechanism and the sexy son hypothesis. These results suggest that the good genes approach to sexual selection may generally lead to erroneous results.Runaway sexual selection seems possible under a wide variety of circumstances. Quantittive genetic models have revealed runaway processes for sexually selected attributes expressed in both sexes and for attributes of parental care. Furthermore, the runaway could occur simultaneously in a series of populations that straddle an environmental gradient. While the models support the feasibility of runaway processes, empirical studies are needed to evaluate whether runaways actually happen. Estimates of critical genetic parameters are particularly needed, as well as measures of natural and sexual selection acting on the same population.The models also show that sexual selection has tremendous potential to produce population differentiation, particularly in epigamic traits. Differentiation is promoted by indeterminancy of evolutionary outcome, transient differences among populations during the final slow approach to equilibrium, sampling drift among equilibrium populations, and the tendency of sexual selection to amplify geographic variation arising from spatial differences in natural selection. Recent work with two- and three-locus models of sexual selection has produced results that parallel the results of the polygenic models^36–38,58. Thus the feature of indeterminate equilibria (outcome dependent on initial conditions) is common to both types of model.