铁路主要干线提速有关问题的研究

在对列车提速的必要性、原则和有限目标进行论述的基础上，就列车提速对运输技术设备的要求和运输组织方面的若干问题进行了初步探讨，分析了多速度客货列车混跑时，不同速度配比的组合方案对线路通过能力的影响，为干线提速的方案选定提供了基础     

钢丝绳张力单片机检测系统

论述了以８０３１为控制核心的钢丝绳张力单片机检测系统的工作原理、数学模型和系统组成，重点介绍了钢丝绳张力检测的工作原理、主要硬件电路的设计特点及软件设计思想，提供了部分硬件电路图及控制软件的流程图。该系统具有置数、动态显示、数据处理、打印及人机对话等功能，并且使用方便，造价低廉。     

马氏体相变的晶体取向关系

本文利用 Bain 应变和马氏体相变的表象理论阐明了马氏体相变的晶体取向关系。正是由于这附加的点阵不变切变和母相的形状协调,某些合金在某种条件下遵循 K—S 关系,而在另一些条件下遵循 N—关系或 G—T 关系。     

c ++起长浮点类的设计及π 的精确计算

文中利用C 面向对象的编程技术，设计并实现了不受计算机位数和软件数据类型限制的，可以完成任意位数的浮点数存储，运算和输入输出的超长浮点类，并据此提出了基于级数的，对圆周率π和自然对数的底e精确到小数点后任意位的计算方法。     

灰铸铁的应力屈服曲面及其三维热弹塑性应力解析

对原有的灰铸铁件应力屈服曲面进行了合理的简化,使其在理论上合理,工程应用上更为方便。建立了灰铸铁件三维非稳态热弹塑性应力场的有限元方法。以灰铸铁哑铃件为对象,用新屈服曲面对其应力发展过程进行了模拟,且比较了用新旧屈服面计算得到的结果。     

Structure of astacin and implications for activation of astacins and zinc-ligation of collagenases.

Astacin, a digestive zinc-endopeptidase from the crayfish Astacus astacus L., is the prototype for the 'astacin family', which includes mammalian metallo-endopeptidases and developmentally regulated proteins of man, fruitfly, frog and sea urchin. Here we report the X-ray crystal structure of astacin, which reveals a deep active-site cleft, with the zinc at its bottom ligated by three histidines, a water molecule and a more remote tyrosine. The third histidine (His 102) forms part of a consensus sequence, shared not only by the members of the astacin family, but also by otherwise sequentially unrelated proteinases, such as vertebrate collagenases. It may therefore represent the elusive 'third' zinc ligand in these enzymes. The amino terminus of astacin is buried forming an internal salt-bridge with Glu 103, adjacent to His 102. Astacin pro-forms extended at the N terminus, as observed for some 'latent' mammalian astacin homologues, did not exhibit this 'active' conformation, indicating an activation mechanism reminiscent of trypsin-like serine proteinases.     

物理实验计算机模拟系统初探

本文用计算机实现了一个物理实验模拟系统,并以利萨如图形为例说明了用计算机模拟物理实验的一般步骤。     

关于中心指数是2的环

本文首先定义了环的中心指数的概念,然后特别讨论了中心指数是2的一些环。     

Ca2+ release induced by inositol 1,4,5-trisphosphate is a steady-state phenomenon controlled by luminal Ca2+ in permeabilized cells.

Low concentrations of inositol 1,4,5-trisphosphate (InsP3) evoke a very rapid mobilization of intracellular Ca2+ stores in many cell types, which can be followed by a further, much slower efflux. Two explanations have been suggested for this biphasic release. The first proposes that the Ca2+ stores vary in their sensitivity to InsP3, and each store releases either its entire contents or nothing (all-or-none release); the second proposes instead that the stores are uniformly sensitive to the effects of InsP3, but that they can release only a fraction of their Ca2+ before their sensitivity is somehow attenuated (steady-state release). Experiments using purified InsP3 receptor molecules reconstituted into lipid vesicles have shown heterogeneity of the receptors in their response to InsP3 under conditions in which the total Ca2+ level at both sides of the receptor is held constant. We now report that in permeabilized A7r5 smooth-muscle cells incubated in Ca(2+)-free medium, the amount of 45Ca2+ remaining in the stores after the rapid transient phase of release is independent of their initial Ca2+ levels, indicating that partially depleted stores are less sensitive to InsP3. Moreover, if the stores are reloaded with 40Ca2+ after the first stimulus, reapplication of the same low concentration of InsP3 will release further 45Ca2+. This recovery of InsP3 sensitivity is almost complete. Under these conditions, Ca2+ release must thus occur by a steady-state mechanism, in which the decreasing Ca2+ content of the stores slows down further release.     

理论价格调整与增加价值调整关系的一个数学模型及分析

本文利用矩阵理论及投入产出分析方法建立了价格指数与增加价值平衡方程,推导出价格调整与增加价值调整关系的数学模型,并对其模型的特点及经济意义进行了分析,为价格调整中的决策者作定量及定性分析时提供参考。