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971.
CIMS中的工程信息管理 总被引:2,自引:0,他引:2
在工程信息管理模式方面提出了使用动态库、受控库和静态库分别管理处于不同阶段的目标数据的管理办法;在管理机制方面讨论了基于这三类库的工程信息管理系统,包括组成该系统的结构管理、设计过程管理、版本管理和计划调度软件,以及支撑上述软件的软件工具.该系统不仅支持工程中的顺序设计,还支持并行设计 相似文献
972.
直流输电系统的暂态仿真 总被引:1,自引:0,他引:1
应用结构矩阵法建立了双桥换流站的数学模型.为提高计算精度,提出以余弦外推公式预报换相电压,以抛物线拟合阀电流以判断阀的截止时刻,采取变步长与定步长相结合的方法来简化处理因阀通断造成的间断点.还讨论了交直流电网的接口问题.算例表明本方法是可行的. 相似文献
973.
根据灵敏度函数的概念,利用模型参考自适应的原理,提出了按系统灵敏度函数与系统传递函数独立设计的二自由度控制方案。论证了二自由度控制原理,并推导了实现这一控制的条件,理论研究和实验结果表明,将这一控制思想运用于参数大范围变化的控制系统设计时,能极大增强系统的鲁棒性。 相似文献
974.
975.
为了回收PDP(等离子显示屏)废电子浆料中的银,实验采用X荧光光谱仪、元素分析仪以及佛尔哈德法(Volhard法)分析其中元素组成及其含量,得到PDP废电子浆料中的银含量为86.20%。采用熔融法回收银,并通过正交实验,确定样品中银回收的最佳实验条件为:还原温度400℃,保温时间3 h,熔融温度1 000℃。银回收率为99.69%,纯度为99.74%。通过XRD分析验证说明回收银产品中含银元素。这种银回收方法工艺简单,环境污染小,易于工业化生产。 相似文献
976.
977.
U. Ruth Michaelis 《Cellular and molecular life sciences : CMLS》2014,71(21):4131-4148
Cell migration plays a central role in a variety of physiological and pathological processes during our whole life. Cellular movement is a complex, tightly regulated multistep process. Although the principle mechanisms of migration follow a defined general motility cycle, the cell type and the context of moving influences the detailed mode of migration. Endothelial cells migrate during vasculogenesis and angiogenesis but also in a damaged vessel to restore vessel integrity. Depending on the situation they migrate individually, in chains or sheets and complex signaling, intercellular signals as well as environmental cues modulate the process. Here, the different modes of cell migration, the peculiarities of endothelial cell migration and specific guidance molecules controlling this process will be reviewed. 相似文献
978.
Blesia Jhon U. Iek Mesak Ratang Westim Hutajulu Halomoan 《Systemic Practice and Action Research》2021,34(1):53-70
Systemic Practice and Action Research - The University of Cenderawasih (Uncen) has developed a locally relevant entrepreneurship education model within its curriculum to increase students’... 相似文献
979.
Piotr Błaszczyk Vladimir Kanovei Karin U. Katz Mikhail G. Katz Taras Kudryk Thomas Mormann David Sherry 《Foundations of Science》2017,22(4):717-731
To explore the extent of embeddability of Leibnizian infinitesimal calculus in first-order logic (FOL) and modern frameworks, we propose to set aside ontological issues and focus on procedural questions. This would enable an account of Leibnizian procedures in a framework limited to FOL with a small number of additional ingredients such as the relation of infinite proximity. If, as we argue here, first order logic is indeed suitable for developing modern proxies for the inferential moves found in Leibnizian infinitesimal calculus, then modern infinitesimal frameworks are more appropriate to interpreting Leibnizian infinitesimal calculus than modern Weierstrassian ones. 相似文献
980.
Hundreds of variants clustered in genomic loci and biological pathways affect human height 总被引:2,自引:0,他引:2
Lango Allen H Estrada K Lettre G Berndt SI Weedon MN Rivadeneira F Willer CJ Jackson AU Vedantam S Raychaudhuri S Ferreira T Wood AR Weyant RJ Segrè AV Speliotes EK Wheeler E Soranzo N Park JH Yang J Gudbjartsson D Heard-Costa NL Randall JC Qi L Vernon Smith A Mägi R Pastinen T Liang L Heid IM Luan J Thorleifsson G Winkler TW Goddard ME Sin Lo K Palmer C Workalemahu T Aulchenko YS Johansson A Zillikens MC Feitosa MF Esko T Johnson T Ketkar S Kraft P Mangino M Prokopenko I Absher D Albrecht E 《Nature》2010,467(7317):832-838
Most common human traits and diseases have a polygenic pattern of inheritance: DNA sequence variants at many genetic loci influence the phenotype. Genome-wide association (GWA) studies have identified more than 600 variants associated with human traits, but these typically explain small fractions of phenotypic variation, raising questions about the use of further studies. Here, using 183,727 individuals, we show that hundreds of genetic variants, in at least 180 loci, influence adult height, a highly heritable and classic polygenic trait. The large number of loci reveals patterns with important implications for genetic studies of common human diseases and traits. First, the 180 loci are not random, but instead are enriched for genes that are connected in biological pathways (P = 0.016) and that underlie skeletal growth defects (P?0.001). Second, the likely causal gene is often located near the most strongly associated variant: in 13 of 21 loci containing a known skeletal growth gene, that gene was closest to the associated variant. Third, at least 19 loci have multiple independently associated variants, suggesting that allelic heterogeneity is a frequent feature of polygenic traits, that comprehensive explorations of already-discovered loci should discover additional variants and that an appreciable fraction of associated loci may have been identified. Fourth, associated variants are enriched for likely functional effects on genes, being over-represented among variants that alter amino-acid structure of proteins and expression levels of nearby genes. Our data explain approximately 10% of the phenotypic variation in height, and we estimate that unidentified common variants of similar effect sizes would increase this figure to approximately 16% of phenotypic variation (approximately 20% of heritable variation). Although additional approaches are needed to dissect the genetic architecture of polygenic human traits fully, our findings indicate that GWA studies can identify large numbers of loci that implicate biologically relevant genes and pathways. 相似文献