环氧乙烷四氢呋喃共聚醚[P(E-CO-T)]的研制

研究了环氧乙烷与四氢呋喃的共聚.使用BF_3络合物为催化剂,1,4-丁二醇为共催化剂.找到了溶液聚合和本体聚合适宜的工艺条件.在此条件下,环齐聚物的生成量为8％左右.找到了方便易行的纯化方法,通过纯化可把聚醚的环齐聚物含量降到1％以下,有效地提高了产品的质量。     

非马尔科夫多值噪声驱动下系统的平均第一通过时间

本文把Weiss等发展的计算平均第一通过时间(简称MFPT)的新方法推广到由非马尔科夫多值噪声驱动的过程.为了简化,本文只限于研究矩形分布这一情形.通过一个重要实例,对MFPT进行了具体计算,并得到了显式解析表示.     

再议网络图的逻辑同构变换

图对同一个逻辑问题可有不同的表达方式，在必要时需进行逻辑同构变换＾［１］。本文对“节点型网络向箭线型网络的逻辑风构变换”的研究进行了补充，增加了三种情况的图开有子模，进一步谁了逐节生长法的通用性。同时，提出了箭线型网络向节点型网络逆向变换的算法；逐线收缩法。     

Effects of vitamin B12 on plasma melatonin rhythm in humans: increased light sensitivity phase-advances the circadian clock?

Vitamin B12 (methylcobalamin) was administered orally (3 mg/day) to 9 healthy subjects for 4 weeks. Nocturnal melatonin levels after exposure to bright light (ca. 2500 lx) were determined, as well as the levels of plasma melatonin over 24 h. The timing of sleep was also recorded. Vitamin B12 was given blind to the subjects and crossed over with placebo. We found that the 24-h melatonin rhythm was significantly phase-advanced (1.1 h) in the vitamin B12 trial as compared with that in the placebo trial. In addition, the 24-h mean of plasma melatonin level was much lower in the vitamin B12 trial than with the placebo. Furthermore, the nocturnal melatonin levels during bright light exposure were significantly lower in the vitamin B12 trial than with the placebo. On the other hand, vitamin B12 did not affect the timing of sleep. These findings raise the possibility that vitamin B12 phase-advances the human circadian rhythm by increasing the light sensitivity of the circadian clock.     

用付里叶—菲涅耳全息图做光学相减和光学微分

提出一种进行光学相减和学学微分的新方法,对离焦象与准确象的差即为物体图象的二阶微分做了证明.实验结果与理论分析吻合     

Retrograde transport of endocytosed Shiga toxin to the endoplasmic reticulum.

Shiga toxin and some other protein toxins that act on targets in the cytosol have previously been shown to enter the trans-Golgi network. Transport by this route may be necessary for translocation of the toxin to the cytosol and for intoxication, but it is not known whether the enzymatically active part of the toxins actually enters the cytosol from the trans-Golgi network. It has been suggested that such toxins are transported in a retrograde manner to the endoplasmic reticulum and that translocation occurs in this organelle, but retrograde transport of endocytosed material beyond the trans-Golgi network has never been demonstrated. Here we show that in butyric acid-treated A431 cells endocytosed Shiga toxin is not only transported to the trans-Golgi network, but also to all Golgi stacks, to the endoplasmic reticulum and to the nuclear envelope. Furthermore, butyric acid sensitizes the cells to Shiga toxin, which is consistent with the possibility that retrograde transport is required for translocation of the toxin to the cytosol.     

存储过程在ASP/ADO中的应用

使用存储过程可以优化对数据库的访问，本文详细介绍存储过程在ASP／ADO中的应用，并通过一个实例说明如何在ASP页面中使用存储过程。