数学概论讲演（1943～2004）

本书作者B．埃克曼是瑞士当代著名数学家，以研究拓扑、微分几何、同调代数和群论而著称。他毕业于瑞士苏黎世著名学府EHT（联邦技术学院），后来曾在瑞士洛桑大学及美国普林斯顿高等研究所任教和从事科研工作，1948年起任EHT的数学教授，     

基于前馈补偿器的无轴承同步磁阻电机解耦控制

建立了无轴承同步磁阻电机径向悬浮力及转矩部分的数学模型,其电磁转矩和径向悬浮力之间以及径向悬浮力自身在两轴方向上存在相互耦合.针对无轴承同步磁阻电机的强耦合、非线性特性,采用磁场定向控制难以直接进行解耦控制,采用基于前馈补偿器的解耦控制策略,成功解除了上述变量间的耦合关系.采用前馈补偿解耦得到的控制系统结构简单,实现方便.仿真研究表明,该解耦方法可实现无轴承同步磁阻电机稳定悬浮运行,能实现上述变量间的完全动态解耦,同时电机具有较好的转矩和调速性能.     

[Co(Ⅲ)(三元胺)(二元胺)Cl]2+型配合物的研究进展

在前人对[Co(Ⅲ)(三元胺)(二元胺)Cl]型配合物研究的基础上,结合作者的研究工作,对这类配合物的目前的研究现状、研究方法进行了归纳,并提出了一些正在开展或拟开展的研究。     

Overexpression of N-cadherin is correlated with metastasis and worse survival in colorectal cancer patients

N-cadherin is related to the progression and metastases of several solid carcinomas. However, it was still unclear whether N-cadherin is overexpressed in colorectal malignant tumors that have stronger malignant tendency. In this study, we used immunohistochemistry to detect the expression patterns of N-cadherin in both the primary tumors and their normal mucosa tissues of 120 patients with colorectal cancer. We revealed that N-cadherin was expressed in 78.3% (94/120) of colorectal tumor tissues and in only 9.2% (11/120) of paired distant normal mucosa tissues with a significant difference (P=0.000). The low, moderate, and high expression of N-cadherin protein was 42.5%, 30.8%, and 26.7%, respectively. N-cadherin overexpression was associated with advanced TNM stage, lymph nodes metastasis and distant metastasis (P<0.05). Patients with N-cadherin overexpressed showed the obvious lower overall survival rate than those with moderate and low expression, and patients with low expression had a better survival rate than those with moderate and high expression (P<0.05). In conclusion, high N-cadherin expression may lead to tumor aggressiveness and metastatic potential in colorectal cancer, and may prove to be a possible prognostic factor.     

大陆与台湾高校学前特殊教育人才培养模式比较

本文对大陆与台湾地区高校学前特殊教育人才培养模式进行分析,论述了两地学前特殊教育课程设置的特点,在此基础上进行比较,并提出台湾地区高校学前特殊教育人才培养模式的可借鉴之处。     

基于方向链码的书空手势识别

介绍了Leapmotion交互设备的结构及运行机制,在该设备产生的交互空间中的x-y平面上定义8个方向的方向链码,通过该设备检测手指在空间中的位置变化,确定手指的移动方向,并结合方向链码定义动态书空手势。为了消除手指在空间运动的不稳定性而导致的噪声干扰,将手指在平面上的位置信息映射到计算机屏幕上显示出手指的移动轨迹,对轨迹进行分段处理,根据分段的比重提取主要移动方向描述输入手势,采用顺序匹配算法对输入手势与模板手势进行匹配,识别输入的书空手势。     

模同态在直和上的分解

环上的（左或右）模在表成子模的直和时,其上的模同态可以分解为子模之间的模同态和。当环是域时,这种分解性质可作为子空间或直和的刻画。     

Preparation of InSb nanocrystals embedded in SiO2 thin films

InSb nanocrystals embedded in SiO₂ thin films were prepared by rf magnetron cosputtering technique. THe observation by transmission electron microscope showed that InSb nanocrystals dispersed uniformly in SiO₂ matrices. InSb nanocrystals with different sizes can be obtained by changing the annealing condition. The average size of InSb nanocrystals depended on annealing temperature and time, but not on the t_1/3 rules. X-ray photoelectron spectroscopy and X-ray diffraction were also applied to the analyses of the composite thin films.     

采用金属阳极溶解法合成纳米MgO前驱体

以金属镁作为“牺牲”阳极,在乙酰丙酮醇溶液中,采用电化学溶解金属镁一步制备了纳米MgO前驱体Mg(OEt)2-x(acac)x,Mg(OBu)2-x(acac)x[acac为乙酰丙酮基]．通过傅里叶变换红外光谱(FT-IR)、拉曼光谱(Raman spectrum)对其进行了表征．可讨论了影响电合成镁醇盐配合物的关键因素．实验表明,温度控制在40～50℃,导电盐Bu4NBr浓度为0．04～0．05mol／L较为适合,同时防止阳极钝化,有利于提高电合成效率。     

Lymphotactin enhances the in-vitro immune efficacy of dendritoma formed by dendritic cells and mouse hepatocellular carcinoma cells

Objective: To investigate the in-vitro antitumor immune responses of dendritoma formed by mouse hepatocellular carcinoma (HCC) cells and lymphotactin (Lptn) gene modified dendritic cells (DCs). Method: DCs prepared from mouse bone marrow were genetically modified by lymphotactin adenovirus, and fused with H22 cells by polyethylene glycol (PEG). RT-PCR and ELISA were employed to identify lymphotactin expression at mRNA and protein level. Cell phenotypes and fusion efficiency was detected by FACS. The stimulatory effect of DC on T cells was detected by mixed lymphocyte reaction. The cytotoxicity activity against H22 cells was assayed by LDH method. Results: Lymphotactin could be efficiently expressed by DCLptn/H22 hybridoma. DCLptn/H22 cells could induce potent T cell proliferation effect and generate strong cytotoxic T lymphocyte (CTL) reaction against allogenic H22 cells. Conclusion: Lymphotactin genetic modification could enhance the in vitro immune activity of the dendritoma.