Separators for Lithium Ion Batteries

1 Results A separator for rechargeable batteries is a microporous membrane placed between electrodes of opposite polarity, keeping them apart to prevent electrical short circuits and at the same time allowing rapid transport of lithium ions that are needed to complete the circuit during the passage of current in an electrochemical cell, and thus plays a key role in determining the performance of the lithium ion battery. Here provides a comprehensive overview of various types of separators for lithium io...     

二维拓扑超导体拓扑数的计算

研究了具有自旋轨道耦合的二维晶格模型的的拓扑结构.\\ 平庸与非平庸拓扑区域以能带的能隙关闭为界.\\ 首先将KITAEV 的Majorana 拓扑数$M(H)$ 推广到二维情况,在动量空间计算了二维晶格的Majorana 拓扑数和相图.\\ 由$M(H)=-1$确定的非平庸拓扑区与用TKNN宇称确定的非平庸拓扑区完全一致,证明Majorana 拓扑数与TKNN 宇称是一致的.\\ 但Majorana 拓扑数比TKNN宇称的计算简单.\\ 文章还计算了有限宽无穷长带的能带和波函数.\\ 如果参数取在非平庸参数区则长带的能带显示 Majorana零能模,而且波函数分布在长带的边界上.\\ 如果参数取在平庸参数区,则能带不受拓扑保护;系统不出现零能态,或者出现偶数个零能态.     

介孔α-MnO2/蒙脱石复合材料对罗丹明B的催化降解性能

摘要：本论文以高锰酸钾和硫酸锰为原料,修饰蒙脱石为载体,通过水热法制备介孔-MnO2和介孔-MnO2-蒙脱石复合材料.采用X射线粉末衍射(XRD)、扫描电镜(SEM)、及比表面(BET)等方法对制备材料进行表征.以介孔-MnO2和介孔-MnO2-蒙脱石复合材料为催化剂,以初始浓度为100mg/L的罗丹明B为模拟废水,研究了罗丹明B的降解动力学,结果表明,介孔-MnO2-蒙脱石复合材料对罗丹明B的降解效果是介孔-MnO2的2倍.并且三次重复实验后,-MnO2-蒙脱石复合材料的催化性基本没有损失,说明-MnO2-蒙脱石复合材料的稳定性.     

深蓝色OLED材料均三嗪衍生物的合成与表征

简要本文合成了三种均三嗪衍生物TPTPA、TITPA和TCTPA,并利用其在电学和光学特性,将其作为发光/电子传输层材料制作有机电致发光器件(OLEDs). 其中,TCTPA具有比TPTPA和TITPA更高的荧光量子产率(PL). 从TPTPA到TCTPA其最低未占据轨道能级(LUMO)逐渐减少,这说明了TCTPA由于其共轭结构的增大,具有更高的电子迁移率. 以TCTPA作为发光/电子传输层的无掺杂OLED器件,在20V时亮度达到最大值2 612 cd/m2,其最大亮度效率和最大功率效率分别为1.72 cd/A 和1.35 lm/W. 将Ir(PPy)3、PVK、PBD等化合物,分别掺杂到以TCTPA为发光/电子传输层的OLED器件中,均展示出比无掺杂器件更高的亮度及效率.     

一种低复杂度逐次逼近型数模转换器设计与仿真

为了降低模数转换器复杂度和功耗,基于低复杂度电容阵列DAC设计了一种低功耗逐次逼近型模数转换器（SAR ADC）. 该结构中,电容阵列DAC每个电容只有两种参考电平选择,降低逻辑控制电路和电容驱动电路的复杂度,电容阵列DAC最低位电容参与转换,使需要的总单位电容数量相比单调结构减少一半;比较器采用两级动态结构,降低功耗;移位寄存器采用动态锁存电路结构,降低功耗和减少误码;电容驱动电路采用CMOS反相器结构,减少晶体管数量. SAR ADC电路仿真结果显示：在1.0 V电源电压和采样速率为100 kHz 时,SAR ADC功耗为0.45 W ,有效位（ENOB）为9.99 bit ,其单步转换功耗为4.4 fJ.     

Cloning and expression of cDNA for a human thromboxane A2 receptor.

Thromboxane A2 is a very unstable arachidonate metabolite, yet a potent stimulator of platelet aggregation and a constrictor of vascular and respiratory smooth muscles. It has been implicated as a mediator in diseases such as myocardial infarction, stroke and bronchial asthma. Using a stable analogue of this compound we recently purified the human platelet thromboxane A2 receptor to apparent homogeneity. Using an oligonucleotide probe corresponding to its partial amino-acid sequence, we have obtained a complementary DNA clone encoding this receptor from human placenta and a partial clone from cultured human megakaryocytic leukaemia cells. The placenta cDNA encodes a protein of 343 amino acids with seven putative transmembrane domains. The protein expressed in COS-7 cells binds drugs with affinities identical to those of the platelet receptor, and that in Xenopus oocytes opens Ca2(+)-activated Cl- channel on agonist stimulation. Northern blot analysis and nucleotide sequences of the two clones suggest that an identical species of the thromboxane A2 receptor is present in platelets and vascular tissues. This first report on the molecular structure of an eicosanoid receptor will promote the molecular pharmacology and pathophysiology of these bioactive compounds.     

Genome-wide association analysis in primary sclerosing cholangitis identifies two non-HLA susceptibility loci

Primary sclerosing cholangitis (PSC) is a chronic bile duct disease affecting 2.4-7.5% of individuals with inflammatory bowel disease. We performed a genome-wide association analysis of 2,466,182 SNPs in 715 individuals with PSC and 2,962 controls, followed by replication in 1,025 PSC cases and 2,174 controls. We detected non-HLA associations at rs3197999 in MST1 and rs6720394 near BCL2L11 (combined P = 1.1 × 10?1? and P = 4.1 × 10??, respectively).     

Effects of thyrotrophic-releasing hormone (TRH) on thermoregulation in the rat

Summary At ambient temperatures (T_a) of both 8 and 22°C, intraventricular administration of TRH (10–80 g) produced a dose-dependent hypothermia in rats. The hypothermia was due to both decreased metabolic heat production and cutaneous vasodilatation. In contrast, at 30°C T_a, TRH increased metabolic heat production (due to behavioral excitation) and led to hyperthermia.This work was supported by the grants from the National Science Council of Republic of China and the Pjing-Ling Neurological Foundation (VGH, Taipei, Taiwan).