基于网络监视器的网络实验设计

本文介绍了网络监视器的基本功能,提供了用网络监视器设计网络实验的基本方法及已开设的网络实验项目,并通过一个具体实例分析实验结果.     

E-mail信息加密技术与算法分析

分析了E-mail信息网络传递的现状，使用E-mail存在的不安全隐患，提出了一种有效地保护E-mail信息安全传递的加密技术和算法。     

一类非精确线搜索下的混合共轭梯度算法

给出了一类在非精确线搜索下的混合共轭梯度算法,对李荣生提出的NCG算法进行了改进,在算法的迭代过程中,保持了迭代方向的下降性,在较弱的条件下证明了全局收敛性,数值试验表明该算法是相当有效的.     

纳米化处理对锆合金腐蚀性能的影响

讨论了表面纳米化处理对锆合金的抗腐蚀性能的影响.对于具有表面纳米及超细晶组织的锆合金来说,其腐蚀性能的影响因素不仅包括晶粒尺度,还包括合金成分、氧化膜的组织与性质、热处理工艺方法、表面状态、反应堆水化学等.而上述因素中的任何一个因素变化,都会导致材料腐蚀性能改变.因此对于锆合金纳米化后腐蚀性能研究,应从全方位考虑,从而对锆合金的腐蚀性能有一个全方位的认识.     

MCM-48介孔分子筛的合成研究

本文以正硅酸乙酯(TEOS)为硅源,以十六烷基三甲基溴化胺(CTAB)为模板剂,用传统的水热合成法合成介孔分子筛MCM-48,探索模板剂、水、氢氧化钠等不同因素对合成MCM-48介孔分子筛的影响。在较低模板剂用量的条件下,合成了介孔分子筛,用XRD对MCM-48介孔分子筛进行了结构表征。     

铌酸锂波长转换器中参量过程的研究

考虑到抽运损耗、走离和非线性等效应,对周期性极化LiNbO3光波导的级联二阶非线性效应波长转换中的参量过程进行了理论研究及数值分析,结果表明:抽运光、信号光和差频光之间会发生周期性能量振荡,最优波导长度随抽运光和信号光的注入功率的增大而减小;脉冲抽运时因走离效应的影响,能量振荡相对较弱.利用这种能量振荡特性,通过调整注入控制光功率大小在实现信号波长转换的同时可以实现脉冲整形.     

微细电解加工实验研究

针对微细电解加工中脉冲电源技术,电解液成分配比,以及加工间隙检测、控制等问题开展了微细电解加工技术的试验研究工作.首先讨论了微细电解加工的工艺特点和主要技术步骤,然后利用高频窄脉冲电源进行了加工实验.通过实验现象和实验结果的研究分析,提出了改进加工实验的方案,通过实验证明了本方案和技术路线的可行性,并获得了很好的微结构加工试验结果.通过微细电解加工实验的研究,为微细电解加工的生产应用提供了依据,显示出微细电解加工方法在金属零件微制造方面有着广阔的应用前景.     

An Authentication Method of Distinguishing Proxy＇s Secure Accident Responsibility

The publish/subscribe （pub/sub） paradigm has asynchronous, loosely-coupled and many-to-many communication properties and is widely used in the application of large-scale distributed computing environment. There is the problem that is mutual trustable between network proxies in terms of pub/sub systems and the problem which is hardly to distinguish accident responsibility while the accident happens in Kerberos based on symmetrical encryption algorithm. A proxy identity authentication algorithm based on RSA encryption is proposed to solve the problem of mutual trust between proxies, and the security of the messages is guaranteed through certificate delegation. The algorithm can distinguish accident responsibility. The feasibility analysis, security analysis and efficiency analysis of the algorithm are carried out.     

微观sdIBM—2＋2q.p中核^138Ce正宇称能谱结构

基于微观相互作用玻色子-费米子形式体系，构造出唯象IBM-1 2q.p模型的微观实现，微观sdIBM-2 2q.p方案，应用它成功地再现了^138Ce核的基态带，β带，γ带和部分高自旋态能谱，给出了近来实验上观察到该核与单粒子态共存的集体性结构的一种解释，模型给出了一个玻色子从退耕，拆散，占据闯入轨道到参加顺排所需要的最小能量值，计算出的概观信息表明：一个d玻色子的突然拆对不会引起核状态突变；核子顺排释放的能量有利于其它玻色子再拆对；^138Ce核的高自旋态属子两准质子转动带。     

Mutations in genes encoding melanosomal proteins cause pigmentary glaucoma in DBA/2J mice.

Pigmentary glaucoma is a significant cause of human blindness. Abnormally liberated iris pigment and cell debris enter the ocular drainage structures, leading to increased intraocular pressure (IOP) and glaucoma. DBA/2J (D2) mice develop a form of pigmentary glaucoma involving iris pigment dispersion (IPD) and iris stromal atrophy (ISA). Using high-resolution mapping techniques, sequencing and functional genetic tests, we show that IPD and ISA result from mutations in related genes encoding melanosomal proteins. IPD is caused by a premature stop codon mutation in the Gpnmb (GpnmbR150X) gene, as proved by the occurrence of IPD only in D2 mice that are homozygous with respect to GpnmbR150X; otherwise, similar D2 mice that are not homozygous for GpnmbR150X do not develop IPD. ISA is caused by the recessive Tyrp1b mutant allele and rescued by the transgenic introduction of wildtype Tyrp1. We hypothesize that IPD and ISA alter melanosomes, allowing toxic intermediates of pigment production to leak from melanosomes, causing iris disease and subsequent pigmentary glaucoma. This is supported by the rescue of IPD and ISA in D2 eyes with substantially decreased pigment production. These data indicate that pigment production and mutant melanosomal protein genes may contribute to human pigmentary glaucoma. The fact that hypopigmentation profoundly alleviates the D2 disease indicates that therapeutic strategies designed to decrease pigment production may be beneficial in human pigmentary glaucoma.