激光复混沌系统构建及其点乘函数投影同步

提出了一种新的激光复混沌系统及其点乘函数投影同步方法。首先,以四维激光实超混沌系统模型为基础,构建了一个新的激光复混沌系统,基于常规动力学分析方法和MATLAB仿真软件研究了系统的耗散性、平衡点、Lyapunov指数谱、相图、分叉图等基本动力学特性。研究结果表明,该系统动力学行为丰富,在一定参数下具有蝴蝶结型混沌、超混沌吸引子,非常适用于混沌加密领域。进一步,以向量点积运算为基础,定义了一种新的点乘函数投影同步方式,基于滑模控制方法实现了蝴蝶结型激光复混沌系统的点乘函数投影同步。数值仿真验证了理论分析的正确性和有效性。     

贵州早寒武世黑色页岩地球化学特征及其意义

贵州早寒武世早期普遍存在一套黑色页岩。通过对麻江羊跳、铜仁、遵义中南、遵义松林小竹、天柱大公塘剖面实地考察与地球化学分析,认为贵州早寒武世黑色页岩是在缺氧环境下,热水沉积为主导,同时受陆源强烈影响,是海水、生物和地球深部物质共同作用下形成的.     

统一碳税和差异化碳税下的再制造绩效评价

分别在无碳税、统一碳税和差异化碳税三种政策下,构建了垄断制造商的利润最大化决策模型,研究实施不同碳税政策对制造商的再制造生产决策、碳排放和社会福利的影响.研究表明:相比于无碳税政策,政府征收碳税减少了制造商的利润和碳排放,在一定条件下有利于提高社会福利;实施碳税政策有利于鼓励更多类型的制造商参与再制造,并且差异化碳税比...     

白腐菌对焦化废水中吲哚的降解及其机理

选用白腐菌BP降解吲哚,研究了白腐菌在不同培养基中对两种浓度吲哚的降解过程和机理,以及吲哚的降解与白腐菌漆酶酶活、生物量、培养基pH值的关系.结果显示不同培养基中白腐菌可去除99%以上的吲哚;吲哚去除率与白腐菌漆酶活率具有较好的相关性,白腐菌漆酶活率达到最高时,可去除97%以上的吲哚;高浓度吲哚会抑制白腐菌的生长,同时也激发了白腐菌漆酶的产生,秸秆滤出液能促进白腐菌的生长和漆酶活力的增长;白腐菌BP最适pH值为6~7,pH值在5~8之间对吲哚都具有较强的降解能力;在HPLC谱图3~6 min之间出现的新物质可能有氧化吲哚和靛红.     

导弹武器模拟训练考评系统设计

为提高某导弹模拟训练系统中评估的客观性与准确性,确立以准确性、熟练度和操作人员状态为主的评价指标体系,构建适当的数学模型计算各子指标分值,将子指标定量化,解决由考官进行评判时存在的主观性强、标准不统一等缺点。利用专家系统,对考评过程中需要的专业知识和评价规则进行存储,实现了评分过程的自动化。针对层次分析法(analytic hierarchy process, AHP)需要多次赋值保证一致性和群组决策特征根法(group eigenvalue method, GEM)结构模糊的缺点,提出了基于AHP和GEM融合算法的权重设置方案,利用AHP-GEM算法对各子指标得分进行加权评估,最终形成了一套合理有效的模拟训练评估系统。最后,通过与考官人工评分的方法进行比较,验证了所提评分系统的合理性和准确性。     

基于中继选择的星地协作传输系统性能分析

由于星地混合协作网络（hybrid satellite-terrestrial cooperative network,HSTCN）能显著提升卫星通信的服务质量,近年来受到了国内外学者的广泛关注,分析了基于中继选择的星地协作传输系统的性能。首先,在卫星和地面用户之间存在直达链路,并且所有中继节点在采用门限判断译码转发协议辅助卫星通信的情况下,得到用户端经过最大比合并后的输出信噪比（signal-to-noise ratio,SNR）表达式。其次,针对卫星信道服从阴影莱斯分布,而地面信道服从Nakagami-m分布,推导出随机选择和最佳选择两种中继选择策略下系统的中断概率闭合表达式。接着,进一步得到高信噪比条件下中断概率的近似表达式,为分析各种参数对系统性能的影响提供了更加便捷的方法。最后,计算机仿真不仅验证了理论推导的正确性,而且定量分析了协作传输带来的好处,从而为HSTCN技术的实际应用提供理论依据。     

城乡结合部道路交叉口多目标信号配时优化模型

随着我国城镇化进程的加快,城乡结合部道路交通问题日益显著。为提高城乡结合部道路交叉口的交通运行效率、环境效益和交通安全,提出信号配时多目标优化方法,综合考虑延误、通行能力、停车次数、车辆尾气排放等因素构建模型,运用遗传算法进行求解。以某典型交叉口为例进行分析,给出改进设计方案并进行交通仿真评价。研究结果表明,通过多目标信号配时优化可以缓解城乡结合部道路交叉口交通拥堵和污染物排放的问题,具有较好的交通信号控制效果。     

Nuclear Norm Subspace System Identification and Its Application on a Stochastic Model of Plague

The discrete-time model of plague is deduced by zero-order holder based on the continuoustime model. Due to the existence of stochastic disturbances, the stochastic model is given corresponding to the discrete-time model. The state estimation and noise reduction of the stochastic model are achieved by designing Kalman filter. Nuclear norm minimization is to structure the low-rank matrix approximation instead of the singular value decomposition in the process of subspace system identification. According to the plague data from the World Health Organization, the system matrices and noise intensity of the model are identified. Simulations are carried out to show the higher approximation capability of the proposed method.     

Fabrication of solenoid-type microinductor with different magnetic core schemes by MEMS technique

0 IntroductionThere has been a great demand on micro DC/DCconverter for the applications of the portable electronicproducts such as mobile communication product CDMA(Code Division Multiple Access) ,portable notebookcom-puter ,microprocessor ,digital camcorder andso on.Mag-netic components such as inductors and transformers arethe essential components in constructingthe power supplytransformer ,filter , oscillator , amplifier and micro DC/DCconverter ,andthe miniaturization of inductor an…     

Lysosomal chymotrypsin B potentiates apoptosis via cleavage of Bid

We have reported that chymotrypsin B (CtrB) is not just a digestive enzyme but is also stored in lysosomes. Herein, we demonstrated a broad distribution of CtrB and explored the involvement of CtrB in apoptosis. Exposure of RH-35 cells to H₂O₂ or palmitate induced the redistribution of lysosomal CtrB into the cytoplasm as a result of lysosomal membrane permeabilization (LMP). Suppression of CtrB significantly blocked apoptosis, while overexpression of CtrB sensitized apoptosis markedly. CtrB could cleave Bid under neutral conditions. In RH-35 cells with Bid silenced, apoptosis induced by CtrB protein was attenuated, suggesting that CtrB mediates apoptosis of RH-35 cells mainly through processing Bid. Our data also suggest that LMP occurs earlier than mitochondrial outer membrane permeabilization; Bid activation initiated by caspase-8 might be reinforced by CtrB in consequence of LMP, which causes a positive feedback loop leading to the accumulation of tBid, and results in lysosome- and mitochondrion-dependent apoptosis.