鲫幼鱼易钓性与标准代谢及形态特征的关联

【目的】考察鲤科(Cyprinidae)鱼类的标准代谢率(Standard metabolic rate)及形态参数与易钓性是否存在关联。【方法】以广布种鲤科鱼类鲫(Carassius auratus)幼鱼为实验对象,在(25±0.5)℃条件下测定体质量相近、体格健康的40尾幼鱼的标准代谢率,随后在实验室环境中连续进行5d垂钓,每日垂钓20尾幼鱼即停止当日垂钓活动。【结果】1)第1d被钓出幼鱼的标准代谢率高于未被钓出幼鱼,具有统计学意义(p<0.05);随后连续4d中被钓出幼鱼与未被钓出幼鱼的标准代谢率之间的差异均不具有统计学意义。另外,每日被钓出幼鱼与未被钓出幼鱼的体质量之间的差异也无统计学意义,但被钓出幼鱼的体长在第3d和第5d大于未被钓出幼鱼,且具有统计学意义(p<0.05),并且被钓出幼鱼的肥满度在第3d高于未钓出组,具有统计学意义(p<0.05)。2)在5d的连续垂钓中,被钓上5次的幼鱼有3尾,被钓上1~4次的幼鱼分别为7,7,6,10尾,仅7尾幼鱼从未被钓出,单因素方差分析显示不同被钓出频次的幼鱼的标准代谢率之间差异不具有统计学意义。3)对5d中均被钓出的幼鱼的钓出顺序进行分析,发现它们每日被钓排序在整体上保持一致,而幼鱼的平均被钓次序号与它们的标准代谢率和形态参数均无统计学意义上的相关性。【结论】鲫幼鱼的首次被钓经历并不影响自身再次被钓;在体质量相近条件下,鲫的易钓性与外部形态(如体质量、体长或肥满度等)以及维持代谢有关。
     

散热器进流系数算法的研究

详细剖析了跨越式单管采暖系统中，水流过散热器的温降对进流系数的影响，并针对现在所采用的计算方法的不合理之处，建立了一种新的数学模型来计算散热器的进流系数。     

Lipoprotein lipase activator deficiency in very low density lipoproteins in rat nephrotic syndrome.

Marked urinary loss of lipoprotein lipase activator in experimental rat nephrotic syndrome may be partly responsible for its deficiency in plasma very low density lipoproteins.     

近五年来阅读障碍研究的新进展

近五年对阅读障碍的研究主要有:对以前研究问题的不断深入与探讨;出现了新的研究方法,研究手段也更多、更新了(ERP及眼动仪的运用);对汉语阅读障碍研究的重视;在阅读障碍的遗传学研究中出现一个新的内容,即阅读障碍基因SNP s(单核苷酸多态性)研究的出现与发展;将阅读障碍与其他的非器质性病变的病症联系起来进行研究,其中最热的一种就是将阅读障碍与注意了缺陷多动症(ADHD)联系起来研究。     

论“宇宙情怀”

人类跨入二十一世纪,精神蚀化,"心"灾丛生,很有必要在文学创作中树立一种神圣的"宇宙情怀"精神,借以慰藉人类躁郁的灵魂。宇宙情怀包涵文学境界精神、诗意超验品度、深在人文向度三个层面的文化内容,其终极关怀指向一种纯精神的诗性坚守。     

Inhibition of E coli ATPase activity by a troponin component, TN-I, and by mitochondrial ATPase inhibitor.

The enzymic activity of Mg2+- or Ca2+-stimulated ATPase from Escherichia coli was inhibited by one of the troponin components, TN-I, and by mitochondrial ATPase inhibitor (F1-inhibitor). The inhibitory ability of component TN-I against Mg2+-stimulated AtPase activity was lost after digestion of component TN-I with trypsin. The Mg2+-stimulated ATPase activity inhibited by component TN-I was completely restored by the addition of another troponin component TN-C.     

Interaction of c-Abl and p73alpha and their collaboration to induce apoptosis.

c-Abl, a non-receptor tyrosine kinase, is activated by agents that damage DNA. This activation results in either arrest of the cell cycle in phase G1 or apoptotic cell death, both of which are dependent on the kinase activity of c-Abl. p73, a member of the p53 family of tumour-suppressor proteins, can also induce apoptosis. Here we show that the apoptotic activity of p73alpha requires the presence of functional, kinase-competent c-Abl. Furthermore, p73 and c-Abl can associate with each other, andthis binding is mediated by a PxxP motif in p73 and the SH3 domain of c-Abl. We find that p73 is a substrate of the c-Abl kinase and that the ability of c-Abl to phosphorylate p73 is markedly increased by gamma-irradiation. Moreover, p73 is phosphorylated in vivo in response to ionizing radiation. These findings define a pro-apoptotic signalling pathway involving p73 and c-Abl.     

A radiation hybrid map of the rat genome containing 5,255 markers.

A whole-genome radiation hybrid (RH) panel was used to construct a high-resolution map of the rat genome based on microsatellite and gene markers. These include 3,019 new microsatellite markers described here for the first time and 1,714 microsatellite markers with known genetic locations, allowing comparison and integration of maps from different sources. A robust RH framework map containing 1,030 positions ordered with odds of at least 1,000:1 has been defined as a tool for mapping these markers, and for future RH mapping in the rat. More than 500 genes which have been mapped in mouse and/or human were localized with respect to the rat RH framework, allowing the construction of detailed rat-mouse and rat-human comparative maps and illustrating the power of the RH approach for comparative mapping.