任务可拆分项目调度问题

经典资源受限的项目调度问题的前提之一是任务不可拆分,即每个任务只能被一次执行,中间不能停顿.但是在企业实际的项目调度中,许多任务是允许被拆分成若干次执行的.针对任务可拆分的项目调度问题提出了总项目工期最短的数学模型,该模型在任务较多、任务工期较长或时间粒度小时解空间很大,不利于精确求解.提出了一种结合邻域搜索方法的混合遗传算法求解该模型.给出了算法的编码方案、解码规则、适值函数、选择方法、交叉算子和变异算子的实现方法.最后通过算例验证了算法的有效性,列出了任务不能拆分和任务可拆分两种情况下算例最优解的甘特图.     

晶格失配对超晶格法向导热系数影响的分子动力学模拟

采用非平衡态分子动力学方法模拟了超晶格的法向导热系数随周期长度的变化关系.模拟结果表明,在晶格匹配的超晶格中,当周期长度同声子平均自由程相当时,超晶格导热系数将出现最小值.而对于具有4%晶格失配的超晶格模拟结果却表明,超晶格导热系数随周期长度的增大而单调上升.这一研究结果表明,材料的晶格失配是大多数实验研究中没有发现超晶格最小导热系数的主要原因.     

氧化铁纳米颗粒固定化溶藻菌的除藻作用

为了探讨氧化铁纳米颗粒固定化溶藻菌后对铜绿微囊藻的溶藻作用和降解微囊藻毒素LR(MC-LR)的作用,从太湖水体中分离获得一株属于芽孢杆菌属的溶藻菌,将其与氧化铁纳米颗粒固定化后,以普通Fe2O3作为实验对照,测定其溶藻作用和降解MC-LR作用.以激光粒度仪测定氧化铁纳米颗粒与溶藻菌两者单独和同时存在时的粒径大小及粒径分布,以获得氧化铁纳米颗粒固定化溶藻菌的证据.分析结果表明:在太湖水体中分离获得的一株芽孢杆菌具有较强的溶藻和降解MC-LR的作用,作用24 h后的溶藻率和MC-LR降解率分别达到44.3%和20.5%.同时加入氧化铁纳米颗粒后,溶藻率达到53.0%,MC-LR的降解率达到33.0%.激光粒度仪检测发现,氧化铁纳米颗粒与细菌混合后的颗粒物的平均直径增加到氧化铁纳米颗粒的12.3倍,同时氧化铁纳米颗粒和细菌单独存在时的粒径分布峰消失.氧化铁纳米颗粒是一种具有良好性能的生物固定化材料,可能通过其特有的途径有效地固定化溶藻菌,并增强溶藻菌的溶藻和降解藻毒素作用.     

基于在线神经网络的自适应控制器的设计与应用

针对线性控制系统自适应性较弱的缺点,在其基础上设计了一种基于在线神经网络的自适应控制器．该控制器根据被控对象和参考模型之间的误差调整网络权值,在线抵消被控对象的未建模动态特性和不确定因素．文中以某小卫星姿态控制系统为例,对所提出的自适应控制器进行设计和仿真．结果表明,神经网络以很高的精度跟踪和在线补偿不确定项,改善了线性控制系统的动态性能．     

激光标刻的复杂区域填充算法

为减少填充线段和提高激光加工效率,对复杂区域的填充算法进行了研究．采用延长某些岛屿边界将包含岛屿的区域变为单连通域;然后用最长边延长法和最长边平行截取法将单连通域分解为简单凸多边形,简单凸多边形填充方法是先找到凸多边形所有边对应的高最小的那条边,平行该边进行填充,最后对填充线段的连接顺序进行优化处理．与传统的固定方向扫描填充相比,采用本算法可使复杂区域和实心文字的激光标刻效率提高20％～40％．     

分年龄段的互惠模型的行波解

讨论了一类分年龄段的生物种群互惠模型的行波解存在性问题．在一般带时滞的互惠Lotka—Volterra模型基础上,考虑了年龄段和空间非局部等因素对反应扩散方程行波解存在性的影响．构造了一对合适的上下解,利用单调迭代方法证明了模型的两个平衡点之间行波解的存在性,进一步丰富了单调方法的内容．     

Incorporating textual and management factors into financial distress prediction: A comparative study of machine learning methods

Financial distress prediction (FDP) has been widely considered as a promising approach to reducing financial losses. While financial information comprises the traditional factors involved in FDP, nonfinancial factors have also been examined in recent studies. In light of this, the purpose of this study is to explore the integrated factors and multiple models that can improve the predictive performance of FDP models. This study proposes an FDP framework to reveal the financial distress features of listed Chinese companies, incorporating financial, management, and textual factors, and evaluating the prediction performance of multiple models in different time spans. To develop this framework, this study employs the wrapper-based feature selection method to extract valuable features, and then constructs multiple single classifiers, ensemble classifiers, and deep learning models in order to predict financial distress. The experiment results indicate that management and textual factors can supplement traditional financial factors in FDP, especially textual ones. This study also discovers that integrated factors collected 4 years prior to the predicted benchmark year enable a more accurate prediction, and the ensemble classifiers and deep learning models developed can achieve satisfactory FDP performance. This study makes a novel contribution as it expands the predictive factors of financial distress and provides new findings that can have important implications for providing early warning signals of financial risk.     

大跨度钢桁梁桥纵向附加力分析与研究

文章以桥上无缝线路梁轨相互作用原理为基础,建立以轨道、桥梁、支座、墩台和基础为整体结构的附加纵向力计算有限元模型,依据"对号入座"法则编制了计算钢桁梁桥上无缝线路的程序。讨论了伸缩调节器设置与否以及设置位置的不同对纵向附加力的影响。     

颗粒增强聚合物复合材料损伤模型

研究了颗粒增强聚合物复合材料的力学行为,研究得知:材料屈服、裂纹形核、扩展与贯通直至最终断裂是一逐渐劣化过程,而损伤理论正是这一劣化过程的良好描述.通过假设自由能和耗散势函数,导出了损伤演化规律,与实验比较,模型和试验结果基本符合.进一步采用改进的Dugdale模型,重点研究损伤对GB/PPO复合材料宏观裂纹起裂的影响,通过建立损伤模型来描述材料的劣化行为和裂纹扩展,结果表明,损伤区域严重影响裂尖的性能,材料损伤对宏观裂纹起裂影响不可忽略.     

The urokinase receptor and integrins in cancer progression

Enhanced levels of expression of urokinase receptor (uPAR) and certain integrins have been linked to cancer cell progression. This has classically been attributed to matrix degradation via the activation of the urokinase (uPA)/plasmin system and modulation of cell motility and survival through integrin engagement. More recently, uPAR has been shown to play multiple roles independent of protease activity. Specifically, uPAR has been shown to be intimately involved in the regulation of cell adhesion, migration and proliferation in part through interactions with other membrane partners, including integrins. The goal of this review is to summarize recent insights in the function of uPAR/integrin interactions, to provide a framework for understanding the importance of these interactions in the context of cancer, and to highlight its potential as a target for therapeutic intervention.