A trusted mobile payment environment based on trusted computing and virtualization technology

In this paper, we propose a trusted mobile payment environment （TMPE） based on trusted computing and virtualization technology. There are a normal operating system （OS） and a trusted OS （TOS） in TMPE. We store the image of TOS in a memory card to hinder tampering. The integrity of TOS is protected by means of a trusted platform module （TPM）. TOS can only be updated through a trusted third party. In addition, virtualization technology is applied to isolate TOS from normal OS. Users complete ordinary affairs in normal OS and security-sensitive affairs in TOS. TMPE can offer users a highly protected environment for mobile payment. Moreover, TMPE has good compatibility in different hardware architectures of mobile platforms. As the evaluation shows, TMPE satisfies the requirement of mobile payment well.     

大学生的中国文化和美国文化象征及其翻译调查

通过对大学生进行中国文化和美国文化象征对比调查,分析大学生认同的美国文化象征及其汉语翻译情况,以及大学生认同的中国文化及其英语翻译情况,了解文科大学生和理工科大学生在选取中国文化象征和美国文化象征上的差异。调查表明大学生能准确地表达美国文化象征及其汉语翻译,而表达中国文化象征及其英语翻译时却不尽如人意。通过在大学英语教学课程目标中明确提出中国文化在英语教学中的作为和地位,在教学内容上增加英语介绍中国文化的相关题材和信息,在教学方法上运用多种策略激发学生学习中国文化,以及教师提高中国文化素养等措施,将中国文化融入大学英语教学,使文化教学和语言教学有机融合。     

双材料反平面非对称界面端研究

研究了双材料非对称的反平面界面端的问题,通过构造应力函数并结合复变方法,在给定的自由边界条件下,得到一组四阶齐次线性方程组,从而求解出双材料反平面非对称界面端的特征方程.在固定角度θ1情况下,选取一组材料参数为验证特征值λ的算例,并通过变换角度θ2、φj(j=1,2)的值来研究特征值θ1的变换规律,给出了不同情形下的相应图形.     

基于EGM2008地球重力场模型-Griddata函数组合模型的似大地水准面精化探讨

介绍了EGM2008地球重力场模型以及MATLAB提供的Griddata函数,结合工程实例,探讨基于EGM2008地球重力场模型-Griddata函数组合模型对似大地水准面进行精化,在Griddata函数提供的4种不同插值方法下进行分析比较,并根据4种不同插值的特点进行改进,最后得出基于EGM2008地球重力场模型-Griddata函数法中利用四格点样条插值法替代3次多项式插值法中凸包点法的拟合效果最好,具有很好的实际推广意义。     

山区中小城市公路客运企业安全投入与产出绩效分析

以秦岭山区的某3家市级客运企业为例,通过改进C-D生产函数理论,建立客运安全投入绩效模型,利用计算数据分析安全投入对产出绩效的影响大小,为企业调整安全投入的分配比例提供科学依据.同时研究发现,山区中小城市的客运市场不同于大城市,现阶段政府的安全投入比较稳定,虽然安全产出增幅较小,但对客运安全仍然起到引导和干预作用.     

Efficient and stable inverted perovskite solar cells enabled by inhibition of self-aggregation of fullerene electron-transporting compounds

Fullerene-based electron-transporting layers (ETLs) significantly influence the defect passivation and device performance of inverted perovskite solar cells (PS...     

Human exposome and biomarker database for soil pollutants at typical sites of industrial contamination

Rapid economic development and industrialization have left many risk sites around the world with significant or potential soil contamination due to industrial p...     

A Simple Linear Regression Approach to Modeling and Forecasting Mortality Rates

Observing that a sequence of negative logarithms of 1‐year survival probabilities displays a linear relationship with the sequence of corresponding terms with a time lag of a certain number of years, we propose a simple linear regression to model and forecast mortality rates. Our model assuming the linearity between two mortality sequences with a time lag each other does not need to formulate the time trends of mortality rates across ages for mortality prediction. Moreover, the parameters of our model for a given age depend on the mortality rates for that age only. Therefore, whether the span of the study ages with the age included is widened or shortened will not affect the results of mortality fitting and forecasting for that age. In the empirical testing, the regression results using the mortality data for the UK, USA and Japan show a satisfactory goodness of fit, which convinces us of the appropriateness of the linear assumption. Empirical illustrations further show that our model's performances of fitting and forecasting mortality rates are quite satisfactory compared with the existing well‐known mortality models. Copyright © 2015 John Wiley & Sons, Ltd.     

Absence of a robust innate immune response in rat neurons facilitates persistent infection of Borna disease virus in neuronal tissue

Borna disease virus (BDV) persistently infects neurons of the central nervous system of various hosts, including rats. Since type I IFN-mediated antiviral response efficiently blocks BDV replication in primary rat embryo fibroblasts, it has been speculated that BDV is not effectively sensed by the host innate immune system in the nervous system. To test this assumption, organotypical rat hippocampal slice cultures were infected with BDV for up to 4 weeks. This resulted in the secretion of IFN and the up-regulation of IFN-stimulated genes. Using the rat Mx protein as a specific marker for IFN-induced gene expression, astrocytes and microglial cells were found to be Mx positive, whereas neurons, the major cell type in which BDV is replicating, lacked detectable levels of Mx protein. In uninfected cultures, neurons also remained Mx negative even after treatment with high concentrations of IFN-α. This non-responsiveness correlated with a lack of detectable nuclear translocation of both pSTAT1 and pSTAT2 in these cells. Consistently, neuronal dissemination of BDV was not prevented by treatment with IFN-α. These data suggest that the poor innate immune response in rat neurons renders this cell type highly susceptible to BDV infection even in the presence of exogenous IFN-α. Intriguingly, in contrast to rat neurons, IFN-α treatment of mouse neurons resulted in the up-regulation of Mx proteins and block of BDV replication, indicating species-specific differences in the type I IFN response of neurons between mice and rats.