对偶算子代数的某些结果

对偶算子代数的 X_Q,_r 性质与不变子空间问题,与算子代数的自反性和超自反性问题都有着十分密切的联系(例如见等).本文进一步研究了这类性质,得到了象遗传性定理、稠密性定理等一些结果.     

软土地基砂井加固三向固结问题的Bessel函数解

在软土地基三向固结理论研究中R·A·Barron给出了二类边界条件下三维拋物型方程混合问题的解(见[1],[2],[3]),但这个解是不准确的。本文一方面指出B·A·Barron解的问题所在,另一方面用一、二类Bessel函数求出了该问题的精确解,指明了解的适定性,给出了高级超越方程J_o(x)Y_1(kx)-J_1(kx)Y_o(x)=0当k<1时的零点求法;最后利用所得结果求出软土路基的平均固结度。     

基于神经网络的喘振早期发现

分析并指出了压缩机组防喘振控制系统的缺陷，在此基础上，针对机组发生哮喘振与机组工艺参数间的关系，建立了一个基于神经网络模型喘振预警系统，以帮助现场机组操作人员及早发现喘振。     

CIMS环境中的一个知识型数据库

本文分析了ＣＩＭＳ环境中软件及硬件管理方面的问题，设计了一个搜集各种知识的知识型数据库，并以注塑模具为研究对象，研究并搜集模具设计、制造过程中生成及应用的知识．     

小型低温强制循环热水采暖系统中的安全设施

本文阐述小型低温强剖循环热水采暖系统中预防炉水汽化、循环水的热膨胀、水击以及排污、放气等安全装置的原理、结构和操作方法。     

平面n次系统奇点(Ⅱ)

本文进一步研究平面n次系统奇点的性质,证明了n次系统的m重奇点是由m个初等奇点汇合而成的。讨论了有限远奇点与无限远奇点的关系。     

Functional consequences of treating turkey liver fructose-1,6-bisphosphatase with penicillin G

Summary Treatment of turkey liver fructose-1,6-bisphosphatase with penicillin G progressively inactivated the enzyme and desensitized the enzyme toward high substrate inhibition. The treatment also led to reduced sensitivity to AMP inhibition and the loss of cooperative interaction among AMP-binding sites. These altered properties were not reversed by dialysis, but were prevented when treatment with penicillin G was performed in the presence of substrate.This work was in part supported by grant RR-8006 from the General Research Branch, Division of Research Resources, NIH (USA) and in part by Faculty Research Grant from Southern School of Pharmacy, Mercer University, Atlanta (Georgia, USA).     

弹性钻柱两个特殊点的意义及计算方法

通过分析钻柱的应力和应变状态,对钻柱的中性点和零轴向力点给出了新的定义和计算方法,并阐述了稳定力和屈曲力(通称为虚轴向力)的概念。导出了处在垂直井眼中的复合钻柱的有关计算公式。文中还与原有的研究结论进行了对比分析,并提出了不同意见。本文以钻柱为研究对象,所得的结果可以推广应用到套管柱及油管柱上。     

Cellular responses to mild heat stress

Since its discovery in 1962 by Ritossa, the heat shock response has been extensively studied by a number of investigators to understand the molecular mechanism underlying the cellular response to heat stress. The most well characterized heat shock response is induction of the heat shock proteins that function as molecular chaperones and exert cell cycle regulatory and anti-apoptotic activities. While most investigators have focused their studies on the toxic effects of heat stress in organisms such as severe heat stress-induced cell cycle arrest and apoptosis, the cellular response to fever-ranged mild heat stress has been rather underestimated. However, the cellular response to mild heat stress is likely to be more important in a physiological sense than that to severe heat stress because the body temperature of homeothermic animals increases by only 1–2°C during febrile diseases. Here we provide information that mild heat stress does have some beneficial role in organisms via positively regulating cell proliferation and differentiation, and immune response in mammalian cells.Received 14 May 2004; received after revision 2 August 2004; accepted 16 August 2004