Musielak-Orlicz-Sobolev空间关于Luxemburg范数的端点

本文着重讨论Musielak-Orlicz-Sobolev空间端点的性质,得到Musielak-Orlicz-Sobolev空间关于Luxemburg范数端点的充分条件.     

A multiple intelligent agent system for credit risk prediction via an optimization of localized generalization error with diversity

Company bankruptcies cost billions of dollars in losses to banks each year. Thus credit risk prediction is a critical part of a bank＇s loan approval decision process. Traditional financial models for credit risk prediction are no longer adequate for describing today＇s complex relationship between the financial health and potential bankruptcy of a company. In this work, a multiple classifier system （embedded in a multiple intelligent agent system） is proposed to predict the financial health of a company. In our model, each individual agent （classifier） makes a prediction on the likelihood of credit risk based on only partial information of the company. Each of the agents is an expert, but has limited knowledge （represented by features） about the company. The decisions of all agents are combined together to form a final credit risk prediction. Experiments show that our model out-performs other existing methods using the benchmarking Compustat American Corporations dataset.     

Watching cartoons activates the medial prefrontal cortex in children

The medial prefrontal cortex (MPFC) of human adults is involved in attributing mental states to real human agents but not to virtual artificial characters. This study examined whether such differential MPFC activity can be observed in children who are more fascinated by cartoons than adults. We measured brain activity using functional magnetic resonance imaging (fMRI) while 10-year-old children watched movie and cartoon clips, simulating real and virtual visual worlds, respectively. We showed neuroimaging evidence that, in contrast to adults, the MPFC of children was activated when perceiving both human agents and artificial characters in coherent visual events. Our findings suggest that, around the age of 10 years, the MPFC activity in children is different from that in adults in that it can be spontaneously activated by non-human agents in a virtual visual world.     

薄圆盘CsI(Tl)晶体闪烁特性蒙特卡罗模拟

利用蒙特卡罗方法,模拟99mTc 140 keV γ射线在圆盘状CsI(Tl)晶体中的闪烁过程及不同厚度不同包装下闪烁光的空间分布,得出点光源模型对于晶体出光平面上闪烁光强分布是适用的结果.模拟表明,CsI(Tl)与铝(Al)之间界面为粗糙时要比光滑时位置分辨更好;闪烁中心离出光面越近,位置分布越窄;晶体厚度为7 mm时光输出最大,光输出半高宽约为7.3 mm.     

信息技术课程教学质量保证体系模式的研究

介绍了信息技术课程的特点,设计了高校信息技术课程教学质量保证体系的模式,讨论了该模式的自适应性、可操作性和可评价性。     

企业危机管理、组织学习和知识管理的战略整合

首先介绍了企业危机管理的动态发展模型,分析后认为组织学习和知识的供给是成功的重要因素;其次,提出为了确保正确的思维方式和学习效果的落实,应该采用三环学习的模式;最后,建立了在三环学习模式下的知识管理执行过程和战略,以保证企业危机管理的知识供给。     

大学生物理模型知识的调查分析

为了有针对性地设计培养学生物理建模能力的实验方案，对在校大学生物理模型基础知识的掌握情况，对物理模型的认知程度以及对待进行物理建模能力训练的态度等方面进行了广泛调查和认真分析，通过定量统计和定性分析，总结归纳了6个方面的现状。     

简单树结构存储系统数据分块标准及其证明

为解决传统磁盘阵列技术受系统总线与设备通道制约的问题,采用树结构和单元控制器构建了新型的存储系统.在树结构存储系统的基础上,通过对简单树结构存储系统数据分块方式的定义、推论,以及冲突系数的证明,给出了简单树结构存储系统的数据分块标准.结果证明:简单树结构存储系统数据分块标准能最大程度降低存储系统有效冲突系数,提升存储系统性能.     

球面巨型网格结构的构成、支承方式与力学模型

提出了一种新的结构形式——球面巨型网壳结构。文中重点研究了肋环型和凯威特型球面巨型网格主体结构的具体构成形式、几何拓扑关系,总结了相应的形体控制参数;对子结构的形式及其与主体结构的连接进行了分析;描述了自动成形过程。使用Auto- CAD环境下的Visual C 二次开发工具编制了相应的自动成形程序;并对其主体结构支承方式与力学分析模型进行了探讨．本文工作为对该结构体系开展全面系统的力学性能研究提供了良好的前处理基础．     

Telomere dysfunction and evolution of intestinal carcinoma in mice and humans

Telomerase activation is a common feature of advanced human cancers and facilitates the malignant transformation of cultured human cells and in mice. These experimental observations are in accord with the presence of robust telomerase activity in more advanced stages of human colorectal carcinogenesis. However, the occurrence of colon carcinomas in telomerase RNA (Terc)-null, p53-mutant mice has revealed complex interactions between telomere dynamics, checkpoint responses and carcinogenesis. We therefore sought to determine whether telomere dysfunction exerts differential effects on cancer initiation versus progression of mouse and human intestinal neoplasia. In successive generations of ApcMin Terc-/- mice, progressive telomere dysfunction led to an increase in initiated lesions (microscopic adenomas), yet a significant decline in the multiplicity and size of macroscopic adenomas. That telomere dysfunction also contributes to human colorectal carcinogenesis is supported by the appearance of anaphase bridges (a correlate of telomere dysfunction) at the adenoma-early carcinoma transition, a transition recognized for marked chromosomal instability. Together, these data are consistent with a model in which telomere dysfunction promotes the chromosomal instability that drives early carcinogenesis, while telomerase activation restores genomic stability to a level permissive for tumor progression. We propose that early and transient telomere dysfunction is a major mechanism underlying chromosomal instability of human cancer.