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Sheila Bhattacharya N. C. Bhattacharya K. K. Nanda 《Cellular and molecular life sciences : CMLS》1976,32(10):1301-1303
Summary Exogenously supplied DNA and RNA hastened root initiation and also increased the formation of roots on hypocotyl cuttings ofImpatients balsamina with intact apex and cotyledons. IAA appreciably increased the nucleic acid-caused enhancement in root formation. In combination with lowe concentrations of nucleic acids, it event stimulated the growth of roots as well as of hypocotyls. Higher concentrations of nucleic acids were, however, toxic.The research has been partly financed by a grant from the United States Department of Agriculture. One of us (SB) is thankful to the Deparmtent of Atomic Engery of the Government of India for financial assistance. 相似文献
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Bessant DA Payne AM Mitton KP Wang QL Swain PK Plant C Bird AC Zack DJ Swaroop A Bhattacharya SS 《Nature genetics》1999,21(4):355-356
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Enzymes of non-agglutinable vibrios and their possible role in the development of enterotoxic factor
B. Guhathakurta S. Bhattacharya G. C. Datta A. K. Ghosh 《Cellular and molecular life sciences : CMLS》1973,29(7):903-904
Résumé Sachant que le facteur entérotoxique peut être développé dans les vibrions non-agglutinables par transfer animal, nous avons déterminé les activités enzymatiques (mucinase, protéase, lécithinase) de ces vibrions. Après ce transfert l'activité lécithinasique a augmenté, et cette activité est semblable à celle d'un virus (V. cholerae). Nous supposons que l'augmentation du facteur entérotoxique est causée par celle de l'activité de la lécithinase.
Thanks are due to Dr.A. Mondal for help and Mr.Manzar Alam for his secretarial assistance. 相似文献
Thanks are due to Dr.A. Mondal for help and Mr.Manzar Alam for his secretarial assistance. 相似文献
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OPA1, encoding a dynamin-related GTPase, is mutated in autosomal dominant optic atrophy linked to chromosome 3q28 总被引:22,自引:0,他引:22
Alexander C Votruba M Pesch UE Thiselton DL Mayer S Moore A Rodriguez M Kellner U Leo-Kottler B Auburger G Bhattacharya SS Wissinger B 《Nature genetics》2000,26(2):211-215
Autosomal dominant optic atrophy (ADOA) is the most prevalent hereditary optic neuropathy resulting in progressive loss of visual acuity, centrocoecal scotoma and bilateral temporal atrophy of the optic nerve with an onset within the first two decades of life. The predominant locus for this disorder (OPA1; MIM 165500) has been mapped to a 1.4-cM interval on chromosome 3q28-q29 flanked by markers D3S3669 and D3S3562 (ref. 3). We established a PAC contig covering the entire OPA1 candidate region of approximately 1 Mb and a sequence skimming approach allowed us to identify a gene encoding a polypeptide of 960 amino acids with homology to dynamin-related GTPases. The gene comprises 28 coding exons and spans more than 40 kb of genomic sequence. Upon sequence analysis, we identified mutations in seven independent families with ADOA. The mutations include missense and nonsense alterations, deletions and insertions, which all segregate with the disease in these families. Because most mutations probably represent null alleles, dominant inheritance of the disease may result from haploinsufficiency of OPA1. OPA1 is widely expressed and is most abundant in the retina. The presence of consensus signal peptide sequences suggests that the product of the gene OPA1 is targeted to mitochondria and may exert its function in mitochondrial biogenesis and stabilization of mitochondrial membrane integrity. 相似文献