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991.
The mystery of female beauty. 总被引:8,自引:0,他引:8
992.
P. Milner V. Ralevic A. M. Hopwood E. Fehér J. Lincoln K. A. Kirkpatrick G. Burnstock 《Cellular and molecular life sciences : CMLS》1989,45(2):121-125
Substance P and choline acetyltransferase have been localised in a small proportion of endothelial cells of rat coronary arteries using electron microscopic immunocytochemistry. During a hypoxic period of 1 min, coronary vasodilatation was produced in the Langendorff heart preparation and increased levels of substance P and acetylcholine were released into the perfusate. The possibility that these substances are released from endothelial cells during hypoxia and contribute to the hyperaemic response is discussed. 相似文献
993.
The in situ activity of 3 beta-hydroxysteroid dehydrogenase was detected in clustered cells which showed steroid-producing morphology, within the capsule of the paraaortic lymph node. In light and electron microscopic studies, the positive reaction products were detected on intracapsular cell clusters. This result indicates that these unique cells may have a steroid secreting function within the lymph nodes. 相似文献
994.
995.
Summary In gerbil adrenal cortex the activity of intramitochondrial NADP-linked isocitric dehydrogenase (IDH) is up to 10-fold greater than the NAD-linked IDH. The NADP-IDH, apparent Km 0.58 mM, Vmax 280 nmoles/min/mg mitochondrial protein, appears to be the major source of reducing equivalents to support adrenal mitochondrial steroid 11B- and 19-hydroxylation in this species. 相似文献
996.
997.
A series of microiontophoretic and VTA stimulation experiments, conducted in intact, GBR-12909-treated, alpha-methylparatyrosine-depleted or 6-hydroxydopamine-denervated rats, provide suggestive evidence for the existence of a presynaptic, positive feedback mechanism triggered by dopamine reuptake and favoring the release of this transmitter in the anterior cingulate cortex. 相似文献
998.
Restoration of normal function in genetically defective myotubes by spontaneous fusion with fibroblasts 总被引:5,自引:0,他引:5
Muscular dysgenesis in mice is a genetic disease of skeletal muscle caused by the recessive mutation mdg. Muscle fibres in affected mice are paralysed because of the failure of excitation-contraction coupling. Unlike normal myotubes in primary culture, dysgenic myotubes do not contract, either spontaneously or in response to electrical stimulation. The deficiency results from mutation of the gene for the skeletal muscle dihydropyridine receptor, an essential sarcolemmal component both of excitation-contraction coupling and of the slow calcium-ion channel. It has recently been shown that the addition of fibroblasts from normal (but not dysgenic) mice to cultures of dysgenic myotubes can restore spontaneous contractions in a small fraction of these myotubes, but the mechanism for this 'rescue' was not determined. In principle, if fibroblast nuclei were able to incorporate into myotubes, such nuclei could then supply the missing muscle-specific gene product. We have now investigated this possibility using nuclear, cytoplasmic and plasmalemmal markers. We report that the rescue to contractile ability in genetically paralysed dysgenic muscle is mediated by the previously unrecognized ability of fibroblasts to fuse spontaneously with developing myotubes. 相似文献
999.
Dose-dependent relaxations were induced by dopamine in human pulmonary arteries that had been contracted with prostaglandin F2 alpha without alpha-adrenergic blocking agents. The dopamine-induced relaxation was inhibited by haloperidol and fluphenazine, but not by domperidone, suggesting that this relaxation was mediated via DA1 receptors. 相似文献
1000.
The tunneling of macroscopic object is one of the most fascinating phenomena in condensed matter physics. During the last decade, the problem of quantum tunneling of magnetization in nanometer-scale magnets has attracted a great deal of theoretical and experimental interest. A review of recent theoretical research of the macroscopic quantum phenomena in nanometer-scale single-domain magnets is presented in this paper. It includes macroscopic quantum tunneling (MQT) and coherence (MQC) in single-domain magnetic particles, the topological phase interference or spin-parity effects, and tunneling of magnetization in an arbitrarily directed magnetic field. The general formulas are shown to evaluate the tunneling rate and the tunneling level splitting for single-domain AFM particles. A nontrivial generalization of Kramers degeneracy for double-well system is provided to coherently spin tunneling for spin systems with m-fold rotational symmetry. The effects induced by the external magnetic field have been studied, where the field is along the easy, medium, hard axis, or arbitrary direction. 相似文献