开孔板加劲型压型钢板加固混凝土界面黏结-滑移机理

为研究开孔板(perfobond leiste,PBL)加劲型压型钢板加固混凝土界面黏结-滑移机理,首先设计了3组试件进行推出试验,分析了其破坏形态、极限荷载值和滑移量,然后在试验基础上建立了有限元模型,分析了PBL连接件极限荷载值的影响参数.研究结果表明:PBL连接件破坏分为弹性阶段、塑性阶段和破坏阶段3个阶段,试件破坏时混凝土出现贯通裂缝,底部混凝土剥落,试件为混凝土剪切破坏,且为延性破坏.影响PBL连接件极限承载力的参数主要有贯穿钢筋、黏结摩擦力、混凝土强度、开孔直径和PBL厚度,其中贯穿钢筋影响最明显,有贯穿钢筋的试件比无贯穿钢筋的试件极限承载力提高了约73.8％,贯穿钢筋也可以提高极限滑移量,增加延性.黏结摩擦力通常作为安全储备,混凝土强度和开孔直径的增加也可显著提高极限承载力,而PBL厚度影响不显著,开孔钢板强度对极限承载力基本无影响.     

在开放边界条件下Nagel-Schreckenberg交通流模型的功率谱

在开放性边界条件下,从功率谱的角度讨论Nagel-Schreckenberg随机交通流模型的相变,数值模拟结果充分地反映了交通流从自由流动相到拥堵相的相变,且在相变点上可以观察到1/f噪声信号.     

用于回归估计的支持向量机

介绍机器学习的表示方式,分析和比较机器学习中经验风险最小化原则和结构风险最小化原则,引出用于回归估计的支持向量机,并用数学方式阐述其基本思想,讨论支持向量机技术发展中存在的主要问题.     

盐胁迫下小麦新品系89122的抗氧化酶活性和内源ABA含量变化的研究

利用花粉管通道法获得的转基因耐盐小麦新品系89122和其受体“陇春13”,在盐胁迫处理下,测定盐胁迫后89122和“陇春13”无性细胞系的抗氧化酶活性的变化和脱落酸的变化,结果表明,89122较其受体“陇春13”能维持较高的SOD,POD,CAT活性,ABA含量明显高于受体。     

利用共焦成像原理实现微米级的三维轮廓测量

以普通光学成像透镜取代显微物镜,以针孔阵列(500×500)取代单针孔,实现了全场并行共焦测量.在数据处理上,采用了内插值三维重构算法,以及二次曲面拟合的系统误差校正算法,可以在较大的采样间距下获取较高的测量精度.实验表明,系统的最大横向测量范围为5mm×5mm,横向测量精度约为13μm,纵向测量精度约为5μm,因此可大大提高测量视场及测量速度.     

Sequence and analysis of chromosome 1 of the plant Arabidopsis thaliana

The genome of the flowering plant Arabidopsis thaliana has five chromosomes. Here we report the sequence of the largest, chromosome 1, in two contigs of around 14.2 and 14.6 megabases. The contigs extend from the telomeres to the centromeric borders, regions rich in transposons, retrotransposons and repetitive elements such as the 180-base-pair repeat. The chromosome represents 25% of the genome and contains about 6,850 open reading frames, 236 transfer RNAs (tRNAs) and 12 small nuclear RNAs. There are two clusters of tRNA genes at different places on the chromosome. One consists of 27 tRNA(Pro) genes and the other contains 27 tandem repeats of tRNA(Tyr)-tRNA(Tyr)-tRNA(Ser) genes. Chromosome 1 contains about 300 gene families with clustered duplications. There are also many repeat elements, representing 8% of the sequence.     

Reversible electromechanical characteristics of carbon nanotubes under local-probe manipulation

The effects of mechanical deformation on the electrical properties of carbon nanotubes are of interest given the practical potential of nanotubes in electromechanical devices, and they have been studied using both theoretical and experimental approaches. One recent experiment used the tip of an atomic force microscope (AFM) to manipulate multi-walled nanotubes, revealing that changes in the sample resistance were small unless the nanotubes fractured or the metal-tube contacts were perturbed. But it remains unclear how mechanical deformation affects the intrinsic electrical properties of nanotubes. Here we report an experimental and theoretical elucidation of the electromechanical characteristics of individual single-walled carbon nanotubes (SWNTs) under local-probe manipulation. We use AFM tips to deflect suspended SWNTs reversibly, without changing the contact resistance; in situ electrical measurements reveal that the conductance of an SWNT sample can be reduced by two orders of magnitude when deformed by an AFM tip. Our tight-binding simulations indicate that this effect is owing to the formation of local sp3 bonds caused by the mechanical pushing action of the tip.     

The gene encoding 5-lipoxygenase activating protein confers risk of myocardial infarction and stroke

We mapped a gene predisposing to myocardial infarction to a locus on chromosome 13q12-13. A four-marker single-nucleotide polymorphism (SNP) haplotype in this locus spanning the gene ALOX5AP encoding 5-lipoxygenase activating protein (FLAP) is associated with a two times greater risk of myocardial infarction in Iceland. This haplotype also confers almost two times greater risk of stroke. Another ALOX5AP haplotype is associated with myocardial infarction in individuals from the UK. Stimulated neutrophils from individuals with myocardial infarction produce more leukotriene B4, a key product in the 5-lipoxygenase pathway, than do neutrophils from controls, and this difference is largely attributed to cells from males who carry the at-risk haplotype. We conclude that variants of ALOX5AP are involved in the pathogenesis of both myocardial infarction and stroke by increasing leukotriene production and inflammation in the arterial wall.