高等植物花粉母细胞间细胞融合现象的研究

细胞融合（ｃｙｔｏｍｉｘｉｓ）是一种重要的细胞生物学现象,在植物的变异和物种进化方面具有重要的意义。从细胞融合现象的的规律、起因、机理和生物学意义４个方面,简要地介绍了笔者近半个世纪以来在这一领域的限苦探索和取得的重要成果。     

一个含参数的强三点积分不等式

利用一种Gr櫣ss型不等式去推出一个新的含参数的强三点积分不等式,精确地给出使不等式中等式成立的函数,由此统一处理并且改进了由MMatic、XLCheng、NUjevic、CEMPearce等人提出的改进的Tay lorc、Ostrowski Gr櫣ss及中点的一些积分不等式。     

中国鞘蛾研究（鳞翅目：鞘蛾科）：线鞘蛾组研究及二新种记述

本文报道鞘蛾科线鞘蛾组（Coleophoraamellivoragroup）分布于中国的4个种,其中包括2个新种1个中国新记录种：齿线鞘蛾（新种）Coleophoraapicidentatasp．nov．分布新疆（乌鲁木齐）;长刺线鞘蛾（新种）Coleophoralongispinasp．nov．分布内蒙古（锡林浩特）;弯茎线鞘蛾ColeophorapandionellaBaldizzone新记录于内蒙古（锡林浩特、赤峰）和黑龙江（黑河）,国外分布俄罗斯（东西伯利亚）;云南线鞘蛾ColeophorayunnanicaBaldizzone分布于云南（丽江）．文中提供了线鞘蛾组中国已知种检索表和新种的外生殖器特征图．模式标本保存在南开大学生物系．     

拓扑分子格上的粘接定理

本文建立了拓扑分子格上的粘接定理,使得经典拓扑学的粘接引理成为其特殊情形。     

钾在水煤气耐硫变换催化剂中的作用

应用微反、XRD和TPR技术,对水煤气耐硫变换Co-Mo-K—O/γ-Al_2O_3催化剂的性能进行了研究。结果表明,原料气中H_2S的浓度在100～500ppm的实验范围内,催化剂中钾的存在能抑制在操作温度大于250℃后产生的反硫化,显著地提高催化剂的活性。K_2O的含量约在10～12%时为佳。XRD观察到,在硫化态的催化剂上,钾以KHSO_3的形式存在。硫化态的催化剂在TPR谱上有两个被H_2还原峰,表明了低温峰(T_m在200～250℃)为硫化物的还原,高温峰(550℃)则对应于氧化物的还原。     

高稳定度稳压电源(GWE-1型)的研制

本文简要介绍了GwE-1型稳压电源的设计原理,已研制出样机,电势差计使用此电源比它用干电池要节约数千元。此电源值得推广应用。     

颈椎数字减影图像三维运动检测系统的探讨

利用微机对数字减影成像ＤＳＡ（ＤｉｇｉｔａｌＳｕｂｔｒａｃｔｉｏｎ）系统的颈椎（模拟）图像进行处理,建立了一种颈椎非接触式三维运动检测系统．该系统拟采用ＤＳＡ对病人颈椎进行正、侧位照影,根据ＤＳＡ的两路ＰＡＬ制式输出,经过预处理、识别和ＤＬＴ测量方法计算,得出被检测颈椎每块椎骨中心点的空间坐标,从而达到确立完成某动作时病人每块椎骨间的相对位移、相对转角等运动参数的目的．     

关于Banach空间中一类非线性积分微分方程(Ⅱ)

以非紧致测度为工具,继续讨论了一类非线性积分微分方程解的存在性和解集对参数的上半连续依赖性,并在序Banach空间中证明了极值解的存在性及相应的比较定理.     

A nuclear-mitochondrial DNA interaction affecting hearing impairment in mice

The pathophysiologic pathways and clinical expression of mitochondrial DNA (mtDNA) mutations are not well understood. This is mainly the result of the heteroplasmic nature of most pathogenic mtDNA mutations and of the absence of clinically relevant animal models with mtDNA mutations. mtDNA mutations predisposing to hearing impairment in humans are generally homoplasmic, yet some individuals with these mutations have severe hearing loss, whereas their maternal relatives with the identical mtDNA mutation have normal hearing. Epidemiologic, biochemical and genetic data indicate that nuclear genes are often the main determinants of these differences in phenotype. To identify a mouse model for maternally inherited hearing loss, we screened reciprocal backcrosses of three inbred mouse strains, A/J, NOD/LtJ and SKH2/J, with age-related hearing loss (AHL). In the (A/J x CAST/Ei) x A/J backcross, mtDNA derived from the A/J strain exerted a significant detrimental effect on hearing when compared with mtDNA from the CAST/Ei strain. This effect was not seen in the (NOD/LtJ x CAST/Ei) x NOD/LtJ and (SKH2/J x CAST/Ei) x SKH2/J backcrosses. Genotyping revealed that this effect was seen only in mice homozygous for the A/J allele at the Ahl locus on mouse chromosome 10. Sequencing of the mitochondrial genome in the three inbred strains revealed a single nucleotide insertion in the tRNA-Arg gene (mt-Tr) as the probable mediator of the mitochondrial effect. This is the first mouse model with a naturally occurring mtDNA mutation affecting a clinical phenotype, and it provides an experimental model to dissect the pathophysiologic processes connecting mtDNA mutations to hearing loss.