凸集上的非线性算子的固有元及固有值

本文利用拓扑度研究集上的具边界条件的非线性集压缩算子的不动点。固有元及固有值的存在性,推广了[3,5,6]中的相应结论,部分回答了[4]的一个猜测。     

β－氨基－α－羟乙基二茂铁及其过渡金属配合物的合成与表征

用β－氨基－α－羟已基二茂铁与二价钴、镍、铜、锌、镉和汞的氯化物及醋酸铜在乙醇－水中反应，合成了七种新的配合物．探索出了适宜的反应条件，经元素分析，确定了新配合物的组成为：Ｌ２ＭＸ２·ｎＨ２Ｏ（Ｌ＝Ｆｃ─ＣＨＣＨ２ＮＨ２；Ｍ＝Ｃｏ２＋、Ｎｉ２＋、Ｃｕ２＋、Ｚｎ２＋、Ｃｄ２＋、Ｈｇ２＋，ｘ＝Ｃｌ－，ＣＨ３ＣＯＯ－；ｎ＝０，２，４）．通过红外光谱和核磁共振氢谱．对这些配合物的结构进行了表征．     

弱内向型非锥映象的不动点及固有元

本文给出了满足弱内向型边界条件的非锥多值映象的正固有元及正不动点存在的一些充分条件。     

Effect of Ag addition on the thermal stability and glass-forming ability of Zr<Subscript>35</Subscript>Ti<Subscript>30</Subscript>Cu<Subscript>7.5</Subscript>Be<Subscript>27.5</Subscript> bulk metallic glass

The thermal stability and glass forming ability (GFA) of Zr35-xTi30Cu7.5Be27.5Agx (x = 0-10) alloys were studied by X-ray diffraction (XRD), differential scanning calorimetry (DSC) and ultrasonic techniques. We found that the addition of 1 at.% Ag can considerably enhance the GFA as indicated by an increase in the critical glass dimension from 15 mm in the Zr35Ti30Cu7.5Be27.5 alloy to 20 mm in the Zr34Ti30Cu7.5Be27.5Ag1 alloy. However, with the addition of more Ag the supercooled liquid region (△Tx) and y parameter (defined as Tx/(Tg+Tl)) drastically decreased from 155 K and 0.436 to 76 K and 0.363, respectively, resulting in a decrease in the GFA. Additionally, the elastic constant (the ratio of shear modulus to bulk modulus or Poisson’s ratio) was also used as a gauge to evaluate the GFA in Zr35-xTi30Cu7.5Be27.5Agx alloys.     

A software reliability growth model for component-based software incorporating debugging delay and imperfect debugging

In view of the problems and the weaknesses of component-based software(CBS) reliability modeling and analysis,and a lack of consideration for real debugging circumstance of integration testing,a CBS reliability process analysis model is proposed incorporating debugging time delay,imperfect debugging and limited debugging resources.CBS integration testing is formulated as a multiqueue multichannel and finite server queuing model(MMFSQM) to illustrate fault detection process(FDP) and fault correction process(FCP).A unified FCP is sketched,given debugging delay,the diversities of faults processing and the limitations of debugging resources.Furthermore,the impacts of imperfect debugging on fault detection and correction are explicitly elaborated,and the expressions of the cumulative number of fault detected and corrected are illustrated.Finally,the results of numerical experiments verify the effectiveness and rationality of the proposed model.By comparison,the proposed model is superior to the other models.The proposed model is closer to real CBS testing process and facilitates software engineer's quantitatively analyzing,measuring and predicting CBS reliability.     

基于分形参数的B超图像纹理分析

利用离散分形布朗随机场模型对Ｂ超图像的分布特征进行顾分析和讨论。鉴于一般的图像不是完全的分形体,文中给出了计算分维数的尺度范围公式,根据上述模型和尺度范围公式,对正常肝脏和有癌变肝脏的两组Ｂ超图像的分维数进行了计算,结果表明在未考虑尺度范围时两组图像的分维数差别不大;而考虑了尺度范围时两幅图像的分维数就有明显的差别,即正常肝脏图像的分维数大于有癌变肝脏图像的分维数。     

添加剂对SnO2气敏特性的影响

氧化锡气敏材料的电导率以及对气体的选择性由氧空位的形成和氧化过程共同完成,氧空位浓度越大,气敏效应越明显。根据氧化锡的这一气敏机理,本文研究了添加剂Ｓｂ２Ｏ３和Ｐｄｃｌ２对氧化锡气敏材料电导率及选择性的影响,分析了其对氧化锡气敏特性的作用机理。     

曲线网格生成调节因子选取研究

指出了常用Ｔｈｏｍｐｓｏｎ方法中调节因子对生成网格曲线所存在的问题,从另一角度发出,借助于自然符合椭圆型函数关系的某些物理现象,导出了一种新的调节因子表达式,并着重探讨分析了该因子的适用条件,长江三峡工程河段,第二松花江曾通河段实际水域的应用检验表明,该调节因子对强弯河段及多连通水域网格布置具有良好的效果。     

温肾醒脑方对血管性痴呆大鼠脑组织Nrf2-ARE信号通路的影响

为探讨温肾醒脑方(WXD)对血管性痴呆(vascular dementia,VD)大鼠认知功能的改善作用及对Nrf2-ARE信号通路的影响,将70只雄性SD大鼠随机分成正常组、假手术组、模型组、脑复康组(0.15 g/kg)、温肾醒脑方给药组(高剂量组5 g/kg、中剂量组2.5 g/kg、低剂量组1.25 g/kg),每组10只动物,采用双侧颈总动脉永久性结扎法建立VD模型,给予温肾醒脑方灌胃治疗后,Morris水迷宫测试治疗后VD大鼠认知功能的改善情况,检测脑组织中超氧化物歧化酶(SOD)、丙二醛(MDA)、谷胱甘肽过氧化物酶(GSH-Px)、一氧化氮合酶(NOS),RT-PCR检测大鼠脑组织醌NADH脱氢酶(NQO1)、血红素氧合酶1(HO-1)的mRNA水平,运用Western Blot检测Kelch样环氧氯丙烷相关蛋白-1(Keap1)、核因子E2相关因子2(Nrf2)、NQO1、HO-1蛋白水平的表达情况以及EMSA检测Nrf2与抗氧化反应元件(ARE)结合力。结果表明:与正常组比较,VD模型组大鼠逃避潜伏期明显较长,游泳总距离明显缩短,穿越站台总数明显减少(P0.01);大脑组织中MDA含量显著增加,SOD、GSH-Px和NOS的含量明显减少(P0.01);同时大脑组织中细胞核Nrf2蛋白表达降低、细胞质中Keap1蛋白表达增加,NQO1、HO-1 mRNA和蛋白表达降低(P0.01)。与VD模型组比较,脑复康和温肾醒脑方组大鼠的认知行为得到很大的提高,脑组织MDA、SOD、GSH-Px、NOS含量和脑组织病理结构得到很好的改善,Nrf2、Keap1、NQO1、HO-1在mRNA水平或蛋白水平上有一定程度改变,特别是温肾醒脑方组大鼠的改善非常显著。可见温肾醒脑方可以改善VD大鼠的认知行为,其机制可能与Nrf2-ARE抗氧化应激信号通路相关。