Destruction of virus infected cells by neutrophils and complement

The paper describes an antibody independent mechanism of cytotoxicity whereby virus infected but not uninfected cells are destroyed by the combined presence of neutrophils and complement.     

Destruction of virus infected cells by neutrophils and complement

Summary The paper describes an antibody independent mechanism of cytotoxicity whereby virus infected but not uninfected cells are destroyed by the combined presence of neutrophils and complement.Supported by A.I. 14981 and by the MRC of Canada.Acknowledgment. We thank C.J. Wust for invaluable discussions.     

Polyphenoloxidase activity in healthy maize plants and in those infected by the maize rough dwarf virus (MRDV)

Zusammenfassung Der Wirkungsgrad der Polyphenoloxydase gesunder Maispflanzen blieb während der Gesamtdauer ihres Wachstums praktisch unverändert. Er verdreifachte sich hingegen in jungen Pflanzen, die mit dem Virus der Rauhverzwergung des Maises systematisch infiziert wurden.

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This work was supported in part by a grant from the Agricultural Research Service of the United States Department of Agriculture, under Public Law 480.     

Inhibition of skin calcification (Calciphylaxis) by polyphosphates

Zusammenfassung Grahamsalz, ein langkettiges Polyphosphat, ist bei subcutaner Verabreichung imstande, eine pathologische Verkalkung der Haut (Calciphylaxe) bei der Ratte zu hemmen. Der Wirkungsmechanismus dieser Hemmung wird kurz diskutiert.     

Effect of human interferon on viral production in a cell line chronically infected by a type D retrovirus (Mason-Pfizer virus)]

Human interferon specifically inhibits the viral multiplication in a human cell line infected by a type D retrovirus, the Mason-Pfizer virus (MPV). This inhibition is less important than for type C viruses.     

Intracellular cyclic AMP in vesicular stomatitis or Senda? virus infected cells (author's transl)

In KB cells, MRC5 and adult skin fibroblasts infected by low doses of Senda? virus, intracellular cyclic AMP levels rose and fell in the first hours following infection, then remained lower than basal level during at least 2 days in KB cells and adult skin fibroblasts. When compared to other viruses or cAMP inducers previously described, this effect appeared specific of Senda? virus. Mechanisms and roles of cAMP variations are discussed. VSV-infected KB cells showed slightly decreased cAMP levels during the first hours following infection.