Responses of the resistance and capacitance vessels reflexly induced from the cardiac chambers

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Neurogenic responses of resistance and capacitance vessels

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A correlation of responses of the resistance and capacitance vessels of the intestine and kidney to changes of impulse in postganglionic nerves under pressor reflexes

Summary The responses of resistance vessels of the intestine and kidney were induced by high amplitude impulses (over 15 V), while those of capacitance vessels in these organs were induced by low amplitude impulses (15 V and lower) of postganglionic sympathetic fibres.     

Relation of the character of capacitance vessel responses in spleen and small intestine elicited by electrical stimulation of the sympathetic fibres to the level of oxygen exchange in the organs

Summary There is a correlation between character and magnitude of capacitance vessel responses and venous blood oxygen saturation under electrical stimulation of sympathetic fibres in the spleen and small intestine.     

Relation of efferent impulse activity in splenic nerve to reflexly induced reactions of resistance and capacitance vessels of spleen

Summary Constrictory responses of splenic resistance vessels arising under pressor reflexes were abolished by hexonium (2 mg/kg) as well as the high amplitude (above 15 V) impulses in sympathetic splenic nerves. Constrictory and dilatory responses of splenic capacitance vessels were preserved after administration of the same dose of hexonium and correlated as to the directivity with the changes of the low amplitude (15 V and lower) impulsation in the splenic nerve.     

Inhibition of noradrenaline release from sympathetic nerves by pentobarbital

Summary Pentobarbital concentrations of 10–100 M selectively inhibited the noradrenaline release evoked by activation of the nicotinic receptors on the terminals sympathetic nerves of the rabbit heart. Higher concentrations also decreased the noradrenaline release induced by KCl or by electrical stimulation of the nerve axons.This paper is dedicated to Professor Dr. G. Malorny on the occasion of his 65th birthday.Acknowledgment: We thank Mrs G. Thielecke for technical assistance.     

Inhibition of noradrenaline release from sympathetic nerves by pentobarbital

Pentobarbital concentrations of 10--100 micrometer selectively inhibited the noradrenaline release evoked by activation of the nicotinic receptors on the terminals sympathetic nerves of the rabbit heart. Higher concentrations also decreased the noradrenaline release induced by KCl or by electrical stimulation of the nerve axons.     

The influence of frequency of stimulation on synaptic transmission at different temperatures

Résumé Durant le refroidissement du ganglion cervical supérieur perfusé du chat, la réaction de la membrane nictitante est notablement plus sensible aux stimulations de haute que de basse fréquence. Aux températures élevées, c'est l'inverse.L'influence de la fréquence des stimulations a aussi été perceptible lors des expériences, au cours desquelles le rendement de l'acétylcholine a été déterminé. A 37°C, le rendement s'accorît sensiblement lorsqu'on accélère le rythme de stimulation. A 20°C, cependant un nombre croissant de chocs préganglionnaires n'augmente pas le rendement total de l'acétylcholine.     

The effect of prolonged sympathetic stimulation on conduit vessel diameter

Zusammenfassung Wird die Reizfrequenz 8 imp/sec nicht überschritten, vermögen mittelgrosse Arterien — ungleich den Resistenzgefässen — während 10 min andauernder Sympathicusreizung ohne Erschlaffung in anhaltender Kontraktion zu verharren. Bei Anwendung höherer Reizfrequenzen ist eine Tendenz zur Erschlaffung der Gefässmuskeln trotz andauernder Stimulation bemerkbar.-Rezeptoren-Erregung scheint zur Erschlaffung quantitativ beizutragen.     

Amylase secretion from rat parotid glands as dependent on co-operation between sympathetic and parasympathetic nerves

Summary A slow, long-lasting degeneration secretion from the parotid gland was brought about in anaesthetized rats by section of the auriculo-temporal nerve 16–19 h in advance. This parasympathetic background activity greatly increased the secretion of amylase elicited by sympathetic nerve stimulation.Supported by grants from the Swedish Medical Research Council (project nr 00539) to N.E. and from the Medical Faculty, University of Lund, to P.G.     

Responses of isolated blood vessels to acetylcholine,epinephrine, norepinephrine,and histamine

Zusammenfassung Acetylcholin verursachte in den Coronar-, Femoral- und Mesenterialarterien, Catecholamine in den Femoral- und Mesenterialgefässen Konstriktionen, in den Coronargefässen hingegen Dilatation.

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This work was completed in the Department of Physiology, University of Mississippi Medical Center, Jackson (Mississippi USA).     

Activation of sustained sympathetic vasodilatation in dog by spinal cord stimulation

Summary Electrical stimulation in lateral sites of the upper cervical spinal cord evoked vasodilatation after adrenergic blockade. Sympathetic fibres mediating sustained vasodilatation were shown to be separate from adrenergic sympathetic fibres since the adrenergic vasoconstrictor response in the paw evoked by vasomotor stimulation in the medulla was not reversed to vasodilatation after bretylium.Supported by USPHS Grant No. HL 08570 and part of thesis work by Dr.Rolewicz.