浅谈缀花草坪的组花形式

缀花草坪,是在草坪上种植布置少量的草本花卉。缀花草坪花卉种植面积不能超过草坪面积的30%,花卉分布疏密有致,自然错落,多用于游憩草坪和观赏草坪。缀花草坪的组花在国外也称野花组合,起源于西方草坪业发展较早的国家,它由多种一年生、二年生和多年生花卉种子混合而成,其大多数的一年生野花具有结实自播的能力,多年生的野花可维持3-5年无需重新播种。经过多年的筛选和培育,目前可供选择的野花已达200种以上,完全可以根据我国不同区域的气候、土壤、栽培条件和使用目的配比出多种组合满足需要。     

浅谈缀花草坪的组花形式

缀花草坪,是在草坪上种植布置少量的草本花卉。缀花草坪花卉种植面积不能超过草坪面积的30%,花卉分布疏密有致,自然错落,多用于游憩草坪和观赏草坪。缀花草坪的组花在国外也称野花组合,起源于西方草坪业发展较早的国家,它由多种一年生、二年生和多年生花卉种子混合而成,其大多数的一年生野花具有结实自播的能力,多年生的野花可维持3-5年无需重新播种。经过多年的筛选和培育,目前可供选择的野花已达200种以上,完全可以根据我国不同区域的气候、土壤、栽培条件和使用目的配比出多种组合满足需要。     

草坪在石化企业的绿化作用及种植养护技术

本文论述石化企业厂区绿化中,草坪绿化优点和作用。阐述了草坪种植方法和养护管理技术,对盐碱地种植草坪和草坪更新进行了详细说明。     

常夏石竹在草坪中的应用及其品种选育与组培快繁

20世纪80年代,我国北方地区的绿化开始应用冷季型草坪.考虑到我国北方地区水资源普遍缺乏,山西省缺水现象更为严重,我们认为在北方种植这类草坪,不符合草坪草种选择的基本原则.常夏石竹的应用弥补了草坪无花和我国北方地区草坪冬季枯黄的缺陷,并在一定程度上解决了我国北方地区因干旱及寒冷给草坪种植带来的难题,促成了草坪业的一次飞跃.然而,常夏石竹叶色发蓝和叶面的白粉,使建植的草坪颜色发灰,从而降低了草坪的质量与观赏价值.     

论草坪自然属性的回归

在传统草坪审美观指导下的精细化管理,极大地限制了草坪自然属性的发挥.通过对草坪形成的条件、草坪生态系统演化、草坪景观及游憩活动特点等方面的讨论,提出回归草坪自然属性的理念.自然化草坪是带有自然属性的草坪类型,是在人工草坪的基础上自然演替形成的草坪植物群落,是在城市环境条件下配合低维护管理措施,形成的带有人工管理属性又不失其自然性的草坪类型.     

暖季型草坪草的养护与管理技术研究

近年来,草坪的应用领域越来越广泛,在园林绿化美化方面逐渐扮演了主要角色。本文以滁州市广泛栽培的暖季型草坪草为研究对象,结合笔者工作实践,对草坪的养护管理核心技术和草坪的辅助养护管理进行了探讨,并针对草坪的养护管理提出优化建议。     

田径足球场的运动草坪设计

运动场草坪质量的好坏,不仅反映了草坪拥有者的层次和实力,也客观地展现着草坪管理者的科学素质、技术水平和工作态度。对场地有特殊要求的足球场草坪是经济、科学、技术管理诸因素的综合体现,而一个设计合理、科学的运动场是决定草坪质量好坏的前提。     

浅谈草坪建植及管理养护应注意事项

草坪作为整体环境绿化的基色,其对环境绿化美化的重要功效,已被人们普遍认可。近年来,我国草坪业的发展迅速,每年草坪草种的引种数量成倍增长,草坪面积不断扩大,这给原本就滞后的草坪管理带来了更加严重的问题。本文针对草坪建植和管理养护中应注意的一些技术问题提出自己的看法,供同仁参考。     

园林施工新工艺的管理措施研究

2000年以来,随着红花岗区冷季型草坪面积的不断增加,如何管理好冷季型草坪,并在节约成本的情况下使其常青常绿,是我们急需解决的一个难题。以园林冷季型草坪草施工为例,在分析草坪在城市绿化中的作用及意义的基础上,重点就冷季型草坪草种植和管理技术进行了分析。     

草坪主要虫害的防治方法

草坪上栖息有多种有害昆虫。它们取食草坪草、污染草地、传播疾病,常使草坪遭受损毁,严重影响草坪的质量。因此,消灭害虫,保护草坪,是草坪建植管理的重要措施之一。     

幽门螺杆菌:胃炎和消化性溃疡的致病菌--2005年诺贝尔生理学或医学奖成果介绍

长期以来,医学界普遍认为,精神压力和生活方式等因素是慢性胃炎和消化性溃疡发病的主要原因.2名澳大利亚科学家不受主流医学思想的限制,开创性地证明了一种新发现的细菌——幽门螺杆菌才是胃炎和消化性溃疡的致病原因,从而改变了人们对这种疾病的认识和治疗手段,使胃炎和消化性溃疡成为一种可治愈、不再反复发作的慢性疾病.为表彰两人的贡献,瑞典皇家Karolinska医学院诺贝尔奖评审委员会决定授予他们2005年诺贝尔生理学或医学奖.     

小胶质细胞在不同炎性环境下亚型和功能的演变及其意义

近年来随着对多种神经系统炎性变性病如阿尔茨海默氏病(AD)、帕金森氏病(PD)和多发性硬化(MS)等发病机制的深入研究,显示脑内慢性免疫炎症反应可能是其重要病理特征之一,而小胶质细胞在其中所起的重要作用也日益受到人们的关注。小胶质细胞是中枢神经系统(CNS)的重要组成部分,同时又是脑内的主要免疫效应细胞。正常成年人脑中,小胶质细胞处于相对静息状态,而在免疫炎性反应过程中,小胶质细胞显示激活的细胞形态,在向病灶区移行的同时,可以产生神经毒性分子和神经营养因子,具有致炎和抗炎的作用。因此小胶质细胞的双重性对于理解疾病的发生发展,以及寻找疾病的治疗靶点具有极其重要的理论和实际意义。     

中国无病毒果树研究的进展

病毒病对果树生产的危害日趋严重，各国对病毒病害的研究和防治极为重视。本文主要论述了中国在果树病毒的检测、脱除以及抗病毒基因工程、生长结果习性和生理生化特性等方面的研究进展和取得的成就，同时指出今后应注意的一些问题。     

Transmissible and genetic prion diseases share a common pathway of neurodegeneration

Prion diseases can be infectious, sporadic and genetic. The infectious forms of these diseases, including bovine spongiform encephalopathy and Creutzfeldt-Jakob disease, are usually characterized by the accumulation in the brain of the transmissible pathogen, an abnormally folded isoform of the prion protein (PrP) termed PrPSc. However, certain inherited PrP mutations appear to cause neurodegeneration in the absence of PrPSc, working instead by favoured synthesis of CtmPrP, a transmembrane form of PrP. The relationship between the neurodegeneration seen in transmissible prion diseases involving PrPSc and that associated with ctmPrP has remained unclear. Here we find that the effectiveness of accumulated PrPSc in causing neurodegenerative disease depends upon the predilection of host-encoded PrP to be made in the ctmPrP form. Furthermore, the time course of PrPSc accumulation in transmissible prion disease is followed closely by increased generation of CtmPrP. Thus, the accumulation of PrPSc appears to modulate in trans the events involved in generating or metabolising CtmPrP. Together, these data suggest that the events of CtmPrP-mediated neurodegeneration may represent a common step in the pathogenesis of genetic and infectious prion diseases.     

An in-depth analysis of proteomics expression profiling in rat glomeruli utilizing LC-MS

Glomeruli are an essential functional element of renal filtration. The majority of renal diseases caused by glomerular sclerosis or fibrosis may result in renal dysfunction. A fomulate protein profile, a comprehensive analysis of glomeruli of normal rats was conducted in this study via protein spectrum. Functional annotation and classification of these proteins were performed and it was found that 26 had the same glomerule (endothelial cells, podocytes and mesangial cells) markers with proteins.     

Therapeutic approaches to protein-misfolding diseases

Several sporadic and genetic diseases are caused by protein misfolding. These include cystic fibrosis and other devastating diseases of childhood as well as Alzheimer's, Parkinson's and other debilitating maladies of the elderly. A unified view of the molecular and cellular pathogenesis of these conditions has led to the search for chemical chaperones that can slow, arrest or revert disease progression. Molecules are now emerging that link our biophysical insights with our therapeutic aspirations.     

The roles of intracellular protein-degradation pathways in neurodegeneration

Many late-onset neurodegenerative diseases, including Parkinson's disease and Huntington's disease, are associated with the formation of intracellular aggregates by toxic proteins. It is therefore crucial to understand the factors that regulate the steady-state levels of these 'toxins', at both the synthetic and degradation stages. The degradation pathways acting on such aggregate-prone cytosolic proteins include the ubiquitin-proteasome system and macroautophagy. Dysfunction of the ubiquitin-proteasome or macroautophagy pathways might contribute to the pathology of various neurodegenerative conditions. However, enhancing macroautophagy with drugs such as rapamycin could offer a tractable therapeutic strategy for a number of these diseases.     

颅内结节状及小环状强化的影像学研究与临床

目的 探讨颅内病变呈结节状及小环状强化的各自特点及鉴别要点。方法 追踪观察了268例在CT,MRI呈结节状及小环状强化的病例。结果 脑脓肿、炎性肉芽肿、结核瘤、脑囊虫病、胶质瘤及转移瘤等13种疾病可见结节状及小环状强化表现,并各有其特点。结论 通过观察影像学表现特点,结合病史及临床表现即可得出诊断。     

蒙古族心血管疾病及其危险因素研究进展

大量流行病学和遗传学研究证实,冠心病、脑卒中等心脑血管疾病是由遗传因素与环境因素共同作用所致的复杂疾病。心血管疾病的重要危险因素有高血压、高血脂、高血糖、肥胖、吸烟等。这些危险因素对预测心血管疾病的发病有重要的意义。现通过查阅文献,对蒙古族心血管疾病及其危险因素研究现状进行综述,为今后进一步进行蒙古族人群心血管疾病研究提供理论依据。     

Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases

Many lines of evidence suggest that mitochondria have a central role in ageing-related neurodegenerative diseases. Mitochondria are critical regulators of cell death, a key feature of neurodegeneration. Mutations in mitochondrial DNA and oxidative stress both contribute to ageing, which is the greatest risk factor for neurodegenerative diseases. In all major examples of these diseases there is strong evidence that mitochondrial dysfunction occurs early and acts causally in disease pathogenesis. Moreover, an impressive number of disease-specific proteins interact with mitochondria. Thus, therapies targeting basic mitochondrial processes, such as energy metabolism or free-radical generation, or specific interactions of disease-related proteins with mitochondria, hold great promise.