大学生思想政治态度的影响因素及对策研究

本文选择大学生思想政治态度为研究对象,分析大学生思想政治态度的现状,目的是了解大学生目前的思想政治状况及其影响因素,以便加强和改进大学生思想政治教育,使大学生思想政治教育更加富有针对性和实效性.     

大学生思想政治教育的价值导向悖论及其重构

教育价值是增强大学生思想政治教育实效性不可忽视的重要因素.从价值导向入手,分析了大学生思想政治教育价值导向的几个悖论,并提出相应对策,以进一步提高大学生思想政治教育的实效性.     

增强大学生思想政治教育实效性的对策研究

高校必须并针对新形势下大学生思想政治教育的新特点、新要求,认真分析制约思想政治教育实效性进一步提高因素,更新思想政治教育观念、充实思想政治教育内容、整合思想政治教育资源、拓宽思想政治教育途径、完善思想政治教育机制,以大学生的全面发展为目标,切实提高思想政治教育实效性。     

大学生思想政治教育的前瞻性思考

要掌握大学生思想政治教育的主动权,必须增强大学生思想政治教育的预见性,提高大学生思想政治教育的针对性、实效性.这样,才能抢占大学生思想政治教育的制高点,掌握德育工作的主动权,提高大学生思想政治教育的针对性、科学性与艺术性,使高校学生思想政治工作得以积极主动、深入扎实地开展,并以此提高大学生思想政治教育的实效性与有效性.     

试论提高大学生思想政治教育的实效性

充实教材是提高大学生思想政治教育实效性的前提;增强教师的适应性是提高大学生思想政治教育实效性的根本;加强教学的针对性是提高大学生思想政治教育实效性的关键;加强综合素质的培养是提高大学生思想政治教育实效性的目标.     

大学生思想政治工作实效性研究

从新形势下提高大学生思想政治教育实效性地位和作用入手，对当前制约大学生思想政治教育实效性的因素进行了较全面和系统的分析，并就如何提高大学生思想政治教育的实效性提出了一些粗浅的看法和意见。     

试谈如何增强大学生思想政治教育的实效性

新时期．大学生思想政治教育面临着一个实效性问题。本文简要论述了如何运用情感因素和激励机制，加强和改进大学生思想政治教育，增强思想政治教育的实效性。     

增强大学生思想政治教育实效性的途径探索

文章对影响大学生思想政治教育取得实效性的各种因素进行了简要分析,并初步提出增强大学生思想政治教育实效性的方法和途径。     

论构建和谐的大学生思想政治教育生态环境

当代大学生的思想政治教育受到各种因素的影响,深刻分析大学生思想政治教育的生态环境,探索构建和谐的大学生思想政治教育生态环境的路径,对于搞好大学生思想政治教育工作,增强大学生思想政治教育的实效性具有非常重要的意义。     

大学生思想政治教育实效性探析、

深刻剖析制约大学生思想政治教育实效性的诸多因素，明确大学生思想政治教育的价值和任务。     

Distribution of factor VIII mRNA and antigen in human liver and other tissues

The cellular site of synthesis of factor VIII (FVIII:C; anti-haemophilic factor) has long been sought. Previous studies suggested the liver as a major site of synthesis, but extrahepatic sources such as spleen and lung have been implicated. Using an immunoradiometric assay (IRMA), we recently localized factor VIII antigen (FVIII:Ag, formerly FVIII:CAg), to whole perfused guinea pig liver and spleen, and to isolated hepatocytes, with lesser or trace amounts in other tissues. Using an immunohistological technique, Stel et al. detected FVIII:Ag in normal human liver sinusoidal endothelial cells, while Exner et al. detected FVIII:Ag by IRMA in extracts of human lymph nodes, lung, liver and spleen. The localization of antigen in tissues does not, however, distinguish sites of factor VIII synthesis from those of storage, and such experiments are subject to misinterpretation due to entrapment of plasma factor VIII in tissues. The recent cloning of the human factor VIII gene provides hybridization probes for the detection of factor VIII messenger RNA in cells, thus directly determining sites of synthesis. During complementary DNA cloning, we detected factor VIII mRNA in liver, and it has been localized by others in liver and placenta and in liver and kidney. In the present study, we detected factor VIII mRNA in isolated human hepatocytes, in spleen and in numerous tissues including lymph nodes and kidney, but not in white blood cells or cultured endothelial cells. We also found that the factor VIII, factor VII, factor IX and protein C antigens in liver are predominantly localized in hepatocytes, while very little von Willebrand factor antigen (vWF:Ag, formerly FVIIIR Ag) is detectable in this organ.     

Ultrastructural localization of factor VIII procoagulant antigen in human liver hepatocytes

Factor VIII is generally believed to circulate in blood as a multimeric complex of two glycoproteins which are physiologically and immunologically distinct. One component of the factor VIII complex is factor VIII procoagulant activity (FVIII:C) which is associated with factor VIII/procoagulant antigen (FVIII:Ag, formerly FVIII/CAg). The second, larger unit of the complex is factor VIII/von Willebrand factor (vWF:Ag, formerly factor VIII-related antigen or FVIIIRAg). FVIII:C has anti-haemophilic activity and is defective or deficient in patients with classical haemophilia, and vWF:Ag is absent in patients with von Willebrand disease. FVIII:Ag was demonstrated recently in endothelial cells lining hepatic sinusoids, by using immunoperoxidase staining and light microscopy, whereas biochemical data had indicated its presence predominantly in the hepatocyte fractions and in lesser amounts in endothelial cells. Moreover, recent hybridization experiments detected FVIII:C messenger RNA in liver and kidney tissues. Despite several efforts, the cells responsible for FVIII:C synthesis have not been unequivocally identified. Here we use protein A-gold complex labelling to demonstrate the ultrastructural localization of FVIII:C in human liver cells; the results indicate that hepatocytes may synthesize FVIII:Ag.     

基于因子分析的沿海可再生能源开发结构优化模型

针对沿海可再生能源特点与开发技术和安全要求,系统地分析并选取开发结构优良性指标,在此基础上应用因子分析法从相关性指标中提取综合因子,包括经济因子、技术因子、环境因子和安全因子,作为能源开发结构优化的综合性能度量.以经济成本、碳排放量、能源开发比例组合效用最大为目标函数,从能源、经济、技术、环境和安全五方面构建约束,提出了沿海可再生能源开发结构优化多目标规划模型.以大连为例,得到该地区目标年的最优能源开发结构.研究成果可为可再生能源开发结构规划与政策制定提供参考.     

医院内肺部真菌感染90例临床分析

目的 :了解医院内肺部真菌感染的基本情况及影响因素。方法 :对90例医院内肺部真菌感染的病例进行回顾性分析。结果 :肺部真菌感染率约为18.5%,主要基础疾病是慢性阻塞性肺部疾病、肿瘤、慢性肾衰、系统性红斑狼疮等疾病。不合理使用抗菌素药物是院内肺部疾真菌感染的一最为常见的危险因素。营养不良是院内肺部真菌感染的另一个重要诱因。结论 :医院内肺部真菌感染主要危险因素之一 ,是不合理使用抗菌素药物及基础疾病的存在 ,应引起我们临床医师的高度重视     

Cr~(4+):Ca_2GeO_4晶体光谱的理论研究

分别用平均共价因子和双共价因子模型研究了Cr~(4+):Ca_2GeO_4晶体的光谱,研究结果表明:平均共价因子模型下,计算值与实验值相差甚远;而双共价因子模型下的计算值与实验数据符合得很好.这说明传统的平均共价因子模型不能很好地解释Cr~(4+):Ca_2GeO_4晶体的光谱,而双共价因子模型能改进平均共价因子的不足,更好地解释Cr~(4+):Ca_2GeO_4晶体的光谱.     

Polymorphisms in the genes for coagulation factor Ⅱ,Ⅴ,Ⅶ in patients undergoing coronary angiography

OBJECTIVE: To determine whether polymorphisms in the genes for coagulation factor II, V, VII could predispose an individual to increase risk for coronary artery disease (CAD) and/or myocardial infarction (MI) in Chinese. METHODS: We screened coagulation factor II(G20210A),V(G1691A),VII (R353Q and HVR4) genotype in 374 patients undergoing coronary angiography by polymerase chain reaction and restriction fragment length polymorphism (PCR-RFLP) assay. RESULTS: The R353Q and HVR4 genotype of the factor VII distribution was in accordance with Hardy-Weinberg equilibrium. The frequencies of FVII genotype or allele did not show statistically significant differences between CAD group and controls or between male and female. The frequencies of the Q allele and (RQ + QQ) genotype were significantly higher among the CAD patients without myocardial infarction (MI) history than among those with MI history (P < 0.05). However, HVR4 polymorphism was not significantly different within groups. We only find one normal control of factor II (G20210A) mutation. No coagulation factor V(G1691A) mutation was found in the CAD patients and controls. CONCLUSION: The factor II(G20210A) ,V(G1691A) mutation is absent and may not be a major genetic factor for CAD and/or MI; the Q allele of the R353Q polymorphism of the factor VII gene may be a protective genetic factor against myocardial infarction in Chinese.     

Trigger factor and DnaK cooperate in folding of newly synthesized proteins.

The role of molecular chaperones in assisting the folding of newly synthesized proteins in the cytosol is poorly understood. In Escherichia coli, GroEL assists folding of only a minority of proteins and the Hsp70 homologue DnaK is not essential for protein folding or cell viability at intermediate growth temperatures. The major protein associated with nascent polypeptides is ribosome-bound trigger factor, which displays chaperone and prolyl isomerase activities in vitro. Here we show that delta tig::kan mutants lacking trigger factor have no defects in growth or protein folding. However, combined delta tig::kan and delta dnaK mutations cause synthetic lethality. Depletion of DnaK in the delta tig::kan mutant results in massive aggregation of cytosolic proteins. In delta tig::kan cells, an increased amount of newly synthesized proteins associated transiently with DnaK. These findings show in vivo activity for a ribosome-associated chaperone, trigger factor, in general protein folding, and functional cooperation of this protein with a cytosolic Hsp70. Trigger factor and DnaK cooperate to promote proper folding of a variety of E. coli proteins, but neither is essential for folding and viability at intermediate growth temperatures.     

农户层面的农用地流转影响因子分析--以三峡库区沿江村为例

采取参与式农村评估法(PRA),以三峡库区3个沿江村为例,共调查农户276户,通过因子分析方法,从农户角度定量分析了三峡库区沿江村农用地流转的影响因素.研究表明:应用因子分析方法可以将农户层面影响三峡库区沿江村农用地流转的因素归纳为务工因子、务农劳动力因子、政府补偿收入因子、户主因子、教育水平因子、收入因子等6个因子.针对此提出积极转移非农从业人员、提高政府补偿收入、重视农户户主、提高农户的教育水平等对策以促进三峡库区农用地的流转.     

围产儿死亡103例临床分析

目的探讨围产儿死亡原因,以降低围产儿死亡率。方法回顾分析1998年8月～2008年8月该院围产儿死亡病历。结果103例围产儿死亡主要原因为早产、胎儿畸形、脐带因素、新生儿窒息和胎盘因素。结论加强产前健康教育,提高产科、新生儿质量及围产儿保健工作,尽早筛查胎儿畸形可降低围产儿死亡率。     

转型期的经济增长——基于制度因素的解释

在罗默(Romer)的研发模型中引入制度因素,建立了含制度因素的内生经济增长模型,从理论上分析了制度因素对长期经济增长的影响,进而从实证的角度分析了改革开放这一制度因素与转型期我国经济增长的关系。研究结果表明:制度因素是推动转型期我国经济增长的重要因素,持续的制度创新是创造我国又一个"经济增长奇迹"的关键。